PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 30743782-9 2008 Treatment with vasopressin analogues is almost always effective in controlling renal water excretion and normalizing plasma sodium concentrations. Sodium 124-130 arginine vasopressin Homo sapiens 15-26 17663307-1 2007 Hyponatraemia often complicates the treatment of underlying conditions in patients who are seriously ill. Arginine vasopressin receptor antagonists block the action of arginine vasopressin and correct sodium and water imbalance in patients with euvolaemic or hypervolaemic hyponatraemia. Sodium 201-207 arginine vasopressin Homo sapiens 115-126 18221617-3 2007 There are conclusive data that vasopressin antagonists improve or normalize serum sodium in this patient population. Sodium 82-88 arginine vasopressin Homo sapiens 31-42 17712057-3 2007 Arginine vasopressin-receptor antagonists, a novel class of agents that block the action of arginine vasopressin on V2 receptors in the renal collecting ducts, may provide specific correction of sodium and water imbalance in hyponatremia by promoting free water clearance while sparing electrolytes (aquaresis). Sodium 195-201 arginine vasopressin Homo sapiens 9-20 17712057-3 2007 Arginine vasopressin-receptor antagonists, a novel class of agents that block the action of arginine vasopressin on V2 receptors in the renal collecting ducts, may provide specific correction of sodium and water imbalance in hyponatremia by promoting free water clearance while sparing electrolytes (aquaresis). Sodium 195-201 arginine vasopressin Homo sapiens 101-112 17292670-3 2007 Arginine vasopressin (AVP) is etiologically critical for hyponatremia, and it has been proven that AVP receptor (AVP-R) antagonists normalize serum sodium levels in hyponatremic patients. Sodium 148-154 arginine vasopressin Homo sapiens 0-20 17356196-1 2007 Hyponatremia, defined as a serum sodium concentration ([Na+]) less than 135 mEq/L, is commonly caused by elevated levels of the hormone arginine vasopressin (AVP), which causes water retention. Sodium 33-39 arginine vasopressin Homo sapiens 145-156 17699429-2 2007 Vasopressin administration has been shown in healthy humans to delay sodium excretion along with its antidiuretic action. Sodium 69-75 arginine vasopressin Homo sapiens 0-11 17664863-1 2007 BACKGROUND: Most cases of hyponatremia--serum sodium concentration ([Na+]) < 135 mEq/l (< 135 mM)--are associated with an elevated plasma arginine vasopressin level. Sodium 46-52 arginine vasopressin Homo sapiens 153-164 17305665-4 2007 The present case report describes the mechanism of severe hyponatraemia in a patient taking deamino arginine vasopressin, and the subsequent development of both central pontine and extrapontine myelinolysis after rapid correction of sodium levels. Sodium 233-239 arginine vasopressin Homo sapiens 109-120 16970147-7 2006 New agents that antagonize arginine vasopressin at the V2 receptor or both the V(1A) and V2 receptors show promise for treating hypervolemic and euvolemic hyponatremia, as they induce desired free water diuresis without inducing sodium excretion. Sodium 229-235 arginine vasopressin Homo sapiens 36-47 16837788-2 2006 Serum osmolality and sodium concentration [Na+] are regulated by thirst, the hormone arginine vasopressin (AVP), and renal water and sodium handling. Sodium 21-27 arginine vasopressin Homo sapiens 94-105 16786909-3 2006 Selective antagonism of the vasopressin 2 (V2) receptor may facilitate a safe diuresis and normalize low serum sodium levels, as demonstrated in preliminary clinical trials. Sodium 111-117 arginine vasopressin Homo sapiens 28-39 16198010-8 2005 AVP injection into the LPO increased sodium renal excretion, but this was reduced by prior AAVP administration. Sodium 37-43 arginine vasopressin Homo sapiens 0-3 16198010-0 2005 Interaction between arginine vasopressin and angiotensin II receptors in the central regulation of sodium balance. Sodium 99-105 arginine vasopressin Homo sapiens 29-69 15914973-10 2005 The treatment of hyponatremia may be facilitated by emerging therapies that block the actions of arginine vasopressin at V2 and V1a receptors to promote aquaresis, the electrolyte-sparing elimination of free water, and elevate serum sodium concentrations. Sodium 233-239 arginine vasopressin Homo sapiens 106-117 16198010-1 2005 We speculated that the influence of lateral preoptic area (LPO) in sodium balance, involves arginine8-vasopressin (AVP) and angiotensin (ANG II) on Na+ uptake in LPO. Sodium 67-73 arginine vasopressin Homo sapiens 92-113 16198010-1 2005 We speculated that the influence of lateral preoptic area (LPO) in sodium balance, involves arginine8-vasopressin (AVP) and angiotensin (ANG II) on Na+ uptake in LPO. Sodium 67-73 arginine vasopressin Homo sapiens 115-118 15888562-9 2005 The possibility that an inappropriate stimulation of ENaC by vasopressin might lead to significant sodium retention in chronic situations remains to be determined. Sodium 99-105 arginine vasopressin Homo sapiens 61-72 15893443-2 2005 Are reported and analyzed molecular mechanisms about sodium retention in collecting duct cells regarding activation and surface expression of epithelial sodium channels (ENaC) and sodium-potassium-ATPase (Na,K-ATPase) by aldosterone, vasopressin, natriuretic peptide system (underfill theory): is necessary a better understanding about the dysregulation of ENaC and Na,K-ATPase surface expression and the resistance to natriuretic peptide system. Sodium 53-59 arginine vasopressin Homo sapiens 234-245 15675318-1 2005 Clinical and laboratory experiments have demonstrated that arginine vasopressin (AVP), renin-aldosterone system and catecholamines play a crucial role in water and sodium retention in edematous diseases, including congestive heart failure, nephrotic syndrome and liver cirrhosis. Sodium 164-170 arginine vasopressin Homo sapiens 68-79 15675318-1 2005 Clinical and laboratory experiments have demonstrated that arginine vasopressin (AVP), renin-aldosterone system and catecholamines play a crucial role in water and sodium retention in edematous diseases, including congestive heart failure, nephrotic syndrome and liver cirrhosis. Sodium 164-170 arginine vasopressin Homo sapiens 81-84 14607044-7 2003 The patient responded well to nasal-spray-administered deamino D arginine vasopressin and increased intravenous fluid intake, with resolution of symptoms and gradual normalization of serum sodium levels. Sodium 189-195 arginine vasopressin Homo sapiens 74-85 15098812-12 2004 Careful monitoring of water and sodium balance is warranted in all patients treated with vasopressin for septic shock. Sodium 32-38 arginine vasopressin Homo sapiens 89-100 15593031-0 2004 Central role of vasopressin in sodium/water retention in hypo- and hypervolemic nephrotic patients: a unifying hypothesis. Sodium 31-37 arginine vasopressin Homo sapiens 16-27 15593031-4 2004 In the transition, a central role is played by vasopressin, which is secreted in the two phases, respectively, by a volume and an osmotic stimulus; therefore, persistent sodium/water retention is maintained through the vascular and tubular effects of this peptide. Sodium 170-176 arginine vasopressin Homo sapiens 47-58 15593031-5 2004 In addition, we propose that vasodilation and sodium/water excretion could ensue when both stimuli for vasopressin release fade away, leading to the resolution of the syndrome. Sodium 46-52 arginine vasopressin Homo sapiens 103-114 14526263-9 2003 Because sodium is a very efficient stimulus for VP secretion, it may be assumed that the fall in P(Na) occurring during AA infusion should have reduced VP secretion and thus P(VP). Sodium 8-14 arginine vasopressin Homo sapiens 48-50 14526263-9 2003 Because sodium is a very efficient stimulus for VP secretion, it may be assumed that the fall in P(Na) occurring during AA infusion should have reduced VP secretion and thus P(VP). Sodium 8-14 arginine vasopressin Homo sapiens 152-154 11382701-6 2001 This partial transection could permit vasopressin to be secreted in response to a larger rise in plasma sodium concentration. Sodium 104-110 arginine vasopressin Homo sapiens 38-49 12500203-0 2003 A vasopressin receptor antagonist (VPA-985) improves serum sodium concentration in patients with hyponatremia: a multicenter, randomized, placebo-controlled trial. Sodium 59-65 arginine vasopressin Homo sapiens 2-13 11824859-9 2002 In conclusion, our results show that in diabetic patients with AN, vasopressin participates in BP control by stimulating vascular and renal V1 receptors, which results in vasoconstriction and sodium reabsorption. Sodium 192-198 arginine vasopressin Homo sapiens 67-78 9789303-4 1998 An increase in peripheral AVP results in suppression of amino acid permeability and increases in transport of sodium, potassium ions and water. Sodium 110-116 arginine vasopressin Homo sapiens 26-29 11035341-0 2000 A simplified index of the plasma sodium threshold for arginine vasopressin secretion-morning fasting, euhydrated sodium levels. Sodium 33-39 arginine vasopressin Homo sapiens 63-74 11035341-16 2000 CONCLUSIONS: The morning fasting, euhydrated sodium level can be used as a simplified index for the plasma osmotic threshold for arginine vasopressin secretion. Sodium 45-51 arginine vasopressin Homo sapiens 138-149 10647278-4 1999 After introducing the therapy with demeclocycline, a tetracycline type antibiotic that inhibits the renal action of antidiuretic hormone, serum sodium levels began to rise gradually, and the urinary sodium excretion slowly decreased. Sodium 144-150 arginine vasopressin Homo sapiens 116-136 11386017-4 2001 MATERIAL AND METHODS: AVP was measured under basal conditions and after stimulation with a low-sodium diet and upright position. Sodium 95-101 arginine vasopressin Homo sapiens 22-25 11226092-0 2001 The release of antidiuretic hormone is appropriate in response to hypovolemia and/or sodium administration in children with severe head injury: a trial of lactated Ringer"s solution versus hypertonic saline. Sodium 85-91 arginine vasopressin Homo sapiens 15-35 11226092-10 2001 In a prospective, randomized, and controlled study in 31 children, we were able to show that the antidiuretic hormone levels are appropriate in response to hypovolemia, sodium load, or both. Sodium 169-175 arginine vasopressin Homo sapiens 97-117 11517684-0 2001 [Effects of anti-atherosclerotic low-sodium diet on dynamics of natural antibodies to angiotensin II, bradykinin and vasopressin in blood of patients with hypertension and obesity]. Sodium 37-43 arginine vasopressin Homo sapiens 117-128 10516282-0 1999 Vasopressin stimulates sodium transport in A6 cells via a phosphatidylinositide 3-kinase-dependent pathway. Sodium 23-29 arginine vasopressin Homo sapiens 0-11 9822114-1 1998 Traditionally, arginine vasopressin modulation of renal water, sodium, and urea excretion has been considered somewhat in isolation from factors that control divalent mineral ion homeostasis. Sodium 63-69 arginine vasopressin Homo sapiens 24-35 9679185-12 1998 The data demonstrate that plasma vasopressin and angiotensin II concentrations decrease during WI in hydrated humans, concomitantly with initial increases in CH2O and sodium excretion. Sodium 167-173 arginine vasopressin Homo sapiens 33-44 9581306-4 1998 The responses to intranasal arginine-vasopressin were prompt, normalizing the serum levels of sodium and increasing urinary osmolality, allowing a better metabolic balance, avoiding continuing damage to the already compromised CNS. Sodium 94-100 arginine vasopressin Homo sapiens 37-48 9432652-3 1997 The normal osmoregulation is an accurate operation which ensures the steadiness of serum sodium level by regulating vasopressin (ADH) release and water intake. Sodium 89-95 arginine vasopressin Homo sapiens 116-127 9397242-3 1997 Plasma VP was determined in 12 normotensive subjects and 12 patients with mild essential hypertension on a regular sodium diet, and in eight hypertensive patients on a high sodium (250 mmol/day) and a low sodium (25 mmol/day) diet. Sodium 115-121 arginine vasopressin Homo sapiens 7-9 9397242-7 1997 Levels of VP in the plasma was higher in the high sodium than in the low sodium period, but the difference was not significant. Sodium 50-56 arginine vasopressin Homo sapiens 10-12 9397242-7 1997 Levels of VP in the plasma was higher in the high sodium than in the low sodium period, but the difference was not significant. Sodium 73-79 arginine vasopressin Homo sapiens 10-12 9095083-6 1997 The renal cytolocalization of the Ang II/AVP receptor to the outer medullary thick ascending limb tubules and inner medullary collecting ducts is consistent with the well-established AVP stimulation of sodium and water reabsorption in these tubules. Sodium 202-208 arginine vasopressin Homo sapiens 41-44 9374816-13 1997 The less efficient sodium excretion occurring at low V is probably due to the influence of vasopressin on water, urea, and sodium movements across the collecting ducts. Sodium 123-129 arginine vasopressin Homo sapiens 91-102 9372692-1 1997 This study was undertaken to define the impact of arginine vasopressin (AVP) and atrial natriuretic peptide (ANP) on sodium homeostasis in patients with lung cancer. Sodium 117-123 arginine vasopressin Homo sapiens 59-70 9185106-3 1997 Among them, the renin-angiotensin-aldosterone axis, the sympathetic nervous system, the non-osmotic release of vasopressin and the endothelins are activated to increase vascular resistance and enhance sodium and water renal retention. Sodium 201-207 arginine vasopressin Homo sapiens 111-122 9152098-10 1997 Only basal positive psychotic symptoms (P < .09) and plasma sodium (P < .18) were even marginally associated with the peak antidiuretic hormone responses, but neither factor could explain the difference in the response by the 2 groups. Sodium 63-69 arginine vasopressin Homo sapiens 129-149 9095083-6 1997 The renal cytolocalization of the Ang II/AVP receptor to the outer medullary thick ascending limb tubules and inner medullary collecting ducts is consistent with the well-established AVP