PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15351705-6	2004	We also found that 5-AzaC demethylated the promoter region of H19 gene, a typical methylated gene during embryonic differentiation.	Azacitidine	19-25	H19 imprinted maternally expressed transcript	Homo sapiens	62-65	
21768203-8	2012	Whereas 5-azacytidine significantly increased the mRNA expression of the epigenetically regulated gene H19, TLR2 expression was not affected.	Azacitidine	8-21	H19 imprinted maternally expressed transcript	Homo sapiens	103-106	
11597130-7	2001	Furthermore, it was demonstrated that H19 was reactivated and demethylated in the HepT1 cell line by 5-azaCytidine, in contrast to IGF2 P3, which was not demethylated or reactivated.	Azacitidine	101-114	H19 imprinted maternally expressed transcript	Homo sapiens	38-41	
28328238-2	2017	In the present study, we attempted to treat the abnormal expression and methylation status of H19 in A549 cells using valproic acid (VPA), ascorbic acid (Vc), and 5-aza-Cytidine (5-Aza).	Azacitidine	163-177	H19 imprinted maternally expressed transcript	Homo sapiens	94-97	
28328238-2	2017	In the present study, we attempted to treat the abnormal expression and methylation status of H19 in A549 cells using valproic acid (VPA), ascorbic acid (Vc), and 5-aza-Cytidine (5-Aza).	Azacitidine	179-184	H19 imprinted maternally expressed transcript	Homo sapiens	94-97	
25707431-0	2015	5-Azacytidine inhibits human rhabdomyosarcoma cell growth by downregulating insulin-like growth factor 2 expression and reactivating the H19 gene product miR-675, which negatively affects insulin-like growth factors and insulin signaling.	Azacitidine	0-13	H19 imprinted maternally expressed transcript	Homo sapiens	137-140	
25707431-5	2015	We observed that AzaC-mediated demethylation of the DMR at the IGF2-H19 locus resulted in downregulation of IGF2 and an increase in the expression of H19.	Azacitidine	17-21	H19 imprinted maternally expressed transcript	Homo sapiens	68-71	
25707431-5	2015	We observed that AzaC-mediated demethylation of the DMR at the IGF2-H19 locus resulted in downregulation of IGF2 and an increase in the expression of H19.	Azacitidine	17-21	H19 imprinted maternally expressed transcript	Homo sapiens	150-153