PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25761110-4	2015	Here, we report the effects of the specifically-neuronal human glucose transporter expression in neurons of a Drosophila model carrying the exon 1 of the human huntingtin gene with 93 glutamine repeats (HQ93).	Glucose	63-70	huntingtin	Homo sapiens	160-170	
19628478-4	2009	We demonstrate that mutant huntingtin impairs glucose-stimulated insulin secretion in insulin-producing beta-cells, without altering stored levels of insulin.	Glucose	46-53	huntingtin	Homo sapiens	27-37	
19602103-5	2009	Furthermore, we expressed N-terminal huntingtin fragments with different polyglutamine lengths in an insulinoma-cell line (INS-1E) to investigate how mutant huntingtin influences glucose-stimulated insulin release in vitro.	Glucose	179-186	huntingtin	Homo sapiens	157-167	
19602103-7	2009	However, the glucose-stimulated induction of insulin release was significantly reduced in the insulinoma-cell line expressing highly expanded huntingtin compared to cells expressing huntingtin with modestly elongated polyglutamine stretches.	Glucose	13-20	huntingtin	Homo sapiens	142-152	
18984744-5	2009	Similarly to Huntington"s disease patients, mice expressing the mutated human huntingtin protein also exhibit neurodegenerative changes, motor dysfunction, perturbed energy metabolism, and elevated blood glucose levels.	Glucose	204-211	huntingtin	Homo sapiens	78-88	
10516137-4	1999	In low AR-expressing cells, 20 mM glucose decreased taurine content, hTT transporter activity, and mRNA levels, and these effects were unaffected by AR inhibition (ARI).	Glucose	34-41	huntingtin	Homo sapiens	69-72	
10516137-5	1999	In these cells, the inhibitory effects of high glucose on hTT appeared to be posttranscriptionally mediated, because 20 mM glucose decreased hTT mRNA stability without affecting hTT transcriptional rate.	Glucose	47-54	huntingtin	Homo sapiens	58-61	
10516137-5	1999	In these cells, the inhibitory effects of high glucose on hTT appeared to be posttranscriptionally mediated, because 20 mM glucose decreased hTT mRNA stability without affecting hTT transcriptional rate.	Glucose	47-54	huntingtin	Homo sapiens	141-144	
10516137-5	1999	In these cells, the inhibitory effects of high glucose on hTT appeared to be posttranscriptionally mediated, because 20 mM glucose decreased hTT mRNA stability without affecting hTT transcriptional rate.	Glucose	47-54	huntingtin	Homo sapiens	141-144	
10516137-5	1999	In these cells, the inhibitory effects of high glucose on hTT appeared to be posttranscriptionally mediated, because 20 mM glucose decreased hTT mRNA stability without affecting hTT transcriptional rate.	Glucose	123-130	huntingtin	Homo sapiens	58-61	
10516137-5	1999	In these cells, the inhibitory effects of high glucose on hTT appeared to be posttranscriptionally mediated, because 20 mM glucose decreased hTT mRNA stability without affecting hTT transcriptional rate.	Glucose	123-130	huntingtin	Homo sapiens	141-144	
10516137-5	1999	In these cells, the inhibitory effects of high glucose on hTT appeared to be posttranscriptionally mediated, because 20 mM glucose decreased hTT mRNA stability without affecting hTT transcriptional rate.	Glucose	123-130	huntingtin	Homo sapiens	141-144	
10516137-6	1999	Inhibition of PKC overcame the decrease in hTT activity in high glucose-exposed cells.	Glucose	64-71	huntingtin	Homo sapiens	43-46	
10516137-8	1999	In these cells exposed to 5 mM glucose, hTT mRNA abundance was decreased and declined further in 20 mM glucose but was corrected by ARI.	Glucose	31-38	huntingtin	Homo sapiens	40-43	
10516137-8	1999	In these cells exposed to 5 mM glucose, hTT mRNA abundance was decreased and declined further in 20 mM glucose but was corrected by ARI.	Glucose	103-110	huntingtin	Homo sapiens	40-43	
10516137-9	1999	In 5 mM glucose, hTT transcriptional rate was markedly decreased in high AR-expressing cells, did not decline further in 20 mM glucose, but was increased by ARI to levels above those observed in low AR-expressing cells.	Glucose	8-15	huntingtin	Homo sapiens	17-20	
10516137-9	1999	In 5 mM glucose, hTT transcriptional rate was markedly decreased in high AR-expressing cells, did not decline further in 20 mM glucose, but was increased by ARI to levels above those observed in low AR-expressing cells.	Glucose	127-134	huntingtin	Homo sapiens	17-20	
10516137-10	1999	Therefore, glucose rapidly and specifically decreases taurine content, hTT activity, and mRNA abundance by AR-unrelated and AR-related posttranscriptional and transcriptional mechanisms.	Glucose	11-18	huntingtin	Homo sapiens	71-74