stimulation of sodium and water reabsorption in these tubules. Sodium 202-208 arginine vasopressin Homo sapiens 183-186 8910804-6 1996 The relationship of plasma vasopressin with serum sodium, but not plasma osmolality, during hyperglycemia showed an apparent hypersecretion of vasopressin. Sodium 50-56 arginine vasopressin Homo sapiens 27-38 9116921-6 1997 In the salt-sensitive patients whose mean arterial pressure increased more than 5 mmHg during high sodium intake, sodium loading increased cardiac index and terminal aortic flow, but decreased superior mesenteric and renal flows, while plasma noradrenaline concentrations remained unchanged and plasma arginine vasopressin increased significantly. Sodium 114-120 arginine vasopressin Homo sapiens 311-322 9116921-7 1997 These hemodynamic responses to sodium in the salt-sensitive patients were more effectively inhibited by benidipine than by nifedipine, although neither of them had any influence on sodium-induced changes in plasma noradrenaline or arginine vasopressin concentration. Sodium 31-37 arginine vasopressin Homo sapiens 240-251 9171309-5 1997 Intravenous administration of the selective nonpeptide vasopressin V2 antagonist OPC-31260 decreased sodium concentration and osmolality in urine to lower values than in plasma. Sodium 101-107 arginine vasopressin Homo sapiens 55-66 8900380-0 1996 Role of urinary arginine vasopressin in the sodium excretion in patients with chronic renal failure. Sodium 44-50 arginine vasopressin Homo sapiens 25-36 8910804-6 1996 The relationship of plasma vasopressin with serum sodium, but not plasma osmolality, during hyperglycemia showed an apparent hypersecretion of vasopressin. Sodium 50-56 arginine vasopressin Homo sapiens 143-154 8910804-9 1996 Together, we found that the positive correlation of vasopressin with sodium or osmolality is maintained but significantly altered in patients with untreated diabetes mellitus. Sodium 69-75 arginine vasopressin Homo sapiens 52-63 8897402-4 1996 AVP is probably important in the related adjustments of renal water excretion whereby changes in plasma sodium concentration induce changes in plasma osmolality and, subsequently, in release of AVP. Sodium 104-110 arginine vasopressin Homo sapiens 0-3 8897402-4 1996 AVP is probably important in the related adjustments of renal water excretion whereby changes in plasma sodium concentration induce changes in plasma osmolality and, subsequently, in release of AVP. Sodium 104-110 arginine vasopressin Homo sapiens 194-197 8790265-2 1996 The renin-angiotensin-aldosterone system, the sympathetic nervous system, and arginine vasopressin are responsible for sodium and water retention in patients with cirrhosis. Sodium 119-125 arginine vasopressin Homo sapiens 87-98 7793338-0 1995 Age-associated alterations in thirst and arginine vasopressin in response to a water or sodium load. Sodium 88-94 arginine vasopressin Homo sapiens 50-61 8769841-2 1996 We hypothesized that NO inhibits antidiuretic hormone (ADH)-stimulated fluid reabsorption (J(r)) by the isolated rat cortical collecting duct (CCD) by decreasing water permeability (Pf) and sodium reabsorption (Jna). Sodium 190-196 arginine vasopressin Homo sapiens 55-58 8769841-16 1996 We concluded that 1) NO decreases ADH-stimulated water and sodium transport in the isolate CCD, and 2) water reabsorption is inhibited by a primary effect on Pf. Sodium 59-65 arginine vasopressin Homo sapiens 34-37 7671566-7 1995 Salt restriction increased the antidiuretic effect of arginine vasopressin (2 fmol min-1 kg-1 arginine vasopressin increased urine osmolality from 67.8 +/- 2.6 to 196.3 +/- 35.7 mosmol/l in the high-salt study and from 268.3 +/- 49 mosmol/l in the low-salt study; P < 0.05 between sodium intakes). Sodium 284-290 arginine vasopressin Homo sapiens 63-74 7671566-7 1995 Salt restriction increased the antidiuretic effect of arginine vasopressin (2 fmol min-1 kg-1 arginine vasopressin increased urine osmolality from 67.8 +/- 2.6 to 196.3 +/- 35.7 mosmol/l in the high-salt study and from 268.3 +/- 49 mosmol/l in the low-salt study; P < 0.05 between sodium intakes). Sodium 284-290 arginine vasopressin Homo sapiens 103-114 7609571-9 1995 The mean plasma arginine vasopressin levels were significantly increased by sodium loading both before and after operation in all groups. Sodium 76-82 arginine vasopressin Homo sapiens 25-36 1325519-8 1992 When the SSC patients became SST with sodium loading, serum sodium and plasma arginine vasopressin decreased and haematocrit increased, suggesting that the excretion of sodium and water accompanied with a decrease in circulating plasma volume may be responsible for the hemodynamic alteration from SSC to SST. Sodium 38-44 arginine vasopressin Homo sapiens 87-98 8287649-0 1993 Control of sodium excretion in patients with cranial diabetes insipidus maintained on desamino-[8-D-arginine]vasopressin. Sodium 11-17 arginine vasopressin Homo sapiens 109-120 7686902-0 1993 Vasopressin increases cytosolic sodium concentration in hepatocytes and activates calcium influx through cation-selective channels. Sodium 32-38 arginine vasopressin Homo sapiens 0-11 8098306-1 1993 Heart failure is a syndrome characterized by the activation of neurohumoral mechanisms (sympathoadrenergic, renin-angiotensin, vasopressin) which cause peripheral vasoconstriction, sodium retention and myocardial hypertrophy. Sodium 181-187 arginine vasopressin Homo sapiens 127-138 8098306-8 1993 Neurohumoral activation can be roughly assessed using some simple laboratory measurements: plasma sodium concentration, for example, is inversely related to the degree of activation of many neurohormones such as norepinephrine, angiotensin II, vasopressin and atrial natriuretic factor. Sodium 98-104 arginine vasopressin Homo sapiens 244-255 8380126-0 1993 Atrial natriuretic factor and arginine vasopressin production in tumor cell lines from patients with lung cancer and their relationship to serum sodium. Sodium 145-151 arginine vasopressin Homo sapiens 39-50 8263145-7 1993 This abnormality in AVP secretion and thirst was corrected after glucocorticoid replacement with normalization of plasma sodium concentrations and osmolality. Sodium 121-127 arginine vasopressin Homo sapiens 20-23 8252410-1 1993 Vasopressin generates a voltage-gated, sodium-dependent current in facial motoneurons in brainstem slices. Sodium 39-45 arginine vasopressin Homo sapiens 0-11 8222513-13 1993 We conclude that the fall in plasma arginine vasopressin concentration during dietary salt restriction, whilst not affecting renal sodium excretion, may be important in the regulation of plasma sodium concentration, plasma renin activity and glomerular filtration. Sodium 194-200 arginine vasopressin Homo sapiens 45-56 8230088-7 1993 Intravascular angiotensin II receptors are implicated in the central release of vasopressin and other hypophyseal hormones, in increasing sympathetic outflow, in the thirst response and, possibly, in cognitive function; in the inotropic and chronotropic effects of angiotensin II on the heart as well as in growth/hypertrophy; in the control of aldosterone release and in the balance between cortisol and aldosterone secretion; and in modulating sodium, chloride and bicarbonate transport within the kidney. Sodium 446-452 arginine vasopressin Homo sapiens 80-91 8345832-2 1993 In the present study, in 12 consecutive hospitalized elderly patients (mean age 82.2 years) with moderate to severe hypernatremia (mean serum sodium 166.9 mEq/l), inappropriately low plasma levels of vasopressin were found in relation to serum osmolality (mean 1.8 pg/ml and 343 mosmol/l, respectively). Sodium 142-148 arginine vasopressin Homo sapiens 200-211 1492537-3 1992 The mean basal plasma vasopressin value in the patients (0.6 +/- 0.1 pmol/l) was significantly lower than that in the normal subjects (2.9 +/- 0.3 pmol/l; p < 0.01), whereas the mean serum sodium, plasma osmolality, plasma renin activity and serum aldosterone values were similar in the two groups. Sodium 192-198 arginine vasopressin Homo sapiens 22-33 1477846-6 1992 Most sodium abnormalities in patients with CNS disease result from altered water excretion secondary to disturbed release of antidiuretic hormone (ADH). Sodium 5-11 arginine vasopressin Homo sapiens 125-145 1477846-6 1992 Most sodium abnormalities in patients with CNS disease result from altered water excretion secondary to disturbed release of antidiuretic hormone (ADH). Sodium 5-11 arginine vasopressin Homo sapiens 147-150 1315019-3 1992 Although it has been presumed that the response to AVP results from augmented sodium transport, there is controversy whether AVP actually does affect sodium transport in mammalian lung epithelium. Sodium 78-84 arginine vasopressin Homo sapiens 51-54 1507492-7 1992 The excretion of ADH in a day was in the urine of diabetic patients positively correlated with HbA1, urinary osmolarity and concentration of sodium in urine, although the pathological meaning of the observed ADH hypersecretion in the development of diabetic complications is currently unknown. Sodium 141-147 arginine vasopressin Homo sapiens 17-20 1531798-8 1992 Although blood pressure was not changed significantly, the enhanced response of ADH to a sodium and volume load may play a role in part in the pathophysiology of EHT. Sodium 89-95 arginine vasopressin Homo sapiens 80-83 1739390-1 1992 Plasma sodium concentration depends on water balance, and is normally maintained in a narrow range by an integrated system involving the precise regulation of water intake via thirst mechanism and control of water output via vasopressin secretion. Sodium 7-13 arginine vasopressin Homo sapiens 225-236 1937681-10 1991 The resultant renal vasoconstriction limits the distal tubular delivery of sodium and water, thus maximizing the water-retaining effect of vasopressin and impairing the normal escape from the sodium-retaining effects of aldosterone. Sodium 75-81 arginine vasopressin Homo sapiens 139-150 1831329-3 1991 Hypertonic saline alone produced a progressive rise in plasma vasopressin with increasing serum sodium. Sodium 96-102 arginine vasopressin Homo sapiens 62-73 1914197-4 1991 Patients with severe hypertension, especially that associated with end-stage renal disease, tended to respond with moderate increases in blood pressure and plasma AVP after sodium overload and had a modest blood pressure fall (10-20 mmHg) in response to a single intravenous bolus of the AVP antagonist. Sodium 173-179 arginine vasopressin Homo sapiens 163-166 1745008-0 1991 Changes in intracellular sodium during the hydroosmotic response to vasopressin. Sodium 25-31 arginine vasopressin Homo sapiens 68-79 1745008-3 1991 We hypothesized that the hydroosmotic response to VP may be associated with a net increase in sodium either as an aid in maintaining the intracellular osmolality or as part of a Na-Ca exchange process. Sodium 94-100 arginine vasopressin Homo sapiens 50-52 1831329-4 1991 During hypertonic saline alone, vasopressin levels began to rise at an increment in serum sodium of 1.67 +/- 0.35 mM in the young and 1.43 +/- 0.32 mM in the elderly and rose linearly with increasing serum sodium. Sodium 90-96 arginine vasopressin Homo sapiens 32-43 1831329-4 1991 During hypertonic saline alone, vasopressin levels began to rise at an increment in serum sodium of 1.67 +/- 0.35 mM in the young and 1.43 +/- 0.32 mM in the elderly and rose linearly with increasing serum sodium. Sodium 206-212 arginine vasopressin Homo sapiens 32-43 1831329-5 1991 When ANP was infused with hypertonic saline (with peak ANP levels of approximately 1,000 pM), vasopressin levels began to rise at an increment in serum sodium of 4.43 +/- 0.67 mM in the young and 4.57 +/- 0.43 mM in the elderly (P less than 0.01 vs. saline alone). Sodium 152-158 arginine vasopressin Homo sapiens 94-105 1831329-6 1991 Furthermore, the vasopressin response for any given serum sodium was significantly reduced in both young and elderly subjects, resulting in a rightward displacement of the curve relating vasopressin response to sodium concentration (P less than 0.001). Sodium 58-64 arginine vasopressin Homo sapiens 17-28 1831329-6 1991 Furthermore, the vasopressin response for any given serum sodium was significantly reduced in both young and elderly subjects, resulting in a rightward displacement of the curve relating vasopressin response to sodium concentration (P less than 0.001). Sodium 58-64 arginine vasopressin Homo sapiens 187-198 1831329-6 1991 Furthermore, the vasopressin response for any given serum sodium was significantly reduced in both young and elderly subjects, resulting in a rightward displacement of the curve relating vasopressin response to sodium concentration (P less than 0.001). Sodium 211-217 arginine vasopressin Homo sapiens 17-28 1831329-6 1991 Furthermore, the vasopressin response for any given serum sodium was significantly reduced in both young and elderly subjects, resulting in a rightward displacement of the curve relating vasopressin response to sodium concentration (P less than 0.001). Sodium 211-217 arginine vasopressin Homo sapiens 187-198 2213375-8 1990 The plasma AVP concentration was significantly lower after 24 hours of maintenance plus replacement fluids than after fluid restriction (p = 0.005), and the change in AVP concentration correlated with the amount of sodium given (p less than 0.02). Sodium 215-221 arginine vasopressin Homo sapiens 167-170 2060872-4 1991 We have examined the effect of the non-pressor (V2) synthetic vasopressin analogue 1-deamino-8-D-arginine (desmopressin) on renal and ileal sodium and water excretion in ileostomy patients during acute adaptation to a low sodium diet. Sodium 140-146 arginine vasopressin Homo sapiens 62-73 1646297-0 1991 Vasopressin generates a persistent voltage-dependent sodium current in a mammalian motoneuron. Sodium 53-59 arginine vasopressin Homo sapiens 0-11 1646297-10 1991 Our results suggest that vasopressin increases the excitability of facial motoneurons by generating a persistent sodium-dependent membrane current that is voltage gated and TTX resistant. Sodium 