PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16249254-9	2006	Glutamine dose-dependently increased GAD-mediated formation of GABA, whereas glucose metabolism shunts part of this GABA to mitochondrial catabolism, involving alpha-KG-induced activation of GABA-T.	Glucose	77-84	4-aminobutyrate aminotransferase	Homo sapiens	191-197	
16249254-10	2006	In absence of extracellular glutamine, glucose also contributed to GABA formation through aminotransferase generation of glutamate from alpha-KG; this stimulatory effect increased GABA release only when GABA-T activity was suppressed.	Glucose	39-46	4-aminobutyrate aminotransferase	Homo sapiens	203-209	
16249254-12	2006	Glucose inhibits glutamine-driven GABA formation and release through increasing GABA-T shunt activity by its cellular metabolism.	Glucose	0-7	4-aminobutyrate aminotransferase	Homo sapiens	80-86