PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31726133-4	2020	A structural analysis showed that the monosaccharides of EPS H31-2 included d-mannose (Man) and d-glucose (Glc).	Glucose	96-105	H3 clustered histone 3	Homo sapiens	61-64	
31726133-4	2020	A structural analysis showed that the monosaccharides of EPS H31-2 included d-mannose (Man) and d-glucose (Glc).	Glucose	107-110	H3 clustered histone 3	Homo sapiens	61-64	
31726133-8	2020	In addition, the supernatant glucose concentration of insulin-resistant HepG2 cells decreased by treatment with 10-8 M of EPS H31-2, which meant EPS H31-2 could promote the glucose uptake of insulin-resistant HepG2 cell.	Glucose	29-36	H3 clustered histone 3	Homo sapiens	126-129	
31726133-8	2020	In addition, the supernatant glucose concentration of insulin-resistant HepG2 cells decreased by treatment with 10-8 M of EPS H31-2, which meant EPS H31-2 could promote the glucose uptake of insulin-resistant HepG2 cell.	Glucose	29-36	H3 clustered histone 3	Homo sapiens	149-152	
31726133-8	2020	In addition, the supernatant glucose concentration of insulin-resistant HepG2 cells decreased by treatment with 10-8 M of EPS H31-2, which meant EPS H31-2 could promote the glucose uptake of insulin-resistant HepG2 cell.	Glucose	173-180	H3 clustered histone 3	Homo sapiens	126-129	
31726133-8	2020	In addition, the supernatant glucose concentration of insulin-resistant HepG2 cells decreased by treatment with 10-8 M of EPS H31-2, which meant EPS H31-2 could promote the glucose uptake of insulin-resistant HepG2 cell.	Glucose	173-180	H3 clustered histone 3	Homo sapiens	149-152