113-119 arginine vasopressin Homo sapiens 25-36 1852105-1 1991 To determine the mechanism by which vasopressin increases sodium transport in sodium-transporting, tight epithelia, we examined single amiloride-blockable Na channels in membrane patches from cultured distal nephron cells (A6) either before or after treatment with arginine vasopressin. Sodium 58-64 arginine vasopressin Homo sapiens 36-47 1656123-6 1991 Treatment with a vasopressin analogue, desamino-D-arginine vasopressin and forced intake of water restored plasma osmolality and serum sodium levels to normal. Sodium 135-141 arginine vasopressin Homo sapiens 17-28 1656123-6 1991 Treatment with a vasopressin analogue, desamino-D-arginine vasopressin and forced intake of water restored plasma osmolality and serum sodium levels to normal. Sodium 135-141 arginine vasopressin Homo sapiens 59-70 1647438-4 1991 When sodium transport was blocked by amiloride, the H+ permeability of the apical membranes of principal cells was negligible but increased dramatically after treatment with antidiuretic hormone (ADH). Sodium 5-11 arginine vasopressin Homo sapiens 174-194 1647438-4 1991 When sodium transport was blocked by amiloride, the H+ permeability of the apical membranes of principal cells was negligible but increased dramatically after treatment with antidiuretic hormone (ADH). Sodium 5-11 arginine vasopressin Homo sapiens 196-199 2079200-1 1990 The capacity of five synthetic analogs of [8-arginine] vasopressin (AVP) to stimulate frog skin sodium transport (natriferic activity) was characterized electrophysiologically using the method of short-circuit current, and compared to that of synthetic AVP. Sodium 96-102 arginine vasopressin Homo sapiens 55-66 1852105-1 1991 To determine the mechanism by which vasopressin increases sodium transport in sodium-transporting, tight epithelia, we examined single amiloride-blockable Na channels in membrane patches from cultured distal nephron cells (A6) either before or after treatment with arginine vasopressin. Sodium 78-84 arginine vasopressin Homo sapiens 36-47 2213375-9 1990 This study supports the hypothesis that serum AVP concentrations are elevated in patients with meningitis because of hypovolemia and become normal when sufficient sodium is given to facilitate reabsorption of water by the proximal tubule of the kidney. Sodium 163-169 arginine vasopressin Homo sapiens 46-49 2193561-6 1990 The renal vasoconstriction, which accompanies either decreased cardiac output or peripheral arterial vasodilation, causes a decreased distal tubular delivery of sodium and water, thus maximizing the water-retaining effect of vasopressin and impairing normal escape from the sodium-retaining effect of aldosterone. Sodium 161-167 arginine vasopressin Homo sapiens 225-236 2162269-7 1990 In the syndrome of inappropriate secretion of antidiuretic hormone, the fractional excretion of filtered urea was inversely correlated to the fractional excretion of filtered sodium (r = -0.66; P less than 0.001), whereas the fractional excretion of filtered uric acid was not dependent on sodium excretion. Sodium 175-181 arginine vasopressin Homo sapiens 46-66 2162269-7 1990 In the syndrome of inappropriate secretion of antidiuretic hormone, the fractional excretion of filtered urea was inversely correlated to the fractional excretion of filtered sodium (r = -0.66; P less than 0.001), whereas the fractional excretion of filtered uric acid was not dependent on sodium excretion. Sodium 290-296 arginine vasopressin Homo sapiens 46-66 2162269-9 1990 Hypouraemia with high fractional excretion of filtered urea in patients with the syndrome of inappropriate secretion of antidiuretic hormone is related to low urinary sodium excretion and thus reflects low sodium intake. Sodium 167-173 arginine vasopressin Homo sapiens 120-140 2162269-9 1990 Hypouraemia with high fractional excretion of filtered urea in patients with the syndrome of inappropriate secretion of antidiuretic hormone is related to low urinary sodium excretion and thus reflects low sodium intake. Sodium 206-212 arginine vasopressin Homo sapiens 120-140 2480537-0 1989 Influence of vasopressin level on osmotic pressure and sodium concentration in plasma and cerebrospinal fluid in patients with intracranial lesions. Sodium 55-61 arginine vasopressin Homo sapiens 13-24 2136743-3 1990 On a high sodium diet there was an increase in urine volume, sodium excretion, osmolal clearance, plasma ANP concentration, and urinary clearance and fractional excretion of AVP, with a decrease in PRA. Sodium 10-16 arginine vasopressin Homo sapiens 174-177 2137185-2 1990 This characteristic of the medullary circulation plays an important role in the kidney"s ability to excrete a dilute or concentrated urine in concert with changes in water and sodium transport in the distal nephron secondary to the action of vasopressin, prostaglandins, the renal nerves, and other hormones without significant other renal hemodynamic changes. Sodium 176-182 arginine vasopressin Homo sapiens 242-253 2377564-3 1990 This case is interesting because after adequate rehydration and normalization of electrolytes we registered a remarkable fall of plasmatic sodium and chloride as a result of hemodilution probably due to a syndrome of inappropriate secretion of antidiuretic hormone, confirmed by values of plasmatic and urinary osmolarity and of urinary electrolytes. Sodium 139-145 arginine vasopressin Homo sapiens 244-264 2276581-2 1990 The effect of vasopressin and insulin on active sodium transport across frog skin in the presence of internal alternariol mycotoxin was studied, using the short-circuit technique. Sodium 48-54 arginine vasopressin Homo sapiens 14-25 2276581-4 1990 Vasopressin stimulates sodium transport across frog skin by decreasing the resistance to sodium entry into the epithelial cells, thus partially removing the inhibition on the short-circuit current due to the action of Alternariol mycotoxin. Sodium 23-29 arginine vasopressin Homo sapiens 0-11 2276581-4 1990 Vasopressin stimulates sodium transport across frog skin by decreasing the resistance to sodium entry into the epithelial cells, thus partially removing the inhibition on the short-circuit current due to the action of Alternariol mycotoxin. Sodium 89-95 arginine vasopressin Homo sapiens 0-11 34422415-11 2021 The combination of IV Furosemide with Vasopressin infusion resulted in tailored diuresis and more controlled titration of serum sodium levels than adjustment in Vasopressin and fluids alone. Sodium 128-134 arginine vasopressin Homo sapiens 38-49 34238463-9 2021 Finally, we report on the effects of drugs such as AVP analogues and/or oxytocin, another neuropeptide that increases sodium excretion in animal models and humans with CDI, and sildenafil, a compound that increases the expression and function of AQP2 channels in animal models and humans with NDI. Sodium 118-124 arginine vasopressin Homo sapiens 51-54 35371779-10 2022 Theoretically, treatment of hydrocephalus-related hyponatremia with CSF-diversion procedures should relieve the pressure on the hypothalamus, mitigating ADH production, which in turn will decrease sodium excretion and ameliorate the hyponatremia. Sodium 197-203 arginine vasopressin Homo sapiens 153-156 2480537-1 1989 To study the influence of the vasopressin level on osmotic pressure and sodium concentration in plasma and cerebrospinal fluid (CSF), plasma and CSF were sampled simultaneously in 27 patients with central nervous system lesions. Sodium 72-78 arginine vasopressin Homo sapiens 30-41 2673748-0 1989 Effects of sodium depletion and orthostasis on plasma and urinary vasopressin in normal subjects. Sodium 11-17 arginine vasopressin Homo sapiens 66-77 2670663-6 1989 The effects of insulin were compared with those of a vasopressin analog (dDAVP), known to stimulate transepithelial sodium transport by increasing the permeability of the apical cell membrane for sodium ions. Sodium 116-122 arginine vasopressin Homo sapiens 53-64 2673748-1 1989 We investigated the effects of sodium depletion and orthostasis on the plasma concentration and urinary excretion of vasopressin (AVP) in eight normal female subjects. Sodium 31-37 arginine vasopressin Homo sapiens 117-128 2673748-4 1989 A significant (p less than 0.05) increase in plasma AVP and a significant (p less than 0.05) decrease in 24-h urinary excretion of AVP were observed during sodium depletion. Sodium 156-162 arginine vasopressin Homo sapiens 52-55 2673748-4 1989 A significant (p less than 0.05) increase in plasma AVP and a significant (p less than 0.05) decrease in 24-h urinary excretion of AVP were observed during sodium depletion. Sodium 156-162 arginine vasopressin Homo sapiens 131-134 2673748-5 1989 One-hour ambulation significantly increased plasma AVP in both control and sodium depleted phases (p less than 0.01). Sodium 75-81 arginine vasopressin Homo sapiens 51-54 2673748-7 1989 The present results suggest that AVP may play an important role in the maintenance of blood pressure during orthostasis in the sodium depleted state. Sodium 127-133 arginine vasopressin Homo sapiens 33-36 2522705-9 1989 Thus, patients with primary hyperparathyroidism have elevated plasma levels of angiotensin II and arginine vasopressin which may be compensatory phenomena counteracting volume depletion owing to a decreased renal concentrating ability induced by hypercalcemia, and owing to PTH-induced inhibition of renal sodium reabsorption. Sodium 306-312 arginine vasopressin Homo sapiens 107-118 2920003-1 1989 In neurosurgical patients with hyponatraemia (plasma sodium less than 130 mmol/l) and natriuresis, increased antidiuretic hormone (ADH) secretion may be appropriate rather than inappropriate. Sodium 53-59 arginine vasopressin Homo sapiens 109-129 2923250-4 1989 Plasma sodium was lower during the hyperglycemic study, but elevation in plasma sodium concentration by infusion of saline caused progressive linear increases in both thirst and plasma vasopressin concentrations in both studies. Sodium 80-86 arginine vasopressin Homo sapiens 185-196 2923250-5 1989 Linear regression analysis defined lowered plasma sodium thresholds for both thirst appreciation and vasopressin release during the hyperglycemic study, although the sensitivity of the osmoreceptors remained unchanged. Sodium 50-56 arginine vasopressin Homo sapiens 101-112 2923250-8 1989 These results show that insulin-dependent diabetic patients osmoregulate appropriately when moderately hyperglycemic but that the threshold plasma sodium for vasopressin secretion and thirst appreciation is lowered by an unknown mechanism. Sodium 147-153 arginine vasopressin Homo sapiens 158-169 2566711-2 1989 Adequate secretion of vasopressin and aldosterone in response to the changes in sodium concentration and plasma osmolality point to the absence of significant functional disorders of the corresponding glands. Sodium 80-86 arginine vasopressin Homo sapiens 22-33 3386276-0 1988 Interaction between aldosterone and vasopressin on vascular smooth muscle permeability to sodium. Sodium 90-96 arginine vasopressin Homo sapiens 36-47 2712937-7 1989 The average serum sodium in the group with other febrile illnesses was higher (146 +/- 5 mEq/L; p less than 0.05) and could represent an appropriate stimulus for antidiuretic hormone release. Sodium 18-24 arginine vasopressin Homo sapiens 162-182 2975995-1 1988 The possible interaction between arginine vasopressin (AVP) and atrial natriuretic factor (ANF) in the control of urinary sodium and water excretion was investigated in man. Sodium 122-128 arginine vasopressin Homo sapiens 33-53 2975995-1 1988 The possible interaction between arginine vasopressin (AVP) and atrial natriuretic factor (ANF) in the control of urinary sodium and water excretion was investigated in man. Sodium 122-128 arginine vasopressin Homo sapiens 55-58 3130264-3 1988 Infusion of vasopressin in normal subjects was associated with an increase in fractional excretion of sodium by the kidney, which continued during the day after the infusion was stopped. Sodium 102-108 arginine vasopressin Homo sapiens 12-23 3130264-5 1988 Infusion of vasopressin in patients lacking adrenocortical function was associated with a smaller increase in fractional excretion of sodium by the kidney in comparison with that seen in normal subjects, and the increase in sodium excretion was confined to the period of the infusion. Sodium 134-140 arginine vasopressin Homo sapiens 12-23 3130264-5 1988 Infusion of vasopressin in patients lacking adrenocortical function was associated with a smaller increase in fractional excretion of sodium by the kidney in comparison with that seen in normal subjects, and the increase in sodium excretion was confined to the period of the infusion. Sodium 224-230 arginine vasopressin Homo sapiens 12-23 3130264-7 1988 We conclude that changes in mineralocorticoid activity are the main factors contributing to the increase in renal sodium excretion seen during, and after, the continuous infusion of vasopressin in salt- and water-restricted man. Sodium 114-120 arginine vasopressin Homo sapiens 182-193 2902974-2 1988 Vasopressin induces a rapid increase in water permeability and stimulates net sodium transport in responsive epithelia through the mediation of cAMP. Sodium 78-84 arginine vasopressin Homo sapiens 0-11 2975718-3 1988 On the contrary, aldosterone increased and vasopressin decreased in the low sodium period but did not change between the control and high sodium periods. Sodium 76-82 arginine vasopressin Homo sapiens 43-54 2552973-3 1989 The patient showed persistent hyponatremia accompanied with continuous loss of sodium in the urine, which resulted from inappropriate secretion of antidiuretic hormone (ADH). Sodium 79-85 arginine vasopressin Homo sapiens 147-167 2552973-3 1989 The patient showed persistent hyponatremia accompanied with continuous loss of sodium in the urine, which resulted from inappropriate secretion of antidiuretic hormone (ADH). Sodium 79-85 arginine vasopressin Homo sapiens 169-172 2478241-4 1989 Our findings suggest that monensin uncouples the