PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31264311-2	2019	Our previous experiments suggested that reactive oxygen species (ROS) can activate Mad2, a key protein in the spindle assembly checkpoint (SAC), and delay the first mitotic, providing time to prevent the formation of embryonic aneuploidy.	Reactive Oxygen Species	40-63	MAD2 mitotic arrest deficient-like 1	Mus musculus	83-87	
33634101-9	2020	Additionally, the appearance of gammaH2AX and the significantly increased expression of Aurora B and MAD2L1 suggested that administration of relatively high doses of eCG caused Aurora B-mediated SAC activation triggered by ROS-induced DNA damage in early mouse IVF-derived embryos for self-correction of aneuploidy formation.	Reactive Oxygen Species	223-226	MAD2 mitotic arrest deficient-like 1	Mus musculus	101-107	
31264311-2	2019	Our previous experiments suggested that reactive oxygen species (ROS) can activate Mad2, a key protein in the spindle assembly checkpoint (SAC), and delay the first mitotic, providing time to prevent the formation of embryonic aneuploidy.	Reactive Oxygen Species	65-68	MAD2 mitotic arrest deficient-like 1	Mus musculus	83-87