PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29262602-0	2017	Dihydroartemisinin inhibits EMT induced by platinum-based drugs via Akt-Snail pathway.	Platinum	43-51	snail family transcriptional repressor 1	Homo sapiens	72-77	
31086816-0	2019	USP1 links platinum resistance to cancer cell dissemination by regulating Snail stability.	Platinum	11-19	snail family transcriptional repressor 1	Homo sapiens	74-79	
31086816-3	2019	Here, we report that the ubiquitin-specific protease 1 (USP1) mediates ovarian cancer cell resistance to platinum, by regulating the stability of Snail, which, in turn, promotes tumor dissemination.	Platinum	105-113	snail family transcriptional repressor 1	Homo sapiens	146-151	
31086816-4	2019	At the molecular level, we observed that upon platinum treatment, USP1 is phosphorylated by ATM and ATR and binds to Snail.	Platinum	46-54	snail family transcriptional repressor 1	Homo sapiens	117-122	
34481526-7	2021	In addition, as a key transcription factor controlling EMT initiation, Snail was found to be degraded by PS-T induced autophagy.	Platinum	105-109	snail family transcriptional repressor 1	Homo sapiens	71-76	
34481526-8	2021	In addition, overexpression of Snail reversed the inhibitory effects of PS-T.	Platinum	72-76	snail family transcriptional repressor 1	Homo sapiens	31-36	
34481526-10	2021	CONCLUSIONS: This study demonstrated that PS-T could inhibit EMT in breast cancer cells by inducing autophagy to degrade Snail protein, thus improving the prognosis of TNBC, offering potential treatment alternatives for TNBC patients.	Platinum	42-46	snail family transcriptional repressor 1	Homo sapiens	121-126