vasopressin-receptor-G protein-adenylate cyclase sequence at some early step, by a mechanism that remains unknown, but that may directly or indirectly involve intracellular sodium. Sodium 222-228 arginine vasopressin Homo sapiens 49-60 2458353-1 1988 Arginine vasopressin (antidiuretic hormone, ADH) stimulation of sodium transport in high electrical resistance epithelia is accompanied by adenylate cyclase stimulation and cAMP accumulation. Sodium 64-70 arginine vasopressin Homo sapiens 9-20 3121240-3 1987 Increased concentrations of antidiuretic hormone (ADH) result in retention of free water, increased excretion of sodium, and hyponatremia. Sodium 113-119 arginine vasopressin Homo sapiens 28-48 3121240-3 1987 Increased concentrations of antidiuretic hormone (ADH) result in retention of free water, increased excretion of sodium, and hyponatremia. Sodium 113-119 arginine vasopressin Homo sapiens 50-53 3963083-0 1986 Sodium excretion in human pregnancy: a role for arginine vasopressin. Sodium 0-6 arginine vasopressin Homo sapiens 57-68 3476800-2 1987 Despite some empirical and theoretical objections, linear regression analysis of the relationship between plasma vasopressin and plasma osmolality or sodium continues to provide a simple and useful way to describe the major functional properties of the osmoregulatory system. Sodium 150-156 arginine vasopressin Homo sapiens 113-124 3035942-2 1987 Exposure of isolated toad bladders to quinidine, calcium ionophores (A23187, X537A), or low-sodium or potassium-free serosal solutions resulted in a dose-dependent decrease in the hydrosmotic response to vasopressin or exogenous adenosine 3",5"-cyclic monophosphate (cAMP). Sodium 92-98 arginine vasopressin Homo sapiens 204-215 3552505-4 1987 Using univariate analysis, all the patients with poor sodium excretion had abnormally high levels of plasma renin activity, plasma aldosterone, and arginine vasopressin. Sodium 54-60 arginine vasopressin Homo sapiens 157-168 3551628-4 1987 This increase in vasopressin was not due to a loss of osmoregulation, because changes in plasma osmolality and sodium, induced by infusion of hypertonic saline or water loading, induced appropriate vasopressin responses under insulin deplete as well as replete conditions. Sodium 111-117 arginine vasopressin Homo sapiens 17-28 3551628-4 1987 This increase in vasopressin was not due to a loss of osmoregulation, because changes in plasma osmolality and sodium, induced by infusion of hypertonic saline or water loading, induced appropriate vasopressin responses under insulin deplete as well as replete conditions. Sodium 111-117 arginine vasopressin Homo sapiens 198-209 2948754-3 1987 Plasma renin activity and aldosterone decreased significantly with increasing sodium intake whereas plasma vasopressin was highest on the high sodium intake. Sodium 143-149 arginine vasopressin Homo sapiens 107-118 3789043-5 1986 Increased fetal plasma and urine arginine vasopressin concentrations were associated with significant increases in fetal urine osmolality (146 +/- 12 to 262 +/- 30 mosm) and sodium concentration (35.8 +/- 2.8 to 76.5 +/- 20 mu Eq/ml), but fetal urine production rates did not change (0.68 +/- 0.11 to 0.62 +/- 0.15 ml/min). Sodium 174-180 arginine vasopressin Homo sapiens 42-53 3457670-7 1986 A significant increase in fractional sodium clearance of approximately 50% occurred during the AVP infusions, which was abolished after pretreatment with indomethacin. Sodium 37-43 arginine vasopressin Homo sapiens 95-98 3963083-2 1986 A significant rise in plasma arginine vasopressin was observed only in second-trimester subjects following sodium loading (5.6 +/- 0.5 to 6.6 +/- 0.5 pg/ml; p less than 0.05). Sodium 107-113 arginine vasopressin Homo sapiens 38-49 3963083-5 1986 It is suggested that arginine vasopressin secretion increases to assist natriuresis following dietary sodium loading in the second trimester of human pregnancy. Sodium 102-108 arginine vasopressin Homo sapiens 30-41 3514728-3 1986 Plasma vasopressin concentration correlated with the percent decrease of systemic vascular resistance (r = 0.70, p less than 0.025), serum sodium (r = 0.72, p less than 0.02) and serum creatinine (r = 0.85, p less than 0.005). Sodium 139-145 arginine vasopressin Homo sapiens 7-18 3950526-0 1986 Vasopressin secretion in normotensive black and white men and women on normal and low sodium diets. Sodium 86-92 arginine vasopressin Homo sapiens 0-11 3950526-2 1986 During the normal sodium diet, the 24-h urinary excretion of vasopressin was significantly (P less than 0.05) higher in men than in women and higher (P less than 0.05) in black than in white subjects. Sodium 18-24 arginine vasopressin Homo sapiens 61-72 3950526-5 1986 Reduction in sodium intake resulted in a significantly (P less than 0.01) decreased excretion of vasopressin in all groups except black women, but had no effect on plasma vasopressin concentrations. Sodium 13-19 arginine vasopressin Homo sapiens 97-108 3535395-8 1986 Thus, during sodium restriction in middle-aged hypertensive men, blood pressure reduction occurs concomitantly with inhibited vasopressin release, despite enhanced renin and catecholamine release. Sodium 13-19 arginine vasopressin Homo sapiens 126-137 3554904-5 1986 Thus, vasopressin was the only pressor hormone which varied directly with sodium intake, blood pressure and body weight during sodium depletion. Sodium 74-80 arginine vasopressin Homo sapiens 6-17 2988106-2 1985 The concentration of AVP in plasma increased 2-4 fold, osmolality in serum increased 12-16 mosmol/kg, and urinary excretion of cAMP increased 20-40% during sodium loading to the same extent in all three groups. Sodium 156-162 arginine vasopressin Homo sapiens 21-24 3554904-5 1986 Thus, vasopressin was the only pressor hormone which varied directly with sodium intake, blood pressure and body weight during sodium depletion. Sodium 127-133 arginine vasopressin Homo sapiens 6-17 2434767-1 1986 Vasopressin increases the permeability of responsive epithelia for a variety of chemical species, such as sodium, urea, water, and lipophilic molecules. Sodium 106-112 arginine vasopressin Homo sapiens 0-11 3944536-5 1986 We conclude that, in man, AVP is selectively secreted in response to both dehydration and high sodium intake, whilst even after the stimulus of rapidly increasing plasma osmolality during intravenous infusion of hypertonic saline the rise in oxytocin is not statistically significant. Sodium 95-101 arginine vasopressin Homo sapiens 26-29 4085913-1 1985 An infusion with Ornipressin (8-ornithin vasopressin) in a patient with decompensated alcoholic liver cirrhosis increased urinary volume from 30 ml/h to 500 ml/h, creatinine clearance from 24 to 65 ml/min, and fractional sodium excretion from 0.86% to 11.1%. Sodium 221-227 arginine vasopressin Homo sapiens 41-52 3898868-3 1985 During osmotic stimulation with hypertonic saline, plasma vasopressin increased in close linear correlation with plasma osmolality or sodium in both groups. Sodium 134-140 arginine vasopressin Homo sapiens 58-69 4003573-9 1985 Finally, the effects on thirst and vasopressin secretion were observed at plasma sodium concentration and osmolality changes that are well within the physiological range. Sodium 81-87 arginine vasopressin Homo sapiens 35-46 3915319-0 1985 Dietary sodium intake increases vasopressin secretion in man. Sodium 8-14 arginine vasopressin Homo sapiens 32-43 3915319-2 1985 Urinary vasopressin excretion decreased from 6.7 +/- 1.0 ng/hr (control) to 3.9 +/- 0.3 ng/hr (p less than 0.01) when sodium excretion decreased from 188 +/ 18 to 16 +/- 2 mmol/24 hr. Sodium 118-124 arginine vasopressin Homo sapiens 8-19 3915319-3 1985 During the first day of high sodium intake, the urinary vasopressin excretion increased to 10.0 +/- 1.2 ng/hr (p less than 0.01) compared with control and remained high throughout the sodium repletion. Sodium 29-35 arginine vasopressin Homo sapiens 56-67 3915319-3 1985 During the first day of high sodium intake, the urinary vasopressin excretion increased to 10.0 +/- 1.2 ng/hr (p less than 0.01) compared with control and remained high throughout the sodium repletion. Sodium 184-190 arginine vasopressin Homo sapiens 56-67 3915319-4 1985 Through all collection periods at low, normal, and high sodium intake, vasopressin excretion increased concomitantly with serum sodium concentration and osmolality. Sodium 56-62 arginine vasopressin Homo sapiens 71-82 3915319-4 1985 Through all collection periods at low, normal, and high sodium intake, vasopressin excretion increased concomitantly with serum sodium concentration and osmolality. Sodium 128-134 arginine vasopressin Homo sapiens 71-82 3915319-5 1985 After low sodium intake for 7 days, the serum vasopressin concentration averaged 2.7 +/- 0.6 ng/l, and this level was maintained throughout the sodium repletion period. Sodium 10-16 arginine vasopressin Homo sapiens 46-57 3915319-5 1985 After low sodium intake for 7 days, the serum vasopressin concentration averaged 2.7 +/- 0.6 ng/l, and this level was maintained throughout the sodium repletion period. Sodium 144-150 arginine vasopressin Homo sapiens 46-57 3915319-6 1985 These results are compatible with a stimulatory effect of dietary sodium intake on pituitary vasopressin secretion in man. Sodium 66-72 arginine vasopressin Homo sapiens 93-104 3915319-7 1985 Dietary sodium may stimulate vasopressin secretion through extracellular osmolality or even by a direct effect of extracellular sodium on periventricular receptors. Sodium 8-14 arginine vasopressin Homo sapiens 29-40 3900253-3 1985 Regulation of water and sodium metabolism: urine-concentrating mechanisms and action of vasopressin]. Sodium 24-30 arginine vasopressin Homo sapiens 88-99 2988106-7 1985 In conclusion, the increase in cAMP excretion in urine during the sodium loading might be explained by an AVP-induced stimulation of renal cAMP production. Sodium 66-72 arginine vasopressin Homo sapiens 106-109 6144625-11 1984 In addition, the serum sodium concentration was significantly reduced by vasopressin (from 134.3 +/- 1.6 to 128.3 +/- 1.4 mEq per liter, p less than 0.001) but not by somatostatin (134.6 +/- 1.1 vs. 133.2 +/- 1.1 mEq per liter). Sodium 23-29 arginine vasopressin Homo sapiens 73-84 6599675-1 1984 The present study was undertaken to examine the possible relationship between dietary sodium intake and arginine vasopressin (AVP). Sodium 86-92 arginine vasopressin Homo sapiens 113-124 6599675-1 1984 The present study was undertaken to examine the possible relationship between dietary sodium intake and arginine vasopressin (AVP). Sodium 86-92 arginine vasopressin Homo sapiens 126-129 6599675-2 1984 In 12 normotensive men (aged 23-26 years) urinary AVP excretion decreased from 6.7 +/- 1.0 to 3.9 +/- 0.3 ng/h (P less than 0.01) when sodium excretion by dietary intervention for one week was reduced from 188 +/- 18 to 16 +/- 2 mmol/24 h. At a high sodium intake (300 mmol/day), AVP excretion increased to 10.0 +/- 1.2 ng/h during the first day (P less than 0.01) and remained high throughout one week of sodium load. Sodium 135-141 arginine vasopressin Homo sapiens 50-53 6599675-2 1984 In 12 normotensive men (aged 23-26 years) urinary AVP excretion decreased from 6.7 +/- 1.0 to 3.9 +/- 0.3 ng/h (P less than 0.01) when sodium excretion by dietary intervention for one week was reduced from 188 +/- 18 to 16 +/- 2 mmol/24 h. At a high sodium intake (300 mmol/day), AVP excretion increased to 10.0 +/- 1.2 ng/h during the first day (P less than 0.01) and remained high throughout one week of sodium load. Sodium 250-256 arginine vasopressin Homo sapiens 50-53 6599675-2 1984 In 12 normotensive men (aged 23-26 years) urinary AVP excretion decreased from 6.7 +/- 1.0 to 3.9 +/- 0.3 ng/h (P less than 0.01) when sodium excretion by dietary intervention for one week was reduced from 188 +/- 18 to 16 +/- 2 mmol/24 h. At a high sodium intake (300 mmol/day), AVP excretion increased to 10.0 +/- 1.2 ng/h during the first day (P less than 0.01) and remained high throughout one week of sodium load. Sodium 250-256 arginine vasopressin Homo sapiens 50-53 6599675-3 1984 These results are compatible with a major physiological role of sodium in AVP secretion in man. Sodium 64-70 arginine vasopressin Homo sapiens 74-77 6495367-8 1984 The administration of low-dose (2-10 microU/kg/min) vasopressin by continuous infusion maintained plasma sodium and osmolality in the normal range over the course of the experiments (24 hr) in the experimental group. Sodium 105-111 arginine vasopressin Homo sapiens 52-63 6741439-4 1984 Plasma ADH markedly increased after NaCl administration and was significantly correlated with plasma sodium (r = 0.67, P less than 0.005) when the patients were awake, whereas it did not change over 30 min and was not correlated with plasma sodium (r = 0.18, P greater than 0.05) under fentanyl anaesthesia. Sodium 101-107 arginine vasopressin Homo sapiens 7-10 6696107-6 1984 These results indicate that exogenous vasopressin may stimulate the release of endogenous AVP, an effect that appears to be enhanced by sodium depletion and is virtually absent in the elderly. Sodium 136-142 arginine vasopressin Homo sapiens 38-49 6741563-8 1984 A negative correlation between renal PG production and VP plasma levels and excretion was demonstrated during the changes of dietary sodium intake. Sodium 133-139 arginine vasopressin Homo sapiens 55-57 6699780-8 1984 A 30 mV, mucosa-positive voltage clamp, applied near the peak of the response to vasopressin, further increased both sodium transport and carbon dioxide production. Sodium 117-123 arginine vasopressin Homo sapiens 81-92 6699780-14 1984 Sodium transport was increased by approximately 35% more when aldosterone-treated hemibladders were voltage clamped after vasopressin, the control paired hemibladders being exposed to vasopressin and voltage clamping alone. Sodium 0-6 arginine vasopressin Homo sapiens 122-133 6699780-14 1984 Sodium transport was increased by approximately 35% more when aldosterone-treated hemibladders were voltage clamped after vasopressin, the