PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
20346917-0	2010	Curcumin dramatically enhances retinoic acid-induced superoxide generating activity via accumulation of p47-phox and p67-phox proteins in U937 cells.	Tretinoin	31-44	CD33 molecule	Homo sapiens	117-120	
20074641-4	2010	Retinoic acid induces neuronal differentiation as revealed by the increased expression of MAP2, the decreased cell doubling rate, and the gain in neuronal morphological features and these events are accompanied by the increased expression level of PKC delta and p67(phox), one of the components of NADPH oxidase.	Tretinoin	0-13	CD33 molecule	Homo sapiens	262-265	
20074641-6	2010	Moreover, using rottlerin to inhibit PKC delta or transfection experiments to overexpress it, we show that retinoic acid acts through this enzyme to induce MAP2 expression and to increase p67(phox) membrane translocation leading to NADPH oxidase activation.	Tretinoin	107-120	CD33 molecule	Homo sapiens	188-191	
24406248-8	2014	Mechanistically, ATRA caused the generation of reactive oxygen species (ROS) through increased expression of NADPH oxidase subunits (p47(phox) and p67(phox)) to facilitate ATRA-induced GSK-3beta activation and cell apoptosis.	Tretinoin	17-21	CD33 molecule	Homo sapiens	147-150	
24406248-8	2014	Mechanistically, ATRA caused the generation of reactive oxygen species (ROS) through increased expression of NADPH oxidase subunits (p47(phox) and p67(phox)) to facilitate ATRA-induced GSK-3beta activation and cell apoptosis.	Tretinoin	172-176	CD33 molecule	Homo sapiens	147-150	
22525672-1	2012	F9 embryonic carcinoma (EC) cells undergo extra-embryonic endodermal (ExE) differentiation in response to retinoic acid (RA) treatment, which induces the expression of two isoforms (p96 and p67) of the adaptor protein, Disabled-2 (Dab2).	Tretinoin	106-119	CD33 molecule	Homo sapiens	190-193	
22525672-1	2012	F9 embryonic carcinoma (EC) cells undergo extra-embryonic endodermal (ExE) differentiation in response to retinoic acid (RA) treatment, which induces the expression of two isoforms (p96 and p67) of the adaptor protein, Disabled-2 (Dab2).	Tretinoin	121-123	CD33 molecule	Homo sapiens	190-193	
20346917-6	2010	On the other hand, immunoblot analysis revealed that co-treatment with RA and curcumin caused remarkable accumulation of protein levels of p47-phox (to 7-fold) and p67-phox (to 4-fold) compared with those of the RA-treatment alone.	Tretinoin	71-73	CD33 molecule	Homo sapiens	164-167	
20346917-7	2010	These results suggested that curcumin dramatically enhances RA-induced O(2)(-)-generating activity via accumulation of cytosolic p47-phox and p67-phox proteins in U937 cells.	Tretinoin	60-62	CD33 molecule	Homo sapiens	142-145	
16403261-3	2005	By treatment of 1 micromol/L ATRA, cell differentiation antigen CD11b was gradually increased ([chi = 47.002, P = 0.000) and CD33 was gradually decreased (chi = 1.614, P = 0.806) with time.	Tretinoin	29-33	CD33 molecule	Homo sapiens	125-129	
12627849-6	2003	Cell surface expression of the chemotactic peptide receptors CD33 and CD10 but not of CD11b and CD11c was up-regulated by ATRA plus G-CSF far more profoundly than by ATRA alone.	Tretinoin	122-126	CD33 molecule	Homo sapiens	61-65	
2167670-6	1990	Cytosol from monocytes cultured for 6 days is immunochemically deficient in p47 but not p67, while cytosol from HL-60 cells induced with retinoic acid for 3 days is deficient in p67 but not p47.	Tretinoin	137-150	CD33 molecule	Homo sapiens	178-181