PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 12399107-3 2002 Such strong co-localisation was maintained following treatment of cells with cytochalasin D, which inhibits actin polymerization and which is capable of inhibiting ICAM-1-induced signalling. Cytochalasin D 77-91 intercellular adhesion molecule 1 Homo sapiens 164-170 20445173-8 2010 A simple explanation is that CD induces both clustering and integrin motion, fixation then stops motion on fixed cells, but integrins continue to diffuse on unfixed cells, increasing the kinetics of integrin/ICAM-1 interactions to enhance adhesion. Cytochalasin D 29-31 intercellular adhesion molecule 1 Homo sapiens 208-214 19114730-7 2009 Cytochalasin D, an actin polymerization inhibitor, suppressed the PCOOH-induced cell adhesion to ICAM-1 and the membrane protrusions. Cytochalasin D 0-14 intercellular adhesion molecule 1 Homo sapiens 97-103 19107907-7 2009 Cytochalasin D (an actin filament disruptor) partially inhibited CSE-induced ICAM-1 and E-selectin surface expression. Cytochalasin D 0-14 intercellular adhesion molecule 1 Homo sapiens 77-83 14688373-8 2004 Inhibition of LPS internalization by cytochalasin D or treatment with dibutyryl cAMP attenuated ICAM-1 expression and TNF production by PBMC. Cytochalasin D 37-51 intercellular adhesion molecule 1 Homo sapiens 96-102 15034928-6 2004 A functioning actin cytoskeleton was important for VCAM-1 and ICAM-1 expression as both cytochalasin D, an actin filament disruptor, and jasplakinolide, an actin filament stabilizer, attenuated the expression of these CAMs. Cytochalasin D 88-102 intercellular adhesion molecule 1 Homo sapiens 62-68 10623819-7 2000 Up-regulated adhesion to ICAM-1 was abolished by cytochalasin D, an inhibitor of cytoskeletal rearrangement. Cytochalasin D 49-63 intercellular adhesion molecule 1 Homo sapiens 25-31