control paired hemibladders being exposed to vasopressin and voltage clamping alone. Sodium 0-6 arginine vasopressin Homo sapiens 184-195 6294273-1 1983 Vasopressin enhances osmotic water flow and sodium transport across the toad urinary bladder by mechanisms involving cyclic AMP and calcium. Sodium 44-50 arginine vasopressin Homo sapiens 0-11 6363775-0 1983 [Effects of vasopressin and insulin on sodium transport in the cultured renal cells]. Sodium 39-45 arginine vasopressin Homo sapiens 12-23 6337910-5 1983 Subsequently, a small but significant decrease in serum arginine vasopressin levels to 3.04 +/- 0.65 microU/ml (p less than 0.05) was associated with a further rise in serum sodium levels above baseline values (138 +/- 1.4 mEq/L, p less than 0.01) and in serum osmolality. Sodium 174-180 arginine vasopressin Homo sapiens 65-76 6137317-7 1983 Vasopressin failed to increase SCC in vivo when the external sodium concentration was 115 mM, being effective only when the sodium concentration was low (5 mM). Sodium 124-130 arginine vasopressin Homo sapiens 0-11 7026596-5 1981 The significance of the plasma sodium rise was assessed by observing the vasopressin response to hypoglycemia in a patient shown previously to have a selective loss of the vasopressin response to osmotic stimulation. Sodium 31-37 arginine vasopressin Homo sapiens 73-84 6286278-0 1982 Vasopressin resistance induced by low sodium and high mannitol in toad bladder. Sodium 38-44 arginine vasopressin Homo sapiens 0-11 7143552-4 1982 Bumetanide, given intravenously, spironolactone, frusemide and ADH increased urinary sodium and all except frusemide intravenously decreased faecal sodium regardless of route of administration. Sodium 85-91 arginine vasopressin Homo sapiens 63-66 7091310-8 1982 Taken together, these data affirm in diabetic rats, as in humans, the occurrence of 1) elevated steady-state levels of AVP in serum; 2) abnormal sensitivity of AVP secretion to changes in serum sodium and osmolality; and 3) an apparently intact end-organ responsiveness to AVP. Sodium 194-200 arginine vasopressin Homo sapiens 119-122 7091310-8 1982 Taken together, these data affirm in diabetic rats, as in humans, the occurrence of 1) elevated steady-state levels of AVP in serum; 2) abnormal sensitivity of AVP secretion to changes in serum sodium and osmolality; and 3) an apparently intact end-organ responsiveness to AVP. Sodium 194-200 arginine vasopressin Homo sapiens 160-163 7091310-8 1982 Taken together, these data affirm in diabetic rats, as in humans, the occurrence of 1) elevated steady-state levels of AVP in serum; 2) abnormal sensitivity of AVP secretion to changes in serum sodium and osmolality; and 3) an apparently intact end-organ responsiveness to AVP. Sodium 194-200 arginine vasopressin Homo sapiens 160-163 7072743-5 1982 The elevated ADH levels occurred despite significantly lower plasma sodium concentration (133 +/- 1 meq/liter versus 138 +/- 2 meq/liter, p less than 0.05) compared wit control values. Sodium 68-74 arginine vasopressin Homo sapiens 13-16 7036728-5 1982 A decrease in sodium and water delivery to the ascending limb or in NaCl reabsorption by the ascending limb will impair urinary diluting ability, as will the presence of ADH. Sodium 14-20 arginine vasopressin Homo sapiens 170-173 6290664-0 1982 Enhanced sensitivity to stimulation of sodium transport and cyclic AMP by antidiuretic hormone after Ca2+ depletion of isolated frog skin epithelium. Sodium 39-45 arginine vasopressin Homo sapiens 74-94 6290664-1 1982 The role of Ca2+ in the stimulation by antidiuretic hormone (ADH) of active sodium transport across the isolated epithelium of frog skin was investigated. Sodium 76-82 arginine vasopressin Homo sapiens 39-59 6290664-1 1982 The role of Ca2+ in the stimulation by antidiuretic hormone (ADH) of active sodium transport across the isolated epithelium of frog skin was investigated. Sodium 76-82 arginine vasopressin Homo sapiens 61-64 6280212-1 1982 The effects of diazoxide and hydrochlorothiazide on vasopressin-induced increments in osmotic water flow and sodium transport across the frog bladder were studied. Sodium 109-115 arginine vasopressin Homo sapiens 52-63 7026596-5 1981 The significance of the plasma sodium rise was assessed by observing the vasopressin response to hypoglycemia in a patient shown previously to have a selective loss of the vasopressin response to osmotic stimulation. Sodium 31-37 arginine vasopressin Homo sapiens 172-183 6787206-10 1981 It is concluded that the predominant effect of vasopressin on transepithelial sodium transport is to increase the sodium conductance of the mucosal plasma membrane. Sodium 78-84 arginine vasopressin Homo sapiens 47-58 6271208-0 1981 Effects of antidiuretic hormone on kinetic and energetic determinants of active sodium transport in frog skin. Sodium 80-86 arginine vasopressin Homo sapiens 11-31 7262983-0 1981 Vasopressin elevation in essential hypertension and increased responsiveness to sodium intake. Sodium 80-86 arginine vasopressin Homo sapiens 0-11 670429-3 1978 When serum sodium decreased to 110 mEq/liter, plasma antidiuretic hormone (ADH) was elevated at 30 pg/ml. Sodium 11-17 arginine vasopressin Homo sapiens 53-73 493037-0 1979 [Variations in urinary antidiuretic hormone levels related to sodium intake (author"s transl)]. Sodium 62-68 arginine vasopressin Homo sapiens 23-43 465171-7 1979 Natriuresis with a higher % of the filtered sodium excretion was observed in the malnourished groups and in normal children with 0.5 mU of vasopressin. Sodium 44-50 arginine vasopressin Homo sapiens 139-150 313658-0 1979 Correlation between the mechanism of insulin and vasopressin actions on sodium transport across isolated frog skin. Sodium 72-78 arginine vasopressin Homo sapiens 49-60 6257881-3 1981 Amrinone enhanced the effect of vasopressin and cyclic AMP on water and urea permeabilities, as well as the effect of vasopressin on sodium transport. Sodium 133-139 arginine vasopressin Homo sapiens 118-129 7011672-11 1980 In unusual circumstances, vasopressin levels can occur that are capable of producing a diuresis and increased urine sodium excretion. Sodium 116-122 arginine vasopressin Homo sapiens 26-37 7027063-3 1980 Time-course of plasma ADH following hypertonic saline administration and sensitivity of the response (increase of plasma ADH related to increase of plasma sodium) were not modified. Sodium 155-161 arginine vasopressin Homo sapiens 121-124 7368628-5 1980 Glycosaminoglycans appear to be not only important participants in the effect of the antidiuretic hormone on the permeability of intersticial structures; they also play a certain role in the increase of osmolality of the renal papillar intersticium under the conditions of the antidiuresis, providing the release of loosely bound sodium. Sodium 330-336 arginine vasopressin Homo sapiens 85-105 670429-3 1978 When serum sodium decreased to 110 mEq/liter, plasma antidiuretic hormone (ADH) was elevated at 30 pg/ml. Sodium 11-17 arginine vasopressin Homo sapiens 75-78 418180-0 1978 Vasopressin-like effects of a hallucinogenic drug--harmaline--on sodium and water transport. Sodium 65-71 arginine vasopressin Homo sapiens 0-11 639473-6 1978 During treatment with carbamazepine plasma sodium increased wheras plasma and urine arginine-vasopressin and urine osmolality decreased. Sodium 43-49 arginine vasopressin Homo sapiens 93-104 731678-2 1978 Changes in cellular sodium produced by amiloride, vasopressin, aldosterone, hypoxia, ouabain, and sodium-free media are consistent with a cellular sodium transport pool. Sodium 20-26 arginine vasopressin Homo sapiens 50-61 846069-2 1977 In group, I, normal salt animals hypotonically expanded with antidiuretic hormone (ADH) (using a protocol known to stimulate aldosterone) presented large amounts of sodium to the distal tubule and excreted an acute HCl load much more efficiently than did animals pretreated with either a normal (NL) or low (LO) salt diet alone. Sodium 165-171 arginine vasopressin Homo sapiens 61-81 877126-0 1977 Effect of vasopressin administration on sodium excretion and plasma phosphate concentration. Sodium 40-46 arginine vasopressin Homo sapiens 10-21 407362-8 1977 These observations are in accord with the theory of Leaf and his associates, that the predominant effect of vasopressin is to enhance sodium entry into the transporting epithelial cells of the toad urinary bladder. Sodium 134-140 arginine vasopressin Homo sapiens 108-119 407360-0 1977 Interrelationships of sodium transport and carbon dioxide production by the toad bladder: response to changes in mucosal sodium concentration, to vasopressin and to availability of metabolic substrate. Sodium 22-28 arginine vasopressin Homo sapiens 146-157 407360-2 1977 The rate of sodium transport was varied by changing the concentration of sodium in the mucosal bath (substitution with choline), by adding vasopressin, by adding metabolic substrates and by adding malonate, and the ratio of the change of sodium transport and CO2 production was determined Mean values for deltaNa/deltaCO2 (equiv/mole) were: Na in equilibrium choline 18.3 +/- 1.1; vasopressin 15.5 +/- 2.8; and pyruvate (corrected for the increment in "nontransport" CO2) 15.4 +/- 3.5. Sodium 12-18 arginine vasopressin Homo sapiens 139-150 407360-2 1977 The rate of sodium transport was varied by changing the concentration of sodium in the mucosal bath (substitution with choline), by adding vasopressin, by adding metabolic substrates and by adding malonate, and the ratio of the change of sodium transport and CO2 production was determined Mean values for deltaNa/deltaCO2 (equiv/mole) were: Na in equilibrium choline 18.3 +/- 1.1; vasopressin 15.5 +/- 2.8; and pyruvate (corrected for the increment in "nontransport" CO2) 15.4 +/- 3.5. Sodium 12-18 arginine vasopressin Homo sapiens 381-392 846069-2 1977 In group, I, normal salt animals hypotonically expanded with antidiuretic hormone (ADH) (using a protocol known to stimulate aldosterone) presented large amounts of sodium to the distal tubule and excreted an acute HCl load much more efficiently than did animals pretreated with either a normal (NL) or low (LO) salt diet alone. Sodium 165-171 arginine vasopressin Homo sapiens 83-86 846069-4 1977 Acid excretion was maximized (ADH) when distal sodium avidity was stimulated in the presence of adequate distal sodium delivery and minimized (F) when distal sodium delivery was limited (despite possible augmentation of distal sodium avidity). Sodium 47-53 arginine vasopressin Homo sapiens 30-33 163889-1 1975 It is well established that active sodium-ion transport and water flow across isolated toad bladder are increased by antidiuretic hormone (ADH) and by cAMP. Sodium 35-41 arginine vasopressin Homo sapiens 117-143 190265-0 1977 Effects of ionophore A23187 on base-line and vasopressin-stimulated sodium transport in the toad bladder. Sodium 68-74 arginine vasopressin Homo sapiens 45-56 917179-0 1977 Concentrations of antidiuretic hormone in plasma during human sodium restriction. Sodium 62-68 arginine vasopressin Homo sapiens 18-38 783200-8 1976 In the presently studied patients, the impairment in renal conservation of sodium appeared to be in part the consequence of an impaired ability of the vasopressin-responsive segments of the distal nephron to generate and maintain appropriately steep transepithelial sodium concentration gradients. Sodium 266-272 arginine vasopressin Homo sapiens 151-162 817031-10 1976 However, stimulation of transepithelial sodium transport by vasopressin appeared not to affect the rate of potassium uptake. Sodium 40-46 arginine vasopressin Homo sapiens 60-71 7705-6 1976 We would conclude from these studies that the effect of vasopressin on the luminal cell membrane is a widespread one, modifying both lipid components and components involved in amide, sodium and water transport. Sodium 184-190 arginine vasopressin Homo sapiens 56-67 163889-5 1975 Thus several agents (ADH, cAMP, theophylline, adenine, prostaglandin E1, and Mn Cl-2) caused a decrease in the amount of radioactive phosphate in the 49,000-dalton protein and also stimulated active sodium transport across the bladder. Sodium 199-205 arginine vasopressin Homo sapiens 21-24 4209873-0 1974 Effects of vasopressin on toad bladder membrane proteins: relationship to transport of sodium and water. Sodium 87-93 arginine vasopressin Homo sapiens 11-22 4349529-0 1973 Control of vasopressin stimulation of sodium transport in the toad bladder. Sodium 38-44 arginine vasopressin Homo sapiens 11-22 4542709-0 1973 Effect of antidiuretic hormone on sodium uptake across outer surface of frog skin. Sodium 34-40 arginine vasopressin Homo sapiens 10-30 4351809-5 1973 These and other results are compatible with the possibility that the regulation by vasopressin and cyclic AMP of sodium and/or water transport in toad bladder may be mediated through regulation of the phosphorylation of this specific protein. Sodium 113-119 arginine vasopressin Homo sapiens 83-94 4155453-0 1973 [The influence of catecholamines and synthetic derivatives of vasopressin on the active transport of sodium ions and other bioelectrical properties of the cell membrane (author"s transl)]. Sodium 101-107 arginine vasopressin Homo sapiens 62-73 4693691-0 1973 Vasopressin-related alterations of sodium reabsorption in the loop of Henle. Sodium 35-41 arginine vasopressin Homo sapiens 0-11 4643745-0 1972 [Effect of antidiuretic hormone on energy-coupled transport of sodium in renal epithelial mitochondria]. Sodium 63-69 arginine vasopressin Homo sapiens 11-31 5032511-0 1972 Effects of change in posture and of sodium depletion on plasma levels of vasopressin and renin in normal human subjects. Sodium 36-42 arginine vasopressin Homo sapiens 73-84 4671214-0 1972 Effect of vasopressin on sodium transport in renal cortical collecting tubules. Sodium 25-31 arginine vasopressin Homo sapiens 10-21 5131851-0 1971 Tubule sodium reabsorption in vasopressin escape. Sodium 7-13 arginine vasopressin Homo sapiens 30-41 5116048-0 1971 Electrically evoked release of vasopressin from isolated