PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9659588-3	1998	In a separate tumorigenesis model, both decaffeinated black tea and decaffeinated green tea inhibited 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK)-induced lung tumor formation.	4-(N-methyl-N-nitrosamino)-1-(3-pyridyl)-1-butanone	102-148	solute carrier family 7 (cationic amino acid transporter, y+ system), member 2	Mus musculus	60-63	
10202397-2	1999	We have previously shown that green tea, administered as drinking water, inhibits lung tumor development in A/J mice treated with 4-(methylnitrosamino)-1-(3-pyridyl)-l-butanone (NNK), a potent nicotine-derived lung carcinogen found in tobacco.	4-(N-methyl-N-nitrosamino)-1-(3-pyridyl)-1-butanone	178-181	solute carrier family 7 (cationic amino acid transporter, y+ system), member 2	Mus musculus	36-39	
9659588-3	1998	In a separate tumorigenesis model, both decaffeinated black tea and decaffeinated green tea inhibited 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK)-induced lung tumor formation.	4-(N-methyl-N-nitrosamino)-1-(3-pyridyl)-1-butanone	150-153	solute carrier family 7 (cationic amino acid transporter, y+ system), member 2	Mus musculus	60-63