PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23052189-0	2012	Interleukin-24 suppresses the growth of vascular smooth muscle cells by inhibiting H(2)O(2)-induced reactive oxygen species production.	Reactive Oxygen Species	100-123	interleukin 24	Mus musculus	0-14	
23052189-5	2012	To elucidate the effect of IL-24 on ROS-induced signaling, Western blot analysis was employed.	Reactive Oxygen Species	36-39	interleukin 24	Mus musculus	27-32	
23052189-8	2012	Interestingly, IL-24 attenuated the H(2)O(2)-induced ROS production by reducing the mitochondrial H(2)O(2) production and enhancing the expression of antioxidant enzymes.	Reactive Oxygen Species	53-56	interleukin 24	Mus musculus	15-20	
23052189-10	2012	CONCLUSION: These findings suggest a novel mechanism in which IL-24 suppresses the growth of normal VSMCs by inhibiting H(2)O(2)-induced ROS production through the regulation of mitochondrial ROS production and expression of antioxidant enzymes.	Reactive Oxygen Species	137-140	interleukin 24	Mus musculus	62-67	
23052189-10	2012	CONCLUSION: These findings suggest a novel mechanism in which IL-24 suppresses the growth of normal VSMCs by inhibiting H(2)O(2)-induced ROS production through the regulation of mitochondrial ROS production and expression of antioxidant enzymes.	Reactive Oxygen Species	192-195	interleukin 24	Mus musculus	62-67	
23052189-3	2012	However, the role of IL-24 in ROS-induced VSMC growth has not yet been investigated.	Reactive Oxygen Species	30-33	interleukin 24	Mus musculus	21-26	
15580305-0	2005	Induction of reactive oxygen species renders mutant and wild-type K-ras pancreatic carcinoma cells susceptible to Ad.mda-7-induced apoptosis.	Reactive Oxygen Species	13-36	interleukin 24	Mus musculus	117-122	
15580305-10	2005	Analysis of signal transduction changes in pancreatic carcinoma cells infected with Ad.mda-7 in combination with a ROS-inducer indicate that cell death correlates with modulation of discrete cassettes of multiple signaling pathways in a pancreatic cancer cell-specific manner, supporting global signaling dysregulation as a potential mediator of apoptosis induction.	Reactive Oxygen Species	115-118	interleukin 24	Mus musculus	87-92	
25103017-7	2015	RESULTS: The Ki67-ZD55-IL-24-treated group generated much more reactive oxygen species than the untreated group.	Reactive Oxygen Species	63-86	interleukin 24	Mus musculus	23-28