PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
11004044-8	2000	LPS enhanced halothane-induced 3.9 and 1.6-fold increases in rCBF at 1.0 and 1.5 minimum alveolar concentration, respectively.	Halothane	13-22	CCAAT/enhancer binding protein zeta	Rattus norvegicus	61-65	
11004044-9	2000	Co-treatment with NS-398 attenuated, but aminoguanidine or dexamethasone abolished the effect of LPS on halothane-induced rCBF increase.	Halothane	104-113	CCAAT/enhancer binding protein zeta	Rattus norvegicus	122-126	
11004044-10	2000	Diethylamine NONOate mimicked the enhanced rCBF response to halothane.	Halothane	60-69	CCAAT/enhancer binding protein zeta	Rattus norvegicus	43-47	
9397032-6	1997	Application of adenosine at various depths in neocortex of halothane-anesthetized rats showed a predominant CBF increase at the level of application.	Halothane	59-68	CCAAT/enhancer binding protein zeta	Rattus norvegicus	108-111	
9626296-4	1998	METHODS: Regional cerebral blood flow (rCBF) was measured by laser-Doppler flowmetry in halothane-anesthetized, artificially ventilated rats for 4 hours after intracerebroventricular administration of LPS.	Halothane	88-97	CCAAT/enhancer binding protein zeta	Rattus norvegicus	39-43	
7856894-3	1995	This study was undertaken to examine the effects of halothane and isoflurance on the distribution of CBF.	Halothane	52-61	CCAAT/enhancer binding protein zeta	Rattus norvegicus	101-104	
7478181-1	1995	We measured the increase of regional cerebral blood flow (rCBF) in the somatosensory cerebral cortex occurring in response to a standard stimulation of the L. side mystacial vibrissae (facial whiskers) in rats anaesthetised with halothane, in conjunction with blocking of activity in the R. side parasympathetic (PS) and sensory fibres innervating the cerebral vessels.	Halothane	229-238	CCAAT/enhancer binding protein zeta	Rattus norvegicus	58-62	
7674850-6	1995	Halothane is known to attenuate the changes in regional glucose utilisation produced by the noncompetitive NMDA antagonist dizocilpine (MK-801), and its effects on ketamine-induced changes in rCBF seen in this study may be due to a similar effect.	Halothane	0-9	CCAAT/enhancer binding protein zeta	Rattus norvegicus	192-196	
7856894-16	1995	CONCLUSIONS: There is a difference in the human rCBF distribution between halothane and isoflurane with higher relative flows in subcortical regions during isoflurane anesthesia.	Halothane	74-83	CCAAT/enhancer binding protein zeta	Rattus norvegicus	48-52	
973690-9	1976	In the absence of transient ischemia during occlusion (that is, rCBF greater than 18 ml/100 g/min), halothane and enflurane anesthesia were associated with significantly higher rCBF"s and lower stump pressures than was neuroleptanesthesia.	Halothane	100-109	CCAAT/enhancer binding protein zeta	Rattus norvegicus	64-68	
973690-9	1976	In the absence of transient ischemia during occlusion (that is, rCBF greater than 18 ml/100 g/min), halothane and enflurane anesthesia were associated with significantly higher rCBF"s and lower stump pressures than was neuroleptanesthesia.	Halothane	100-109	CCAAT/enhancer binding protein zeta	Rattus norvegicus	177-181	
973690-10	1976	Pre-occlusion and post-occlusion rCBF measurements also demonstrated cerebral vasodilation by halothane and enflurane (halothane greater than enflurane) and vasoconstriction by neuroleptanesthesia.	Halothane	94-103	CCAAT/enhancer binding protein zeta	Rattus norvegicus	33-37	
973690-10	1976	Pre-occlusion and post-occlusion rCBF measurements also demonstrated cerebral vasodilation by halothane and enflurane (halothane greater than enflurane) and vasoconstriction by neuroleptanesthesia.	Halothane	119-128	CCAAT/enhancer binding protein zeta	Rattus norvegicus	33-37