PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
35179404-10	2022	FUNDC1 was further high expression in SRA01/04 cells with H2O2 treatment whereas hypomethylation of FUNDC1 in cataract lens cells under oxidative stress.	Hydrogen Peroxide	58-62	FUN14 domain containing 1	Homo sapiens	0-6	
30284230-0	2019	Hydrogen peroxide-induced mitophagy contributes to laryngeal cancer cells survival via the upregulation of FUNDC1.	Hydrogen Peroxide	0-17	FUN14 domain containing 1	Homo sapiens	107-113	
30284230-9	2019	Hydrogen peroxide significantly induced the elevation of FUNDC1, a mitophagic factor, in a time- and dose-dependent manner in laryngeal cancer cells, which was dependent on ERK signal activation.	Hydrogen Peroxide	0-17	FUN14 domain containing 1	Homo sapiens	57-63	
30284230-10	2019	Knockdown of FUNDC1 by the siRNA attenuated the survival of laryngeal cancer cells under hydrogen peroxide stimulation.	Hydrogen Peroxide	89-106	FUN14 domain containing 1	Homo sapiens	13-19	
30284230-11	2019	Moreover, the elevated FUNDC1 was required for the occurrence of mitophagy under hydrogen peroxide stimulation, which was identified by transmission electron microscopy, the alterations of mitochondrial permeability transition and the specific mitochondrial protein, hsp60.	Hydrogen Peroxide	81-98	FUN14 domain containing 1	Homo sapiens	23-29	
30284230-13	2019	CONCLUSIONS: Hydrogen peroxide upregulated the expression of FUNDC1 through the activation of ERK1/2 signal to trigger a mitophagic response, giving laryngeal cancer cells a befit for survival.	Hydrogen Peroxide	13-30	FUN14 domain containing 1	Homo sapiens	61-67