PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9309155-1	1997	Azatyrosine is known to convert ras, raf or c-erbB-2-transformed NIH3T3 cells to a normal phenotype.	beta-(5-hydroxy-2-pyridyl)alanine	0-11	erb-b2 receptor tyrosine kinase 2	Mus musculus	44-52	
9309155-2	1997	We attempted to identify the signal-transduction process triggered by oncogenic c-ErbB-2 that was inhibited by azatyrosine.	beta-(5-hydroxy-2-pyridyl)alanine	111-122	erb-b2 receptor tyrosine kinase 2	Mus musculus	80-88	
9309155-5	1997	Furthermore, azatyrosine inhibited activation of the 12-O-tetradecanoylphorbol-13-acetate (TPA) response element in response to stimulation by oncogenic c-ErbB-2.	beta-(5-hydroxy-2-pyridyl)alanine	13-24	erb-b2 receptor tyrosine kinase 2	Mus musculus	153-161	
21594311-2	1996	We report herein that azatyrosine also inhibits the growth of c-erbB-2-transformed cells and induces normal morphology.	beta-(5-hydroxy-2-pyridyl)alanine	22-33	erb-b2 receptor tyrosine kinase 2	Mus musculus	62-70	
21594424-1	1996	We reported recently that azatyrosine inhibits the growth of c-Ha-ras, c-raf or c-erbB-2-transformed NIH3T3 cells and converts the transformed cells to cells with a normal phenotype.	beta-(5-hydroxy-2-pyridyl)alanine	26-37	erb-b2 receptor tyrosine kinase 2	Mus musculus	80-88	
21594311-8	1996	These results suggest that azatyrosine acts downstream of Ras in signal transduction from oncogenic c-ErbB-2 to nuclear factors.	beta-(5-hydroxy-2-pyridyl)alanine	27-38	erb-b2 receptor tyrosine kinase 2	Mus musculus	100-108	
21594314-0	1996	Stimulation of the expression of rhoB contributes to the ability of azatyrosine to convert c-erbB-2-transformed cells to a normal morphology.	beta-(5-hydroxy-2-pyridyl)alanine	68-79	erb-b2 receptor tyrosine kinase 2	Mus musculus	91-99	
21594314-1	1996	We have reported recently that azatyrosine inhibits the growth of ras-, raf-, or c-erbB-2 transformed NIH3T3 cells and converts the transformed cells to cells with normal morphology.	beta-(5-hydroxy-2-pyridyl)alanine	31-42	erb-b2 receptor tyrosine kinase 2	Mus musculus	81-89	
21594314-7	1996	These results suggest that expression of rhoB is involved in the conversion of c-erbB-2-transformed cells to a normal phenotype that is induced by azatyrosine, but such expression is insufficient by itself for complete reversion.	beta-(5-hydroxy-2-pyridyl)alanine	147-158	erb-b2 receptor tyrosine kinase 2	Mus musculus	79-87	
21594311-3	1996	We attempted to identify the signal-transduction process triggered by oncogenic c-ErbB-2 that was inhibited by azatyrosine.	beta-(5-hydroxy-2-pyridyl)alanine	111-122	erb-b2 receptor tyrosine kinase 2	Mus musculus	80-88	
21594311-6	1996	However, azatyrosine inhibited increases in phosphorylation of c-Raf-1 and c-Jun induced by oncogenic c-ErbB-2.	beta-(5-hydroxy-2-pyridyl)alanine	9-20	erb-b2 receptor tyrosine kinase 2	Mus musculus	102-110	
21594311-7	1996	Furthermore, azatyrosine inhibited activation of the 12-O-tetradecanoylphorbol-13-acetate (TPA) response element in response to stimulation by oncogenic c-ErbB-2.	beta-(5-hydroxy-2-pyridyl)alanine	13-24	erb-b2 receptor tyrosine kinase 2	Mus musculus	153-161	
1328988-1	1992	An antibiotic, azatyrosine [L-beta-(5-hydroxy-2-pyridyl) alanine], specifically converts ras-, raf- or c-erbB2 (neu)-transformed NIH3T3 cells to apparently normal phenotype.	beta-(5-hydroxy-2-pyridyl)alanine	15-26	erb-b2 receptor tyrosine kinase 2	Mus musculus	103-110	
1328988-1	1992	An antibiotic, azatyrosine [L-beta-(5-hydroxy-2-pyridyl) alanine], specifically converts ras-, raf- or c-erbB2 (neu)-transformed NIH3T3 cells to apparently normal phenotype.	beta-(5-hydroxy-2-pyridyl)alanine	15-26	erb-b2 receptor tyrosine kinase 2	Mus musculus	112-115