PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19079715-0	2008	G-protein-coupled receptor 30 and estrogen receptor-alpha are involved in the proliferative effects induced by atrazine in ovarian cancer cells.	Atrazine	111-119	G protein-coupled estrogen receptor 1	Homo sapiens	0-29	
19079715-3	2008	OBJECTIVES: Given the ability of atrazine to exert estrogen-like activity in cancer cells, we evaluated the potential of atrazine to signal through GPR30 in stimulating biological responses in cancer cells.	Atrazine	121-129	G protein-coupled estrogen receptor 1	Homo sapiens	148-153	
19079715-7	2008	Using specific signaling inhibitors and gene silencing, we demonstrated that atrazine stimulated the proliferation of ovarian cancer cells through the GPR30-epidermal growth factor receptor transduction pathway and the involvement of ERalpha.	Atrazine	77-85	G protein-coupled estrogen receptor 1	Homo sapiens	151-156	
19079715-9	2008	On the basis of the present data, atrazine should be included among the environmental contaminants potentially able to signal via GPR30 in eliciting estrogenic action.	Atrazine	34-42	G protein-coupled estrogen receptor 1	Homo sapiens	130-135	
26955487-2	2016	GPR30, a G protein coupled receptor, was identified as a potential orphan receptor that may interact with triazine herbicides such as atrazine, one of the most commonly utilized chlorotriazines in agricultural practices in the United States.	Atrazine	134-142	G protein-coupled estrogen receptor 1	Homo sapiens	0-5	
25616260-3	2015	OBJECTIVES: We aimed to evaluate the potential of atrazine to trigger GPER-mediated signaling in cancer cells and cancer-associated fibroblasts (CAFs).	Atrazine	50-58	G protein-coupled estrogen receptor 1	Homo sapiens	70-74	
25616260-5	2015	Conversely, atrazine was able to bind to GPER to induce ERK activation and the expression of estrogen target genes, which, interestingly, appeared to rely on both GPER and ERalpha expression.	Atrazine	12-20	G protein-coupled estrogen receptor 1	Homo sapiens	41-45	
25616260-5	2015	Conversely, atrazine was able to bind to GPER to induce ERK activation and the expression of estrogen target genes, which, interestingly, appeared to rely on both GPER and ERalpha expression.	Atrazine	12-20	G protein-coupled estrogen receptor 1	Homo sapiens	163-167	
25616260-6	2015	As a biological counterpart, atrazine stimulated the proliferation of ovarian cancer cells that depend on GPER and ERalpha, as evidenced by gene silencing experiments and the use of specific signaling inhibitors.	Atrazine	29-37	G protein-coupled estrogen receptor 1	Homo sapiens	106-110	
25616260-7	2015	Of note, through GPER, atrazine elicited ERK phosphorylation, gene expression, and migration in CAFs, thus extending its stimulatory role to these main players of the tumor microenvironment.	Atrazine	23-31	G protein-coupled estrogen receptor 1	Homo sapiens	17-21	
25616260-9	2015	On the basis of our data, atrazine should be included among the environmental contaminants that may elicit estrogenic activity through GPER-mediated signaling.	Atrazine	26-34	G protein-coupled estrogen receptor 1	Homo sapiens	135-139	
21660819-11	2011	Atrazine does not appear to interact strongly with estrogen receptors alpha or beta but may interact with putative estrogen receptor GPR30 (G-protein-coupled receptor).	Atrazine	0-8	G protein-coupled estrogen receptor 1	Homo sapiens	133-138	
24486480-0	2014	Retraction: G-protein-coupled receptor 30 and estrogen receptor-alpha are involved in the proliferative effects induced by atrazine in ovarian cancer cells.	Atrazine	123-131	G protein-coupled estrogen receptor 1	Homo sapiens	12-41