PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31949826-5	2018	Furthermore, the inhibitor improved fibrosis induced by high levels of glucose in HK-2 cells by suppressing E-cadherin, alpha-SMA, and FSP-1 expression.	Glucose	71-78	atlastin GTPase 1	Homo sapiens	135-140	
23153508-5	2013	We found that in vitro high glucose treatment led to remarkable down-regulation of nephrin and P-cadherin, as well as significant up-regulation of alpha-SMA and FSP-1, suggesting that in the presence of high glucose, podocytes underwent EMT, during which Rac1/PAK1 signaling was activated.	Glucose	28-35	atlastin GTPase 1	Homo sapiens	161-166	
23153508-5	2013	We found that in vitro high glucose treatment led to remarkable down-regulation of nephrin and P-cadherin, as well as significant up-regulation of alpha-SMA and FSP-1, suggesting that in the presence of high glucose, podocytes underwent EMT, during which Rac1/PAK1 signaling was activated.	Glucose	208-215	atlastin GTPase 1	Homo sapiens	161-166