PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
17192185-0	2006	Up-regulation of HLA class-I antigen expression and antigen-specific CTL response in cervical cancer cells by the demethylating agent hydralazine and the histone deacetylase inhibitor valproic acid.	Hydralazine	134-145	MHC class I polypeptide-related sequence A	Homo sapiens	17-36	
22576685-5	2012	Both VPA and hydralazine increased the expression of cell-surface MICA and B in osteosarcoma cells, and their combination induced a greater increase in their expression.	Hydralazine	13-24	MHC class I polypeptide-related sequence A	Homo sapiens	66-70	
21805029-6	2011	The results show that hydralazine and valproic acid not only increase the expression of MICA and MICB ligands of target cells, but also reduce their shedding to the supernatant.	Hydralazine	22-33	MHC class I polypeptide-related sequence A	Homo sapiens	88-92	
21805029-9	2011	In conclusion, our results demonstrate the ability of hydralazine and valproate to increase the NK activity against epithelial cancer cell lines and suggest that these drugs could reduce the levels of soluble MICA and MICB helping in avoiding tumor-induced suppression of NK cytotoxicity against the tumor.	Hydralazine	54-65	MHC class I polypeptide-related sequence A	Homo sapiens	209-213