PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27011246-0	2016	[New perspective on the role of WNK1 and WNK4 in the regulation of NaCl reabsorption and K(+) secretion by the distal nephron].	Sodium Chloride	67-71	WNK lysine deficient protein kinase 1	Homo sapiens	32-36	
18591455-8	2008	In combination, the same variants (ADD1 Trp/WNK1 GG/Nedd4L GA+AA) showed a consistent effect on renal Na handling (P=0.009) and acute BP response to a saline infusion (P=0.021), BP lowering after thiazide treatment (P=0.008), and nocturnal systolic BP (P=0.044).	Sodium Chloride	151-157	WNK lysine deficient protein kinase 1	Homo sapiens	44-48	
16820787-4	2006	In this mini review, we discuss WNK1 and WNK4 gene products and their regulatory effects on sodium chloride and potassium handling in the aldosterone-sensitive distal nephron.	Sodium Chloride	92-107	WNK lysine deficient protein kinase 1	Homo sapiens	32-36	
15980941-1	2005	Several monogenic hypertensive disorders are caused by genetic mutations leading to the deranged function and/or regulation of renal tubular NaCl transport, such as mutations of the renal epithelial Na+ channel (ENaC) in Liddle syndrome, of the kinase WNK1 (with no K) in Gordon syndrome, and of the mineralocorticoid receptor, or of 11beta-hydroxysteroid dehydrogenase.	Sodium Chloride	141-145	WNK lysine deficient protein kinase 1	Homo sapiens	252-256