PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9487800-3	1998	Single C-->T transitions at dipyrimidine sequences located on the nontranscribed DNA strand were the most predominant type of p53 mutation.	dipyrimidine	31-43	transformation related protein 53, pseudogene	Mus musculus	129-132	
9683186-6	1998	All of the p53 mutations occurred at dipyrimidine sequences.	dipyrimidine	37-49	transformation related protein 53, pseudogene	Mus musculus	11-14	
1423288-5	1992	Three base substitutions were located at codon 148, and all dipyrimidine-derived mutations occurred at sites where the sequence is present in the nontranscribed DNA strand, indicating some site and strand specificity of the ultraviolet B-induced p53 mutations.	dipyrimidine	60-72	transformation related protein 53, pseudogene	Mus musculus	246-249	
7970701-3	1994	Moreover, the selective removal of CPD from the transcribed strand of the p53 gene correlates well with the known strand bias of u.v.-induced mutations at dipyrimidine sites in the p53 gene of u.v.-induced mouse skin tumors.	dipyrimidine	155-167	transformation related protein 53, pseudogene	Mus musculus	74-77	
7970701-3	1994	Moreover, the selective removal of CPD from the transcribed strand of the p53 gene correlates well with the known strand bias of u.v.-induced mutations at dipyrimidine sites in the p53 gene of u.v.-induced mouse skin tumors.	dipyrimidine	155-167	transformation related protein 53, pseudogene	Mus musculus	181-184	
9457740-2	1997	Most of the mutations occurred at dipyrimidine sequences, suggesting that pyrimidine dimers in the p53 gene play a role in the pathogenesis of cutaneous squamous cell carcinoma.	dipyrimidine	34-46	transformation related protein 53, pseudogene	Mus musculus	99-102	
22844076-6	2012	Most of the p53 mutations found were G:C   A:T transitions at dipyrimidine sites in both the NB-UVB- and BB-UVB-exposed groups.	dipyrimidine	62-74	transformation related protein 53, pseudogene	Mus musculus	12-15	
22844076-8	2012	Our results strongly suggest that NB-UVB induces highly malignant tumours caused by p53 dipyrimidine mutations through the formation of CPDs.	dipyrimidine	88-100	transformation related protein 53, pseudogene	Mus musculus	84-87