neurohypophyses in sodium-free media. Sodium 76-82 arginine vasopressin Homo sapiens 31-42 5087392-0 1971 Effects of prolonged administration of vasopressin on plasma sodium and on renal excretion of electrolytes and water. Sodium 61-67 arginine vasopressin Homo sapiens 39-50 4399668-0 1971 Glycine-amide effect on the vasopressin stimulated sodium and water transport across toad bladder. Sodium 51-57 arginine vasopressin Homo sapiens 28-39 24173388-1 1971 Isolated sheets of epithelial cells as well as epithelial cells scraped from paired hemibladders mounted in chambers both showed significant increases in water, sodium and chloride contents after exposure to vasopressin (100 mU/ml), without any change in potassium content. Sodium 161-167 arginine vasopressin Homo sapiens 208-219 24173388-2 1971 In the isolated cells these changes were prevented by amiloride (10(-5) M), suggesting that the gain of sodium after vasopressin occurs across the mucosal membrane. Sodium 104-110 arginine vasopressin Homo sapiens 117-128 5575414-0 1971 Simultaneous measurement of the effect of vasopressin on sodium and water transport across toad bladder. Sodium 57-63 arginine vasopressin Homo sapiens 42-53 5479370-0 1970 The effect of vasopressin on the reabsorption of sodium, potassium and urea by the renal tubules in man. Sodium 49-55 arginine vasopressin Homo sapiens 14-25 4915450-1 1970 Phase microscopy of toad urinary bladder has demonstrated that vasopressin can cause an enlargement of the epithelial intercellular spaces under conditions of no net transfer of water or sodium. Sodium 187-193 arginine vasopressin Homo sapiens 63-74 5821919-0 1969 Effect of vasopressin on sodium and potassium reabsorption by the renal tubules in man. Sodium 25-31 arginine vasopressin Homo sapiens 10-21 5762042-7 1969 The effects of ADH have been interpreted in terms of the formation of water-filled sodium-permselective pores in the outer facing membranes which occupy, at most, 0.3% of the skin surface. Sodium 83-89 arginine vasopressin Homo sapiens 15-18 5773772-0 1969 Effect of vasopressin on the entry of sodium into the renal tubule. Sodium 38-44 arginine vasopressin Homo sapiens 10-21 4304235-6 1968 The results are consistent with ADH causing an increase in permeability of the outer facing membranes to sodium ions.4. Sodium 105-111 arginine vasopressin Homo sapiens 32-35 4304235-12 1968 Separate explanations for the increase in sodium transport by ADH, theophylline and cyclic 3",5"-AMP are discussed. Sodium 42-48 arginine vasopressin Homo sapiens 62-65 5665176-0 1968 Seasonal changes of antidiuretic hormone action on sodium transport across frog skin. Sodium 51-57 arginine vasopressin Homo sapiens 20-40 5658591-4 1968 Application of vasopressin to the bladder serosa shortly after toxin resulted in only weak and transient stimulation of sodium transport; once maximal toxin activity had been established, exposure to the hormone was without effect. Sodium 120-126 arginine vasopressin Homo sapiens 15-26 5664238-7 1968 Vasopressin and aldosterone increased sodium transport from solutions of low Na concentration, but amphotericin B was ineffective until a level of about 40-60 mM was present.4. Sodium 38-44 arginine vasopressin Homo sapiens 0-11 5654401-0 1968 The site of the stimulatory action of vasopressin on sodium transport in toad bladder. Sodium 53-59 arginine vasopressin Homo sapiens 38-49 5654401-1 1968 Vasopressin increases the net transport of sodium across the isolated urinary bladder of the toad by increasing the mobility of sodium ion within the tissue. Sodium 43-49 arginine vasopressin Homo sapiens 0-11 5654401-1 1968 Vasopressin increases the net transport of sodium across the isolated urinary bladder of the toad by increasing the mobility of sodium ion within the tissue. Sodium 128-134 arginine vasopressin Homo sapiens 0-11 5748208-0 1968 [Influence of insulin and vasopressin on sodium distribution in tissues]. Sodium 41-47 arginine vasopressin Homo sapiens 26-37 5840797-5 1965 However, employing an indirect method for the estimation of sodium transport (oxygen consumption), it is possible to show that vasopressin exerts its usual effect on Q(oo2) when sodium is present in the bathing medium. Sodium 60-66 arginine vasopressin Homo sapiens 127-138 5948254-0 1966 The effect of small amounts of antidiuretic hormone on sodium and urate excretion in pregnancy. Sodium 55-61 arginine vasopressin Homo sapiens 31-51 5840797-5 1965 However, employing an indirect method for the estimation of sodium transport (oxygen consumption), it is possible to show that vasopressin exerts its usual effect on Q(oo2) when sodium is present in the bathing medium. Sodium 178-184 arginine vasopressin Homo sapiens 127-138 14485417-0 1962 Influence of hydration on renal function and medullary sodium during vasopressin infusion. Sodium 55-61 arginine vasopressin Homo sapiens 69-80 5860051-0 1965 [Effect of the antidiuretic hormone on the renal excretion of sodium during pregnancy]. Sodium 62-68 arginine vasopressin Homo sapiens 15-35 5846793-0 1965 Action of aldosterone and vasopressin on the active transport of sodium by the isolated toad bladder. Sodium 65-71 arginine vasopressin Homo sapiens 26-37 14101494-0 1963 THE MECHANISM OF INCREASED SODIUM EXCRETION DURING WATER LOADING WITH 2-5 PERCENT DEXTROSE AND VASOPRESSIN. Sodium 27-33 arginine vasopressin Homo sapiens 96-107 14414942-0 1960 Some effects of mammalian neurohypophyseal hormones on metabolism and active transport of sodium by the isolated toad bladder. Sodium 90-96 arginine vasopressin Homo sapiens 26-42 14444029-0 1960 Further observations on hyponatremia and renal sodium loss probably resulting from inappropriate secretion of antidiuretic hormone. Sodium 47-53 arginine vasopressin Homo sapiens 110-130 14437815-0 1959 The syndrome of hyponatremia and renal sodium loss probably resulting from inappropriate secretion of antidiuretic hormone. Sodium 39-45 arginine vasopressin Homo sapiens 102-122 13221777-0 1955 Active sodium uptake by the toad and its response to the antidiuretic hormone. Sodium 7-13 arginine vasopressin Homo sapiens 57-77 14867963-0 1951 Effect of vasopressin (pitressin)-induced water retention on sodium excretion. Sodium 61-67 arginine vasopressin Homo sapiens 10-21 33532104-1 2021 Syndrome of inappropriate antidiuretic hormone (SIADH) is a disorder of fluid and sodium balance characterized by hypotonic hyponatremia, low plasma osmolality, and increased urine osmolality caused by excessive release of antidiuretic hormone (ADH). Sodium 82-88 arginine vasopressin Homo sapiens 26-46 33160031-8 2021 Considering all patients as a whole, copeptin correlated with systolic blood pressure (SBP; rho = -0.209, P = 0.027), eGFR rho = -0.271, P = 0.004), and serum sodium (rho = -0.208, P = 0.027), but not with ABI (rho = -0.068, P = 0.476). Sodium 159-165 arginine vasopressin Homo sapiens 37-45 33597311-8 2021 During the second course of ifosamide, AVP infusion was commenced at the outset and tighter control of urine output and sodium levels was achieved. Sodium 120-126 arginine vasopressin Homo sapiens 39-42 33597311-12 2021 The use of a continuous AVP infusion and titrating with a sliding scale is more effective than oral desmopressin in regulating plasma sodium and fluid balance during hyperhydration therapy. Sodium 134-140 arginine vasopressin Homo sapiens 24-27 33597311-14 2021 Adjustment of the AVP infusion rate depends on urine output, fluid balance, plasma sodium levels and sensitivity/response of the child to titrated AVP doses. Sodium 83-89 arginine vasopressin Homo sapiens 18-21 33532104-1 2021 Syndrome of inappropriate antidiuretic hormone (SIADH) is a disorder of fluid and sodium balance characterized by hypotonic hyponatremia, low plasma osmolality, and increased urine osmolality caused by excessive release of antidiuretic hormone (ADH). Sodium 82-88 arginine vasopressin Homo sapiens 50-53 30205538-1 2018 Changes in serum sodium concentration ([Na+]serum) can permit evaluation of the treatment effect of vasopressin antagonists (vaptans) in patients with worsening heart failure (HF) or cirrhotic ascites; that is, they may act as a treatment stratification biomarker. Sodium 17-23 arginine vasopressin Homo sapiens 100-111 33210088-9 2020 Subsequently, a vasopressin infusion was started with normalisation of diuresis and plasma sodium concentration by the end of surgery. Sodium 91-97 arginine vasopressin Homo sapiens 16-27 31816431-4 2020 RESULTS: - Higher baseline copeptin correlated with higher HbA1c, lower 24-hour urine volume and sodium excretion, after correcting for age, sex, systolic blood pressure, and HbA1c. Sodium 97-103 arginine vasopressin Homo sapiens 27-35 31365096-1 2019 CONTEXT: Although the physiology of sodium, water and arginine vasopressin (AVP), also known as antidiuretic hormone (ADH), has long been known, accumulating data suggest that this system operates as a more complex network than previously thought. Sodium 36-42 arginine vasopressin Homo sapiens 76-79 31365096-1 2019 CONTEXT: Although the physiology of sodium, water and arginine vasopressin (AVP), also known as antidiuretic hormone (ADH), has long been known, accumulating data suggest that this system operates as a more complex network than previously thought. Sodium 36-42 arginine vasopressin Homo sapiens 96-116 31365096-1 2019 CONTEXT: Although the physiology of sodium, water and arginine vasopressin (AVP), also known as antidiuretic hormone (ADH), has long been known, accumulating data suggest that this system operates as a more complex network than previously thought. Sodium 36-42 arginine vasopressin Homo sapiens 118-121 32514438-7 2020 Conclusion: The incidence of hyponatremia is not low during treatment with terlipressin; a higher baseline serum sodium level is a risk factor for hyponatremia during treatment with terlipressin, and the mechanism may be related to endogenous vasopressin preconditioning. Sodium 113-119 arginine vasopressin Homo sapiens 243-254 31339776-8 2019 Furthermore, plasma sodium levels were associated with log2 copeptin (males: st. beta=0.18, P<0.001; females: st. beta=0.17, P<0.001), log2 NT-proBNP (males: st. beta=0.07, P=0.008; females: st. beta=0.12, P<0.001) and log2 aldosterone (males: st. beta= -0.06, P=0.005; females: st. beta= -0.09, P<0.001). Sodium 20-26 arginine vasopressin Homo sapiens 60-68 31339776-9 2019 Copeptin and NT-proBNP showed interaction in their association with plasma sodium. Sodium 75-81 arginine vasopressin Homo sapiens 0-8 31339776-10 2019 Thus, our data support that [1] osmoregulation, as estimated from copeptin levels, is a main associate of plasma sodium, and moreover, [2] show a consistent association with volume markers, with higher NT-proBNP and lower aldosterone in subjects with higher plasma sodium. Sodium 113-119 arginine vasopressin Homo sapiens 66-74 31049746-1 2019 BACKGROUND: A wide range of interesting mathematical models has been derived to predict the effect of intravenous fluid therapy on the serum sodium concentration (most notably the Adrogue-Madias equation), but unfortunately, these models cannot be applied to patients with disorders characterized by aberrant antidiuretic hormone (ADH) release, such as the syndrome of inappropriate ADH secretion (SIADH). Sodium 141-147 arginine vasopressin Homo sapiens 309-329 31150497-12 2019 CONCLUSIONS: Exercise induces a continuous rise of plasma copeptin levels in healthy male volunteers independently of sodium levels and fluid intake. Sodium 118-124 arginine vasopressin Homo sapiens 58-66 32097945-6 2019 Disorders of vasopressin secretion and action result in disturbances of body fluids tonicity, which are clinically recognized as abnormalities in reduced plasma sodium concentration or hyponatremia. Sodium 161-167 arginine vasopressin Homo sapiens 13-24 30984869-0 2018 CENTRAL DIABETES INSIPIDUS AND CHEMOTHERAPY: USE OF A CONTINUOUS ARGININE VASOPRESSIN INFUSION FOR FLUID AND SODIUM BALANCE. Sodium 109-115 arginine vasopressin Homo sapiens 74-85 32826430-11 2020 In exploratory analysis, the association of sodium levels and interleukin-6 levels (which has been linked to nonosmotic release of vasopressin) was assessed. Sodium 44-50 arginine vasopressin Homo sapiens 131-142 32685251-3 2020 Close control and prompt substitution of serum sodium is required in neonates with advanced heart failure on high-dose vasopressin therapy. Sodium 47-53 arginine vasopressin Homo sapiens 119-130 31530276-6 2019 AVP (1 muM) treated PV cardiomyocytes had higher late sodium (Na+) and Na+/Ca2+ exchanger (NCX) currents than control PV cardiomyocytes. Sodium 54-60 arginine vasopressin Homo sapiens 0-3 29664160-12 2018 This suggests that stress- and vasopressin-independent mechanisms are responsible for the association of low sodium levels with mortality. Sodium 109-115 arginine vasopressin Homo sapiens 31-42 30134847-14 2018 CONCLUSIONS: A transient intradialytic increase in plasma sodium did not affect indices of endothelial function or sympathetic activity compared with standard hemodialysis, but coincided with higher plasma vasopressin levels. Sodium 58-64 arginine vasopressin Homo sapiens 206-217 30134847-15 2018 The beneficial effect of higher intradialytic sodium levels on hemodynamic stability might be mediated by vasopressin. Sodium 46-52 arginine vasopressin Homo sapiens 106-117 30627311-4 2018 Treatment with a vasopressin analogue, desamino-D-arginine vasopressin (DDAVP) and forced intake of water restored plasma osmolality and serum sodium levels to normal. Sodium 143-149 arginine vasopressin Homo sapiens 17-28 30627311-4 2018 Treatment with a vasopressin analogue, desamino-D-arginine vasopressin (DDAVP) and forced intake of water restored plasma osmolality and serum sodium levels to normal. Sodium 143-149 arginine vasopressin Homo sapiens 59-70 29079359-10 2018 In multivariate models, race time, serum sodium, creatinine and cortisol were significant predictors of copeptin levels. Sodium 41-47 arginine vasopressin Homo sapiens 104-112 29726784-8 2018 DIND was associated with a combination of decreasing sodium levels