PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28326946-6	2017	In addition, dendrimer-conjugated triamcinolone acetonide administration (intrathecal) inhibited peripheral nerve injury-induced spinal cord microglial activation and the expression of pain-related genes in the spinal cord, including Nox2, IL-1beta, TNF-alpha, and IL-6.	Triamcinolone Acetonide	34-57	cytochrome b-245, beta polypeptide	Mus musculus	234-238