PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34784434-2	2021	Previous studies have postulated that retinoic acid (RA), an active metabolite of vitamin A and high-affinity ligand for RA receptor (RAR), is reduced in airway inflammatory condition and contributes to multiple features of asthma including airway hyperresponsiveness and excessive accumulation of airway smooth muscle (ASM) cells.	Tretinoin	38-51	H19 imprinted maternally expressed transcript	Homo sapiens	320-323	
34784434-7	2021	Further, basal RA levels and RA biosynthetic capabilities were decreased in asthmatic human ASM cells.	Tretinoin	15-17	H19 imprinted maternally expressed transcript	Homo sapiens	92-95	
9720909-0	1998	The effect of retinoic acid on the activation of the human H19 promoter by a 3" downstream region.	Tretinoin	14-27	H19 imprinted maternally expressed transcript	Homo sapiens	59-62	
9720909-5	1998	Moreover, the activation of the H19 promoter by retinoic acid in cells derived from human testicular germ cell tumors is dependent upon the 3" downstream region.	Tretinoin	48-61	H19 imprinted maternally expressed transcript	Homo sapiens	32-35	
9720909-6	1998	The possibility that the action of retinoic acid on the H19 promoter is an indirect one and involves a member of the AP2 transcription factor family is discussed.	Tretinoin	35-48	H19 imprinted maternally expressed transcript	Homo sapiens	56-59	
34784434-2	2021	Previous studies have postulated that retinoic acid (RA), an active metabolite of vitamin A and high-affinity ligand for RA receptor (RAR), is reduced in airway inflammatory condition and contributes to multiple features of asthma including airway hyperresponsiveness and excessive accumulation of airway smooth muscle (ASM) cells.	Tretinoin	53-55	H19 imprinted maternally expressed transcript	Homo sapiens	320-323	
34784434-7	2021	Further, basal RA levels and RA biosynthetic capabilities were decreased in asthmatic human ASM cells.	Tretinoin	29-31	H19 imprinted maternally expressed transcript	Homo sapiens	92-95	
34784434-8	2021	Treatment of human ASM cells with all-trans RA (ATRA) or the RARgamma-specific agonist (CD1530) resulted in the inhibition of mitogen-induced cell proliferation and AP-1-dependent transcription.	Tretinoin	44-46	H19 imprinted maternally expressed transcript	Homo sapiens	19-22	
34784434-8	2021	Treatment of human ASM cells with all-trans RA (ATRA) or the RARgamma-specific agonist (CD1530) resulted in the inhibition of mitogen-induced cell proliferation and AP-1-dependent transcription.	Tretinoin	48-52	H19 imprinted maternally expressed transcript	Homo sapiens	19-22