PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
7116155-3	1982	These data contrast with our previous findings that physostigmine-induced theta (I theta) is specifically associated with a decrease in 2-DG uptake in the stratum lacunosum moleculare of hippocampal CA1-CA2 areas.	Physostigmine	52-65	carbonic anhydrase 1	Rattus norvegicus	199-202	
3215282-6	1988	Perfusion of physostigmine (10(-5) M) or bicuculline (10(-6) M) elicited second spikes following the population spikes in CA1 pyramidal cells.	Physostigmine	13-26	carbonic anhydrase 1	Rattus norvegicus	122-125	
3691642-8	1987	injections of oxotremorine, arecoline and physostigmine in doses that induce theta activity diminished the excitability of CA1 pyramidal cells in a dose-dependent manner, as judged by the reduction in the amplitude of the population spike and the dendritic epsp.	Physostigmine	42-55	carbonic anhydrase 1	Rattus norvegicus	123-126	
28571775-11	2017	Moreover, intra-CA1 injection of sub-threshold dose of physostigmine (0.0001mug/rat) and scopolamine (0.01mug/rat) could restore the memory acquisition deficit induced by RSD, while scopolamine could restore TSD-induced amnesia.	Physostigmine	55-68	carbonic anhydrase 1	Rattus norvegicus	16-19	
16443290-6	2006	In another series of experiments, intra-CA1 injection of physostigmine (2, 3 and 4 microg/rat) improved memory retention.	Physostigmine	57-70	carbonic anhydrase 1	Rattus norvegicus	40-43	
22909986-5	2012	Also, the result revealed that, intra-CA1/MS administration subthreshold dose of muscimol reduced improvement of memory induced by physostigmine in the MS/CA1, respectively (cross injection).	Physostigmine	131-144	carbonic anhydrase 1	Rattus norvegicus	38-41	
22909986-5	2012	Also, the result revealed that, intra-CA1/MS administration subthreshold dose of muscimol reduced improvement of memory induced by physostigmine in the MS/CA1, respectively (cross injection).	Physostigmine	131-144	carbonic anhydrase 1	Rattus norvegicus	155-158	
16191443-3	2006	Using a 3-day schedule of conditioning, it was found that intra-CA1 administration of the anticholinesterase, physostigmine (2, 4 and 8 microg/rat) significantly potentiated the morphine (0.5 mg/kg)-induced CPP.	Physostigmine	110-123	carbonic anhydrase 1	Rattus norvegicus	64-67	
16191443-5	2006	On the other hand, atropine (7 microg/rat, intra-CA1) reversed the physostigmine-induced potentiation of the morphine response.	Physostigmine	67-80	carbonic anhydrase 1	Rattus norvegicus	49-52	
11517282-4	2001	In the current experiments, elevating endogenous levels of acetylcholine in ovariectomized rats by 3 d of continuous administration of physostigmine, an acetylcholinesterase inhibitor, increased NMDA receptor binding in CA1 as measured by quantitative autoradiography.	Physostigmine	135-148	carbonic anhydrase 1	Rattus norvegicus	220-223	
11190098-1	2000	Statistical analysis of EEG spectra averaged over 10-min periods showed that inhibitor of acetylcholinesterase physostigmine induced the long-term (tens of minutes) characteristic changes in the electric activity of the dorsal hippocampus (CA1 field) and somatosensory cortex of unrestrained rats.	Physostigmine	111-124	carbonic anhydrase 1	Rattus norvegicus	240-243	
9405519-4	1997	The effects of the afferent stimulus were greatly enhanced in CA1 neurons exposed to the catalytic AChE inhibitors neostigmine, physostigmine, or 9-amino-1,2,3, 4-tetrahydro-acridine.	Physostigmine	128-141	carbonic anhydrase 1	Rattus norvegicus	62-65