PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 31264311-2 2019 Our previous experiments suggested that reactive oxygen species (ROS) can activate Mad2, a key protein in the spindle assembly checkpoint (SAC), and delay the first mitotic, providing time to prevent the formation of embryonic aneuploidy. Reactive Oxygen Species 40-63 MAD2 mitotic arrest deficient-like 1 Mus musculus 83-87 33634101-9 2020 Additionally, the appearance of gammaH2AX and the significantly increased expression of Aurora B and MAD2L1 suggested that administration of relatively high doses of eCG caused Aurora B-mediated SAC activation triggered by ROS-induced DNA damage in early mouse IVF-derived embryos for self-correction of aneuploidy formation. Reactive Oxygen Species 223-226 MAD2 mitotic arrest deficient-like 1 Mus musculus 101-107 31264311-2 2019 Our previous experiments suggested that reactive oxygen species (ROS) can activate Mad2, a key protein in the spindle assembly checkpoint (SAC), and delay the first mitotic, providing time to prevent the formation of embryonic aneuploidy. Reactive Oxygen Species 65-68 MAD2 mitotic arrest deficient-like 1 Mus musculus 83-87