PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
17301081-5	2007	Since the reaction catalysed by succinyl-CoA synthase in the tricarboxylic acid (TCA) cycle represents a distal step of the methylmalonic acid pathway, we investigated the SUCLA2 gene as a candidate gene in our patients.	Tricarboxylic Acids	61-79	succinate-CoA ligase ADP-forming subunit beta	Homo sapiens	172-178	
25315394-8	2014	SUCLA2 and PDHB are involved in the tricarboxylic acid cycle, whereas PHGPx and PRDX5 are involved in glutathione metabolism.	Tricarboxylic Acids	36-54	succinate-CoA ligase ADP-forming subunit beta	Homo sapiens	0-6	
17301081-5	2007	Since the reaction catalysed by succinyl-CoA synthase in the tricarboxylic acid (TCA) cycle represents a distal step of the methylmalonic acid pathway, we investigated the SUCLA2 gene as a candidate gene in our patients.	Tricarboxylic Acids	81-84	succinate-CoA ligase ADP-forming subunit beta	Homo sapiens	172-178	
33230181-3	2020	Here, we show that succinyl-CoA accumulates in cells derived from patients with recessive mutations in the tricarboxylic acid cycle (TCA) gene succinyl-CoA ligase subunit-beta (SUCLA2), causing global protein hyper-succinylation.	Tricarboxylic Acids	107-125	succinate-CoA ligase ADP-forming subunit beta	Homo sapiens	177-183	
27913098-1	2017	Mutations in SUCLA2 result in succinyl-CoA ligase (ATP-forming) or succinyl-CoA synthetase (ADP-forming) (A-SCS) deficiency, a mitochondrial tricarboxylic acid cycle disorder.	Tricarboxylic Acids	141-159	succinate-CoA ligase ADP-forming subunit beta	Homo sapiens	13-19