and increasing copeptin concentrations.CONCLUSIONSIncreased AVP may be the unifying factor explaining the co-occurrence of hyponatremia and DIND. Sodium 53-59 arginine vasopressin Homo sapiens 127-130 29556787-6 2018 Thirdly, the cooperative action of Ang II acting on AT1R and AVP stimulating both V1aR and V2 receptors in the kidney is necessary for the appropriate regulation of renal blood flow and the efficient resorption of sodium and water. Sodium 214-220 arginine vasopressin Homo sapiens 61-64 29953167-15 2018 Vasopressin receptor antagonists probably increase the intermediate outcome of serum sodium concentration (21 studies, 2641 participants: MD 4.17 mmol/L, 95% CI 3.18 to 5.16), corresponding to two and a half as many people having a 5 to 6 mmol/L increase in sodium concentration compared with placebo at 4 to 180 days (moderate certainty evidence due to risk of attrition bias) (18 studies, 2014 participants: RR 2.49, 95% CI 1.95 to 3.18). Sodium 85-91 arginine vasopressin Homo sapiens 0-11 29953167-15 2018 Vasopressin receptor antagonists probably increase the intermediate outcome of serum sodium concentration (21 studies, 2641 participants: MD 4.17 mmol/L, 95% CI 3.18 to 5.16), corresponding to two and a half as many people having a 5 to 6 mmol/L increase in sodium concentration compared with placebo at 4 to 180 days (moderate certainty evidence due to risk of attrition bias) (18 studies, 2014 participants: RR 2.49, 95% CI 1.95 to 3.18). Sodium 258-264 arginine vasopressin Homo sapiens 0-11 29953167-19 2018 AUTHORS" CONCLUSIONS: In people with chronic hyponatraemia, vasopressin receptor antagonists modestly raise serum sodium concentration at the cost of a 3% increased risk of it being rapid. Sodium 114-120 arginine vasopressin Homo sapiens 60-71 29512852-7 2018 These purinergic receptor subtypes are permeable to sodium vs. calcium in varying amounts and this could play an important role in the release of vasopressin vs. oxytocin during bursting activity. Sodium 52-58 arginine vasopressin Homo sapiens 146-157 27742514-5 2017 We reviewed the clinical histories, intensive care unit treatment, medication administration records, and laboratory values of these patients, and we found recurrent elevated serum sodium and urine output and decreased urine specific gravity after discontinuation of AVP. Sodium 181-187 arginine vasopressin Homo sapiens 267-270 28649750-1 2017 Vasopressin (AVP) plays a major role in the regulation of water and sodium homeostasis by its antidiuretic action on the kidney, mediated by V2 receptors. Sodium 68-74 arginine vasopressin Homo sapiens 0-11 28649750-1 2017 Vasopressin (AVP) plays a major role in the regulation of water and sodium homeostasis by its antidiuretic action on the kidney, mediated by V2 receptors. Sodium 68-74 arginine vasopressin Homo sapiens 13-16 28503166-9 2017 Phenytoin at a concentration of 25 microg/mL also decreased AVP mRNA levels in Lu-165 cells under the sodium challenge with added sodium at 10 mEq/L, but not at 20 mEq/L. Sodium 102-108 arginine vasopressin Homo sapiens 60-63 28503166-9 2017 Phenytoin at a concentration of 25 microg/mL also decreased AVP mRNA levels in Lu-165 cells under the sodium challenge with added sodium at 10 mEq/L, but not at 20 mEq/L. Sodium 130-136 arginine vasopressin Homo sapiens 60-63 28503166-12 2017 These results suggest that Lu-165 cells are sensitive to phenytoin and sodium to control of AVP release and its gene expression. Sodium 71-77 arginine vasopressin Homo sapiens 92-95 27882774-5 2017 Urinary sodium excretion to plasma copeptin (copeptin/UNa Secretion) ratio was significantly lower in Syndrome of Inappropriate Antidiuretic Hormone (SIADH) Secretion patients than in patients with hyponatremia of other origin. Sodium 8-14 arginine vasopressin Homo sapiens 35-43 27882774-5 2017 Urinary sodium excretion to plasma copeptin (copeptin/UNa Secretion) ratio was significantly lower in Syndrome of Inappropriate Antidiuretic Hormone (SIADH) Secretion patients than in patients with hyponatremia of other origin. Sodium 8-14 arginine vasopressin Homo sapiens 45-53 28127476-15 2017 A protocol using dilute vasopressin bolus can be an alternative for managing acute, central DI postoperatively, particularly in setting of hypovolemic shock resulting in a consistent control of serum sodium. Sodium 200-206 arginine vasopressin Homo sapiens 24-35 27677909-6 2017 RESULTS: Patients treated with vasopressin in addition to other vasoactive drugs had significantly higher sodium changes compared to those treated with other vasoactive drugs (-4.7 +- 6 vs -0.1 +- 2.4 mmol/L, respectively, p value 0.001). Sodium 106-112 arginine vasopressin Homo sapiens 31-42 27677909-10 2017 Serum sodium should be monitored closely when vasopressin is being used in the SAH population. Sodium 6-12 arginine vasopressin Homo sapiens 46-57 27677909-11 2017 Further studies are needed to confirm the effect of exogenous vasopressin on serum sodium levels in SAH populations. Sodium 83-89 arginine vasopressin Homo sapiens 62-73 27631841-11 2016 Sodium levels were found correlated with copeptin levels; yet, an even stronger correlation was found between copeptin levels and APACHE II score (r = 0.52; P<.001). Sodium 0-6 arginine vasopressin Homo sapiens 41-49 27188842-8 2016 We review evidence from experiments conducted in vivo and in cultured cells that indicates aldosterone and vasopressin, the two major hormones regulating sodium reabsorption by CD, generate a coordinated stimulation of apical ENaC and basolateral Na(+),K(+)-ATPase. Sodium 154-160 arginine vasopressin Homo sapiens 107-118 27037930-2 2016 The main reason for this abnormality is related to the fact that these patients have portal hypertension and this leads to systemic vasodilation that in turn activates sodium-retaining and water-retaining systems such as the renin-angiotensin-aldosterone system and arginine vasopressin (AVP). Sodium 168-174 arginine vasopressin Homo sapiens 275-286 27037930-2 2016 The main reason for this abnormality is related to the fact that these patients have portal hypertension and this leads to systemic vasodilation that in turn activates sodium-retaining and water-retaining systems such as the renin-angiotensin-aldosterone system and arginine vasopressin (AVP). Sodium 168-174 arginine vasopressin Homo sapiens 288-291 27156761-7 2016 The copeptin to urinary sodium ratio differentiates accurately between volume-depleted and normovolemic disorders. Sodium 24-30 arginine vasopressin Homo sapiens 4-12 27274855-6 2016 Treatment of diabetes insipidus (DI) with replacement antidiuretic hormone (ADH) was complicated by extreme difficulties in achieving a satisfactory sodium and water balance. Sodium 149-155 arginine vasopressin Homo sapiens 54-80 26041443-0 2015 Vasopressin regulation of sodium transport in the distal nephron and collecting duct. Sodium 26-32 arginine vasopressin Homo sapiens 0-11 26311597-5 2015 Although currently available vasopressin (V2 receptors) antagonists have been shown to increase serum sodium concentrations and improve ascites control, their role in the treatment of hyponatremia in liver disease patients remains questionable because of adverse effect profiles, high cost, and poor data on long-term mortality benefits. Sodium 102-108 arginine vasopressin Homo sapiens 29-40 28124939-1 2016 During the past 50 years the molecular mechanisms of renal reabsorption of sodium and water have been described and molecules specifically interfering with these mechanisms have been developed (diuretics, vasopressin receptor antagonists). Sodium 75-81 arginine vasopressin Homo sapiens 205-216 28124939-3 2016 Vaptans (vasopressin antagonists, vasopressin V2-receptor inhibitors) reduce reabsorption of water in the distal nephron, they increase free water excretion and normalize serum concentrations of sodium in normovolemic and hypervolemic conditions associated with hyponatremia. Sodium 195-201 arginine vasopressin Homo sapiens 9-20 26041443-4 2015 The antinatriuretic effects of AVP are mediated by the regulation of sodium transport throughout the distal nephron, from the thick ascending limb through to the collecting duct, which in turn partially facilitates osmotic movement of water. Sodium 69-75 arginine vasopressin Homo sapiens 31-34 25500109-15 2015 CONCLUSIONS: Important lifestyle- and diet-related factors associated with copeptin concentration are current smoking, alcohol use, protein and potassium intake, and particularly fluid and sodium intake. Sodium 189-195 arginine vasopressin Homo sapiens 75-83 26142600-10 2015 T&A led to normalization of ADH and BNP, probably through a calcium- and sodium-dependent mechanism. Sodium 77-83 arginine vasopressin Homo sapiens 32-35 25252899-2 2015 This study was designed to assess the usefulness of copeptin measurement in children with community-acquired pneumonia (CAP) and copeptin"s relation with disease severity and sodium equilibrium. Sodium 175-181 arginine vasopressin Homo sapiens 52-60 25252899-2 2015 This study was designed to assess the usefulness of copeptin measurement in children with community-acquired pneumonia (CAP) and copeptin"s relation with disease severity and sodium equilibrium. Sodium 175-181 arginine vasopressin Homo sapiens 129-137 25252899-8 2015 Yet there was only a weak reverse correlation between the sodium and the copeptin concentrations (Spearmann"s rank coefficient = -0.19). Sodium 58-64 arginine vasopressin Homo sapiens 73-81 23392370-7 2013 Mean lowest sodium in the first 72 hours, however, was 134.7 +- 3.8 in those who received arginine vasopressin as compared with 137.1 +- 4.3 in the control group, p = 0.019. Sodium 12-18 arginine vasopressin Homo sapiens 99-110 25374805-5 2014 Increased levels of antidiuretic hormone can result in hyponatremia, defined as a plasma sodium level < 136 mmol/L, which causes cells to draw in excess water and swell. Sodium 89-95 arginine vasopressin Homo sapiens 20-40 24408226-11 2014 The findings suggest that appropriate AVP secretion in response to an increase in serum osmolality caused by renal sodium retention is necessary for excess extracellular fluid accumulation in NS. Sodium 115-121 arginine vasopressin Homo sapiens 38-41 24511399-8 2014 The following case report analyzes the role of vasopressin antagonists in the treatment of hyponatremia and the need for daily dosing of tolvaptan and the monitoring of serum sodium levels to avoid rapid overcorrection which can result in osmotic demyelination syndrome (ODS). Sodium 175-181 arginine vasopressin Homo sapiens 47-58 24840297-2 2014 Extracellular fluid (ECF) sodium concentrations ([Na]) reflect the overall body sodium content, but are also influenced by the osmoregulatory system, which is regulated by the posterior pituitary hormone arginine vasopressin (AVP). Sodium 26-32 arginine vasopressin Homo sapiens 213-224 24923603-2 2014 CASE PRESENTATION: We describe the case of a 63-year-old Korean male with inappropriate antidiuretic hormone syndrome due to an ectopic antidiuretic hormone-producing advanced gastric adenocarcinoma manifested with overt serum hypo-osmolar hyponatremia and high urinary sodium concentrations. Sodium 270-276 arginine vasopressin Homo sapiens 88-108 22959761-3 2012 Serum sodium concentration is linked to water homeostasis, which is dependent on the thirst mechanism, arginine vasopressin, and kidney function. Sodium 6-12 arginine vasopressin Homo sapiens 112-123 24558627-5 2013 Syndrome of inappropriate antidiuretic hormone hypersecretion (SIADH) and hypervolemic hyponatremia caused by heart failure or cirrhosis are treated with vasopressin antagonists (vaptans) since they increase plasma sodium (Na(2+)) concentration via their aquaretic effects (augmentation of free-water clearance). Sodium 215-221 arginine vasopressin Homo sapiens 154-165 23018767-7 2013 At baseline, serum sodium concentration negatively correlated with pulmonary capillary wedge pressure (r=-0.71, P<0.001) and with plasma arginine vasopressin level (r=-0.68, P=0.008). Sodium 19-25 arginine vasopressin Homo sapiens 149-160 23514343-7 2013 The underfilling is sensed by the arterial baroreceptors, which activate primarily the sympathetic nervous system and renin-angiotensin-aldosterone system, as well as vasopressin, to restore the arterial filling by increasing the vascular tone and retaining sodium and water. Sodium 258-264 arginine vasopressin Homo sapiens 167-178 22771918-4 2012 Treatment with vasopressin allowed a normalization of sodium levels and urine output. Sodium 54-60 arginine vasopressin Homo sapiens 15-26 22765308-1 2012 AIM: To investigate the sodium composition of maintenance intravenous fluids (mIVF) used by paediatric residents throughout the United States in common clinical scenarios of arginine vasopressin (AVP) excess. Sodium 26-32 arginine vasopressin Homo sapiens 185-196 21233458-1 2011 BACKGROUND AND OBJECTIVES: Acute infusion of the potent V2 receptor agonist 1-desamino-8-d-arginine vasopressin (dDAVP) reduces sodium excretion in humans, through an effect attributed to the stimulation of the amiloride sensitive epithelial sodium channel, ENaC, in ex vivo/in vivo experiments. Sodium 128-134 arginine vasopressin Homo sapiens 100-111 21925797-3 2011 Elevated vasopressin is also the most common cause of low blood sodium (hyponatremia)--most serious in the brains of children. Sodium 64-70 arginine vasopressin Homo sapiens 9-20 23776817-1 2012 Arginine vasopressin (AVP) plays an important role in water and sodium homeostasis. Sodium 64-70 arginine vasopressin Homo sapiens 9-20 22555263-6 2012 In multivariate Cox proportional hazard models adjusted for confounders, including plasma sodium, loop diuretic dose, and NT-proBNP, copeptin was a significant predictor of hospitalization or death (hazard ratio 1.4, 95% confidence interval 1.1-1.9; P < .019) but did not predict mortality independently from NT-proBNP. Sodium 90-96 arginine vasopressin Homo sapiens 133-141 22555263-8 2012 CONCLUSIONS: Plasma copeptin levels predict mortality in outpatients with chronic heart failure independently from clinical variables, plasma sodium, and