PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15107607-6	2004	Further, high NaCl inhibits the activity of key components of the classical DNA damage response such as Mre11, chk1 and H2AX.	Sodium Chloride	14-18	H2A.X variant histone	Mus musculus	120-124	
22106305-4	2011	Repair of the breaks after NaCl is reduced is accompanied by formation of foci containing phosphorylated H2AX (gammaH2AX), which occurs around DNA double-strand breaks and contributes to their repair.	Sodium Chloride	27-31	H2A.X variant histone	Mus musculus	105-109	
22106305-4	2011	Repair of the breaks after NaCl is reduced is accompanied by formation of foci containing phosphorylated H2AX (gammaH2AX), which occurs around DNA double-strand breaks and contributes to their repair.	Sodium Chloride	27-31	H2A.X variant histone	Mus musculus	111-120	
22106305-7	2011	By chromatin immunoprecipitation using anti-gammaH2AX antibody, followed by massive parallel sequencing (ChIP-Seq), we find that during repair of double-strand breaks induced by high NaCl, gammaH2AX is predominantly localized to regions of the genome devoid of genes ("gene deserts"), indicating that the high NaCl-induced double-strand breaks are located there.	Sodium Chloride	183-187	H2A.X variant histone	Mus musculus	44-53	
22106305-7	2011	By chromatin immunoprecipitation using anti-gammaH2AX antibody, followed by massive parallel sequencing (ChIP-Seq), we find that during repair of double-strand breaks induced by high NaCl, gammaH2AX is predominantly localized to regions of the genome devoid of genes ("gene deserts"), indicating that the high NaCl-induced double-strand breaks are located there.	Sodium Chloride	183-187	H2A.X variant histone	Mus musculus	189-198	
22106305-7	2011	By chromatin immunoprecipitation using anti-gammaH2AX antibody, followed by massive parallel sequencing (ChIP-Seq), we find that during repair of double-strand breaks induced by high NaCl, gammaH2AX is predominantly localized to regions of the genome devoid of genes ("gene deserts"), indicating that the high NaCl-induced double-strand breaks are located there.	Sodium Chloride	310-314	H2A.X variant histone	Mus musculus	44-53	
22106305-7	2011	By chromatin immunoprecipitation using anti-gammaH2AX antibody, followed by massive parallel sequencing (ChIP-Seq), we find that during repair of double-strand breaks induced by high NaCl, gammaH2AX is predominantly localized to regions of the genome devoid of genes ("gene deserts"), indicating that the high NaCl-induced double-strand breaks are located there.	Sodium Chloride	310-314	H2A.X variant histone	Mus musculus	189-198