PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
17046820-4	2006	We show here that the [Ca(2+)](i) oscillations stimulated by aromatic amino acids were selectively abolished by TRPC1 down-regulation using either a pool of small inhibitory RNAs (siRNAs) or two different individual siRNAs that targeted different coding regions of TRPC1.	Amino Acids, Aromatic	61-81	transient receptor potential cation channel subfamily C member 1	Homo sapiens	112-117	
17046820-4	2006	We show here that the [Ca(2+)](i) oscillations stimulated by aromatic amino acids were selectively abolished by TRPC1 down-regulation using either a pool of small inhibitory RNAs (siRNAs) or two different individual siRNAs that targeted different coding regions of TRPC1.	Amino Acids, Aromatic	61-81	transient receptor potential cation channel subfamily C member 1	Homo sapiens	265-270	
17046820-5	2006	Furthermore, [Ca(2+)](i) oscillations stimulated by aromatic amino acids were also abolished by inhibition of TRPC1 function with an antibody that binds the pore region of the channel.	Amino Acids, Aromatic	52-72	transient receptor potential cation channel subfamily C member 1	Homo sapiens	110-115	
20648625-6	2010	The intracellular calcium oscillations in response to aromatic amino acids were of transient type, that is, Ca(2+) spikes that returned to baseline levels, and required an intact actin cytoskeleton, a functional Rho, Filamin A and the ion channel TRPC1.	Amino Acids, Aromatic	54-74	transient receptor potential cation channel subfamily C member 1	Homo sapiens	247-252