PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
2380172-2	1990	Cholesterol-dependent enhancement of self-quenching also occurs when N-(lissamine-rhodamine-B-sulfonyl)dioleoylphosphatidylethanolamine is substituted for C18-Rh and resembles that in dipalmitoylphosphatidylcholine vesicles below, as opposed to above, the phase transition.	1,2-Dipalmitoylphosphatidylcholine	184-214	Bardet-Biedl syndrome 9	Homo sapiens	155-158	
35483419-6	2022	Using large unilamellar vesicles, we found that upon replacing 10 mol% DPPC with either C16:0 or C18:0 ceramide, or 16:0 diacylglycerol (dag), lipid domains persisted to higher temperatures.	1,2-Dipalmitoylphosphatidylcholine	71-75	Bardet-Biedl syndrome 9	Homo sapiens	97-100	
35483419-7	2022	However, when DPPC was replaced with short chain (C6:0 or C12:0) or very long chain (C24:0) ceramides, or ceramides with unsaturated acyl chains of any length (C6:1(3), C6:1(5), C18:1, and C24:1), as well as C18:1-dag, lipid domains were destabilized, melting at lower temperatures than those in the DPPC/DOPC/cholesterol system.	1,2-Dipalmitoylphosphatidylcholine	14-18	Bardet-Biedl syndrome 9	Homo sapiens	178-181	
35483419-7	2022	However, when DPPC was replaced with short chain (C6:0 or C12:0) or very long chain (C24:0) ceramides, or ceramides with unsaturated acyl chains of any length (C6:1(3), C6:1(5), C18:1, and C24:1), as well as C18:1-dag, lipid domains were destabilized, melting at lower temperatures than those in the DPPC/DOPC/cholesterol system.	1,2-Dipalmitoylphosphatidylcholine	14-18	Bardet-Biedl syndrome 9	Homo sapiens	208-211	
29499181-5	2018	Long-tailed analogues (C16, C18) are cohesively integrated into DPPC monolayers due to their smaller cone angles at the interfacial region and increased hydrocarbon compatibility in the hydrophobic region.	1,2-Dipalmitoylphosphatidylcholine	64-68	Bardet-Biedl syndrome 9	Homo sapiens	28-31