PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
1336929-3	1992	Exposure to 20 mM NH4Cl immediately alkalinized pHi: replacement with a Na(+)-rich solution acidified pHi before recovery to resting levels.	Ammonium Chloride	18-23	glucose-6-phosphate isomerase	Bos taurus	48-51	
7598914-9	1995	Following an NH4Cl-induced alkaline load, the pHi of both control- and heparin-treated sperm recovered toward the resting pHi with a half-time of recovery of 1.5-1.7 min.	Ammonium Chloride	13-18	glucose-6-phosphate isomerase	Bos taurus	122-125	
9657902-6	1998	Following acid loading in NaCl Ringers with a 20 mm NH4Cl prepulse, pHi recovery was partially inhibited by exposure to either Na-free (NMGCl) Ringers, 100 microM DMA or 20 microM bafilomycin A1.	Ammonium Chloride	52-57	glucose-6-phosphate isomerase	Bos taurus	68-71	
7598914-9	1995	Following an NH4Cl-induced alkaline load, the pHi of both control- and heparin-treated sperm recovered toward the resting pHi with a half-time of recovery of 1.5-1.7 min.	Ammonium Chloride	13-18	glucose-6-phosphate isomerase	Bos taurus	46-49	
8057082-5	1994	In cell-attached patches pHi was changed by extracellular NH4Cl or through the opened cell.	Ammonium Chloride	58-63	glucose-6-phosphate isomerase	Bos taurus	25-28	
1336929-3	1992	Exposure to 20 mM NH4Cl immediately alkalinized pHi: replacement with a Na(+)-rich solution acidified pHi before recovery to resting levels.	Ammonium Chloride	18-23	glucose-6-phosphate isomerase	Bos taurus	102-105	
1336929-4	1992	When NH4Cl was replaced by a low Na+ solution, acidification was sustained but pHi recovery occurred after Na(+)-rich solution.	Ammonium Chloride	5-10	glucose-6-phosphate isomerase	Bos taurus	79-82	
1906784-1	1991	The effects of NH4Cl on cytoplasmic free calcium concentration ([Ca2+]i) and pH (pHi) in single bovine anterior pituitary cells were determined using fluorescence imaging microscopy.	Ammonium Chloride	15-20	glucose-6-phosphate isomerase	Bos taurus	81-84	
1909500-7	1991	However, increasing pHi by the external application of NH4Cl at physiological pHo caused a rapid decline and not an increase in the K(Ca2+) channel activity triggered by BK.	Ammonium Chloride	55-60	glucose-6-phosphate isomerase	Bos taurus	20-23	
1906784-4	1991	NH4Cl also increased pHi and this, like the [Ca2+]i plateau, remained elevated during the continued presence of NH4Cl.	Ammonium Chloride	0-5	glucose-6-phosphate isomerase	Bos taurus	21-24	
3184169-3	1988	Following an acid load imposed by a NH4Cl prepulse, pHi was regulated in the absence of HCO3- by a Na+-dependent process inhibitable to a large extent by 1 mM amiloride and 0.1 mM dimethylamiloride.	Ammonium Chloride	36-41	glucose-6-phosphate isomerase	Bos taurus	52-55	
2172243-4	1990	NH4Cl-induced, dose-dependent (5-20 mM) increases in [Ca2+]i (maximum change = 195 +/- 26 nM) which were temporally similar to the NH4Cl-induced pHi increases.	Ammonium Chloride	0-5	glucose-6-phosphate isomerase	Bos taurus	145-148	
2172243-4	1990	NH4Cl-induced, dose-dependent (5-20 mM) increases in [Ca2+]i (maximum change = 195 +/- 26 nM) which were temporally similar to the NH4Cl-induced pHi increases.	Ammonium Chloride	131-136	glucose-6-phosphate isomerase	Bos taurus	145-148	
2910860-9	1989	Measurement of cytosolic pH (pHi) with the fluorescent probe 2",7"-bis-carboxyethyl-5(6)-carboxyfluorescein (BCECF) revealed an alkalinization by NH4Cl (2.5-25 mM) of 0.1-0.23 pH units and acidification by sodium propionate (10-20 mM) of 0.2-0.25 pH units, with intermediate combined effects.	Ammonium Chloride	146-151	glucose-6-phosphate isomerase	Bos taurus	29-32	
3370221-5	1988	Elevating pHi with NH4Cl was shown to inhibit secretion from chromaffin cells.	Ammonium Chloride	19-24	glucose-6-phosphate isomerase	Bos taurus	10-13	
2841075-8	1988	After intracellular acidification, induced by NH4Cl-prepulse technique, pHi was regulated back towards normal steady state pHi.	Ammonium Chloride	46-51	glucose-6-phosphate isomerase	Bos taurus	72-75	
2841075-8	1988	After intracellular acidification, induced by NH4Cl-prepulse technique, pHi was regulated back towards normal steady state pHi.	Ammonium Chloride	46-51	glucose-6-phosphate isomerase	Bos taurus	123-126	
2832823-3	1988	(1) Regulation of pHi after induction of an acid load by removal of NH4Cl could be blocked either totally by removal of extracellular sodium, or subtotally (about 90%) by application of amiloride (1 mmol/l).	Ammonium Chloride	68-73	glucose-6-phosphate isomerase	Bos taurus	18-21	
32818510-6	2020	Increasing pHi with NH4Cl also increased lactate production in BF or BR, indicating that the increased lactate production in BR is not due to HCO3- itself.	Ammonium Chloride	20-25	glucose-6-phosphate isomerase	Bos taurus	11-14