PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
21143607-3	2011	Using COH-BR1 breast cancer cells sensitized in mitochondria with 5-aminolevulinic acid (ALA)-generated protoporphyrin IX as a model for ALA-based PDT, we found that caspase-9 activation and apoptotic death following irradiation were strongly enhanced by 1400W, an inhibitor of inducible nitric oxide synthase (iNOS).	5-amino levulinic acid	66-87	caspase 9	Homo sapiens	166-175	
26200607-9	2016	ALA-PDT induced a relatively weak and shorter caspase-9 activation but a strong and long lasting caspase-3 activation.	5-amino levulinic acid	0-3	caspase 9	Homo sapiens	46-55	
26200607-10	2016	However, mRNA levels of caspase-9 and -3 genes in cells treated with ALA-PDT were unchanged.	5-amino levulinic acid	69-72	caspase 9	Homo sapiens	24-40	
21143607-3	2011	Using COH-BR1 breast cancer cells sensitized in mitochondria with 5-aminolevulinic acid (ALA)-generated protoporphyrin IX as a model for ALA-based PDT, we found that caspase-9 activation and apoptotic death following irradiation were strongly enhanced by 1400W, an inhibitor of inducible nitric oxide synthase (iNOS).	5-amino levulinic acid	89-92	caspase 9	Homo sapiens	166-175	
21143607-3	2011	Using COH-BR1 breast cancer cells sensitized in mitochondria with 5-aminolevulinic acid (ALA)-generated protoporphyrin IX as a model for ALA-based PDT, we found that caspase-9 activation and apoptotic death following irradiation were strongly enhanced by 1400W, an inhibitor of inducible nitric oxide synthase (iNOS).	5-amino levulinic acid	137-140	caspase 9	Homo sapiens	166-175