PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24323505-6	2014	Taken together, these results strongly suggest that acclimation-induced cross-tolerance is largely attributed to RBOH1-dependent H2O2 production at the apoplast, which may subsequently activate MPK1/2 to induce stress responses.	Hydrogen Peroxide	129-133	Mitogen-activated protein kinase	Solanum lycopersicum	194-198	
29868714-5	2018	Crosstalk among H2O2, NO, and MPK1/2 contributes to the homeostasis of H2O2 and NO, activation of MPK1/2, and cold tolerance.	Hydrogen Peroxide	71-75	Mitogen-activated protein kinase	Solanum lycopersicum	30-34	
29868714-5	2018	Crosstalk among H2O2, NO, and MPK1/2 contributes to the homeostasis of H2O2 and NO, activation of MPK1/2, and cold tolerance.	Hydrogen Peroxide	71-75	Mitogen-activated protein kinase	Solanum lycopersicum	30-36	
22994632-6	2013	Silencing of either MPK1 or MPK2, on the other hand, reduced RBOH1 transcript, H2 O2 accumulation and MPK1/2 activity.	Hydrogen Peroxide	79-84	Mitogen-activated protein kinase	Solanum lycopersicum	20-24	
22994632-9	2013	RBOH1, MPK1 and MPK2 were involved in the stress tolerance and BR-induced stress tolerance likely involved a positive feedback loop among RBOH1, H2 O2 and MPK2, leading to sustained apoplastic accumulation of H2 O2 and related signalling processes.	Hydrogen Peroxide	145-150	Mitogen-activated protein kinase	Solanum lycopersicum	7-11