PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15585841-9	2004	These data indicate that negative regulation of TCR signaling by beta1,6GlcNAc N-glycans promotes development of Th2 over Th1 responses, enhances polarization of Th2 cells, and suggests a mechanism for the increased autoimmune disease susceptibility observed in Mgat5(-/-) mice.	beta1,6glcnac	65-78	heart and neural crest derivatives expressed 2	Mus musculus	113-116	
15585841-4	2004	In this study we examined the role of beta1,6GlcNAc N-glycan expression in Th1/Th2 cytokine production and differentiation.	beta1,6glcnac	38-51	heart and neural crest derivatives expressed 2	Mus musculus	79-82	
15585841-9	2004	These data indicate that negative regulation of TCR signaling by beta1,6GlcNAc N-glycans promotes development of Th2 over Th1 responses, enhances polarization of Th2 cells, and suggests a mechanism for the increased autoimmune disease susceptibility observed in Mgat5(-/-) mice.	beta1,6glcnac	65-78	heart and neural crest derivatives expressed 2	Mus musculus	162-165