PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23185555-10	2012	In conclusion, cinnamaldehydes treatment at proapoptotic concentrations caused tubulin aggregation and dysegulation of cell cycle regulatory proteins cdk1 and cdc25C that contributed at least in part to arresting cells at G(2) phase, resulting in apoptotic cell death characterized by emergence of cleaved forms of caspase 3 and poly (ADP-ribose) polymerase (PARP).	cinnamaldehyde	15-30	poly(ADP-ribose) polymerase 1	Homo sapiens	359-363	
20038571-7	2011	Further studies on apoptotic mechanisms of Cin showed that it downregulated the expression of Bcl-(XL), upregulated CD95 (APO-1), p53 and Bax proteins, as well as cleaving the poly (ADP-ribose) polymerase (PARP) in a time-dependent pattern.	cinnamaldehyde	43-46	poly(ADP-ribose) polymerase 1	Homo sapiens	176-204	
20038571-7	2011	Further studies on apoptotic mechanisms of Cin showed that it downregulated the expression of Bcl-(XL), upregulated CD95 (APO-1), p53 and Bax proteins, as well as cleaving the poly (ADP-ribose) polymerase (PARP) in a time-dependent pattern.	cinnamaldehyde	43-46	poly(ADP-ribose) polymerase 1	Homo sapiens	206-210	
17673346-5	2007	Cin down-regulated the expression of Bcl-(XL), up-regulated mutant p53 and Bax proteins and promoted caspase-3 to active forms, as well as cleaving poly (ADP-ribose) polymerase (PARP) in a time-dependent pattern.	cinnamaldehyde	0-3	poly(ADP-ribose) polymerase 1	Homo sapiens	148-176	
17673346-5	2007	Cin down-regulated the expression of Bcl-(XL), up-regulated mutant p53 and Bax proteins and promoted caspase-3 to active forms, as well as cleaving poly (ADP-ribose) polymerase (PARP) in a time-dependent pattern.	cinnamaldehyde	0-3	poly(ADP-ribose) polymerase 1	Homo sapiens	178-182	
17673346-9	2007	PFTalpha effectively blocked PARP cleavage in cells treated with Cin, and also markedly prevented the phosphorylation of JNK, p38 and ERK proteins.	cinnamaldehyde	65-68	poly(ADP-ribose) polymerase 1	Homo sapiens	29-33