loop diuretic doses. Sodium 142-148 arginine vasopressin Homo sapiens 20-28 21926664-11 2012 The lowest serum sodium in the first 48 hrs was significantly lower in arginine vasopressin+ (132 vs. 137 mmol/L, p = .01). Sodium 17-23 arginine vasopressin Homo sapiens 80-91 22378093-0 2012 Plasma arginine vasopressin level in hypothyroid women in relation to dietary sodium supply. Sodium 78-84 arginine vasopressin Homo sapiens 16-27 22378093-8 2012 As a result of the low sodium diet and the upright position, a significant increase of arginine vasopressin secretion was observed in the control group and hypothyroid women in the euthyroid state. Sodium 23-29 arginine vasopressin Homo sapiens 96-107 21990032-0 2011 The use of copeptin, the stable peptide of the vasopressin precursor, in the differential diagnosis of sodium imbalance in patients with acute diseases. Sodium 103-109 arginine vasopressin Homo sapiens 11-19 21990032-0 2011 The use of copeptin, the stable peptide of the vasopressin precursor, in the differential diagnosis of sodium imbalance in patients with acute diseases. Sodium 103-109 arginine vasopressin Homo sapiens 47-58 21990032-5 2011 This study assessed copeptin concentrations in sick patients with serum sodium imbalance of different aetiology on admission to the emergency department. Sodium 72-78 arginine vasopressin Homo sapiens 20-28 21187188-1 2011 BACKGROUND: low serum sodium concentrations are associated with an increased risk of death in the general population, but causality is uncertain due to confounding from clinical conditions such as congestive heart failure and cirrhosis, in which hyponatremia results from elevated levels of arginine vasopressin. Sodium 22-28 arginine vasopressin Homo sapiens 300-311 21283124-4 2011 Probably as a result of this rise in plasma sodium, thirst and plasma vasopressin concentration increase, potentionally limiting the effects of the vasopressin antagonists. Sodium 44-50 arginine vasopressin Homo sapiens 148-159 21617327-5 2011 Second, the relationship between serum sodium level and plasma AVP was investigated. Sodium 39-45 arginine vasopressin Homo sapiens 63-66 21617327-7 2011 Plasma AVP level was higher in the hyponatremia group (4.5 vs. 2.7 pg/ml, P=0.003), and it had a negative correlation with serum sodium level (r=-0.28, P=0.02). Sodium 129-135 arginine vasopressin Homo sapiens 7-10 21603106-7 2011 Thus far, clinical trials with vasopressin-receptor antagonists have demonstrated an increase in free-water excretion, improvement in serum sodium, modest improvements in dyspnea but no improvement in mortality. Sodium 140-146 arginine vasopressin Homo sapiens 31-42 20736986-0 2010 Vasopressin regulation of renal sodium excretion. Sodium 32-38 arginine vasopressin Homo sapiens 0-11 20953635-2 2011 We evaluated the relationship between the change in serum sodium and AVP levels following percutaneous renal biopsy in children receiving either hypotonic or isotonic fluids. Sodium 58-64 arginine vasopressin Homo sapiens 69-72 20953635-6 2011 Patients receiving 0.6% NaCl with elevated AVP levels experienced a fall in serum sodium of 1.9 +- 1.5 mEq/L, whereas those receiving 0.9% NaCl had a rise in serum sodium of 0.85 +- 0.34 mEq/L with no patients developing hyponatremia. Sodium 82-88 arginine vasopressin Homo sapiens 43-46 20719429-7 2010 He was managed with parenteral fluids and a regimen of intranasal DDAVP (1-desamino 8-d-arginine vasopressin), leading to improved plasmatic sodium levels, urine output, and urinary specific gravity. Sodium 141-147 arginine vasopressin Homo sapiens 97-108 20826569-9 2010 This led us to discuss how excessive vasopressin-dependent ENaC stimulation could be a risk factor for sodium retention and resulting increase in blood pressure. Sodium 103-109 arginine vasopressin Homo sapiens 37-48 20736986-9 2010 Thus, we need to modify slightly our understanding of vasopressin and its renal actions to include the idea that while vasopressin decreases free water excretion to dilute plasma, it does this, in part, by promoting sodium reabsorption and consequently decreasing sodium excretion via ENaC activated along the distal nephron. Sodium 264-270 arginine vasopressin Homo sapiens 119-130 20736986-3 2010 In comparison, vasopressin actions on renal electrolytes including sodium excretion and its consequences have been less well understood. Sodium 67-73 arginine vasopressin Homo sapiens 15-26 20736986-7 2010 Moreover, although vasopressin action on renal sodium excretion via the V2 receptor is critical to water transport, it is masked easily being situational--for instance, dependent on hydration state. Sodium 47-53 arginine vasopressin Homo sapiens 19-30 20736986-8 2010 It is now clear that an increase in sodium reabsorption along the distal nephron (CNT + CD) mediated by activation of the epithelial Na(+) channel (ENaC) by vasopressin makes an important contribution to maintenance of the axial corticomedullary osmotic gradient necessary for maximal water reabsorption. Sodium 36-42 arginine vasopressin Homo sapiens 157-168 20736986-9 2010 Thus, we need to modify slightly our understanding of vasopressin and its renal actions to include the idea that while vasopressin decreases free water excretion to dilute plasma, it does this, in part, by promoting sodium reabsorption and consequently decreasing sodium excretion via ENaC activated along the distal nephron. Sodium 216-222 arginine vasopressin Homo sapiens 54-65 20736986-9 2010 Thus, we need to modify slightly our understanding of vasopressin and its renal actions to include the idea that while vasopressin decreases free water excretion to dilute plasma, it does this, in part, by promoting sodium reabsorption and consequently decreasing sodium excretion via ENaC activated along the distal nephron. Sodium 216-222 arginine vasopressin Homo sapiens 119-130 20639747-7 2010 A slope of the relation between arginine vasopressin and plasma sodium levels of < 0.5 pg/mEq defined nonresponders. Sodium 64-70 arginine vasopressin Homo sapiens 41-52 21180368-4 2010 Tolvaptan antagonises receptors for arginine vasopressin, a hormone that regulates blood sodium levels by stimulating renal water resabsorption. Sodium 89-95 arginine vasopressin Homo sapiens 45-56 20524712-8 2010 Inappropriately, high plasma AVP concentrations ([AVP](p)) associated with low blood sodium concentrations facilitate fluid retention and sodium loss, thereby possibly reconciling both the water intoxication and sodium loss theories of hyponatraemia that are currently under debate. Sodium 85-91 arginine vasopressin Homo sapiens 50-53 20539224-8 2010 Recently approved vasopressin antagonists now make it possible to normalize the serum sodium concentration on a chronic basis, but practical considerations have limited their use. Sodium 86-92 arginine vasopressin Homo sapiens 18-29 20812145-1 2010 Vasopressin (also known as arginine vasopressin [AVP]) is a small cyclic peptide that acts at the V1a, V1b and V2 GPCRs to regulate a wide range of physiological functions, including vasoconstriction, smooth muscle contractility, response to stress, and excretion of water and sodium via the kidney. Sodium 277-283 arginine vasopressin Homo sapiens 0-11 20812145-1 2010 Vasopressin (also known as arginine vasopressin [AVP]) is a small cyclic peptide that acts at the V1a, V1b and V2 GPCRs to regulate a wide range of physiological functions, including vasoconstriction, smooth muscle contractility, response to stress, and excretion of water and sodium via the kidney. Sodium 277-283 arginine vasopressin Homo sapiens 36-47 20538391-11 2010 Although there was an increased rate of rapid sodium correction (RR, 2.52; 95% CI, 1.26-5.08, 8 trials) with vasopressin antagonists, hypernatremia rates were not significantly higher (RR, 2.21; 95% CI, 0.61-7.96; 5 trials), adverse events were not increased, and there were no reports of osmotic demyelination syndrome. Sodium 46-52 arginine vasopressin Homo sapiens 109-120 20608824-2 2010 The result of elevated vasopressin is renal water retention and hyponatremia, a low serum sodium concentration. Sodium 90-96 arginine vasopressin Homo sapiens 23-34 20524712-8 2010 Inappropriately, high plasma AVP concentrations ([AVP](p)) associated with low blood sodium concentrations facilitate fluid retention and sodium loss, thereby possibly reconciling both the water intoxication and sodium loss theories of hyponatraemia that are currently under debate. Sodium 138-144 arginine vasopressin Homo sapiens 29-32 20524712-8 2010 Inappropriately, high plasma AVP concentrations ([AVP](p)) associated with low blood sodium concentrations facilitate fluid retention and sodium loss, thereby possibly reconciling both the water intoxication and sodium loss theories of hyponatraemia that are currently under debate. Sodium 138-144 arginine vasopressin Homo sapiens 50-53 20524712-8 2010 Inappropriately, high plasma AVP concentrations ([AVP](p)) associated with low blood sodium concentrations facilitate fluid retention and sodium loss, thereby possibly reconciling both the water intoxication and sodium loss theories of hyponatraemia that are currently under debate. Sodium 138-144 arginine vasopressin Homo sapiens 29-32 20524712-8 2010 Inappropriately, high plasma AVP concentrations ([AVP](p)) associated with low blood sodium concentrations facilitate fluid retention and sodium loss, thereby possibly reconciling both the water intoxication and sodium loss theories of hyponatraemia that are currently under debate. Sodium 138-144 arginine vasopressin Homo sapiens 50-53 20467590-2 2010 In response to elevated serum osmolality, vasopressin acts on V2 renal receptors increasing water reabsorption and causing serum sodium to decrease. Sodium 129-135 arginine vasopressin Homo sapiens 42-53 19572935-9 2009 RBF decreased and GFR, filtration fraction, sodium reabsorption, RVO2, RO2Ex and renal vascular resistance increased dose-dependently with vasopressin. Sodium 44-50 arginine vasopressin Homo sapiens 139-150 20427980-9 2010 Vasopressin receptor antagonists have also been shown to be effective at increasing serum sodium levels in patients with either euvolemic or hypervolemic hyponatremia and represent another therapeutic option. Sodium 90-96 arginine vasopressin Homo sapiens 0-11 21468184-6 2009 Activation of sympathetic nervous system, renin-angiotensin-aldosterone system and non-osmotic vasopressin release stimulate the renal tubular reabsorption of sodium and water. Sodium 159-165 arginine vasopressin Homo sapiens 95-106 18984663-6 2009 In controls, a close correlation between plasma copeptin and serum sodium (r(2) = 0.62, P < 0.001) or urine osmolality (r(2) = 0.39, P = 0.001) was observed. Sodium 67-73 arginine vasopressin Homo sapiens 48-56 19132338-5 2009 Vasopressin infusion was associated with an 8 mmol/L fall in serum sodium concentration (p < 0.01) and with higher incidence of hyponatraemia (8 vs. 66%, p < 0.01). Sodium 67-73 arginine vasopressin Homo sapiens 0-11 19132338-8 2009 CONCLUSION: Low-dose vasopressin infusion in haemodynamically stable, but critically ill, children is associated with reduction in urine output and decreased serum sodium level, yielding a high incidence of hyponatraemia. Sodium 164-170 arginine vasopressin Homo sapiens 21-32 19412798-0 2009 Plasma vasopressin, oxytocin, estradiol, and progesterone related to water and sodium excretion in normal pregnancy and gestational hypertension. Sodium 79-85 arginine vasopressin Homo sapiens 7-18 18985840-1 2008 The syndrome of inappropriate antidiuretic hormone secretion (SIADH) is a disorder of sodium and water balance characterized by hypotonic hyponatremia and impaired water excretion in the absence of renal insufficiency , adrenal insufficiency or any recognized stimulus for the antidiuretic hormone (ADH). Sodium 86-92 arginine vasopressin Homo sapiens 30-50 18985840-1 2008 The syndrome of inappropriate antidiuretic hormone secretion (SIADH) is a disorder of sodium and water balance characterized by hypotonic hyponatremia and impaired water excretion in the absence of renal insufficiency , adrenal insufficiency or any recognized stimulus for the antidiuretic hormone (ADH). Sodium 86-92 arginine vasopressin Homo sapiens 64-67 18596120-0 2008 Sodium excretion in response to vasopressin and selective vasopressin receptor antagonists. Sodium 0-6 arginine vasopressin Homo sapiens 32-43 18596120-0 2008 Sodium excretion in response to vasopressin and selective vasopressin receptor antagonists. Sodium 0-6 arginine vasopressin Homo sapiens 58-69 18596120-1 2008 The mechanisms by which arginine vasopressin (AVP) exerts its antidiuretic and pressor effects, via activation of V2 and V1a receptors, respectively, are relatively well understood, but the possible associated effects on sodium handling are a matter of controversy. Sodium 221-227 arginine vasopressin Homo sapiens 33-44 18596120-6 2008 Vasopressin reduced sodium excretion at 1 mug/kg but increased it at doses >5 umg/kg,an effect that was abolished by the V1a antagonist. Sodium 20-26 arginine vasopressin Homo sapiens 0-11 18596120-8 2008 In the usual range of regulation, antidiuretic effects of vasopressin may be associated with variable sodium retention. Sodium 102-108 arginine vasopressin Homo sapiens 58-69 18560669-4 2008 Hormonal mechanisms including the renin-angiotensin system, aldosterone, and vasopressin are involved in modifying fetal renal excretion, reabsorption of sodium and water, and regulation of vascular volume. Sodium 154-160 arginine vasopressin Homo sapiens 77-88 18719325-2 2008 Endocrinopathy had well been controlled by hormone replacement therapy and administration of 1-amino-8-d-arginine-vasopressin with serum sodium concentration within the normal range. Sodium 137-143 arginine vasopressin Homo sapiens 114-125 18436727-7 2008 Because of their effects on sodium, vasopressin antagonists need to be carefully monitored to ensure that serum sodium levels do not increase too quickly and put the patient at risk for overcorrection or osmotic demyelination syndrome. Sodium 112-118 arginine vasopressin Homo sapiens 36-47