PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22036635-1	2012	BCR-ABL1 kinase-positive leukemia cells accumulate high numbers of DNA double-strand breaks (DSBs) induced by the reactive oxygen species (ROS) or cytotoxic agents.	dsbs	93-97	BCR activator of RhoGEF and GTPase	Homo sapiens	0-8	
22036635-5	2012	Moreover, Ac-H3 and Ac-H4 readily co-localized with the spontaneous and mitomycin C-induced DSBs in BCR-ABL1-positive leukemia cells suggesting that histone acetylation and chromatin re-modeling is important for efficient repair of numerous DSBs.	dsbs	92-96	BCR activator of RhoGEF and GTPase	Homo sapiens	100-108	
16297667-2	2006	We find that after gamma-irradiation BCR/ABL-positive leukemia cells accumulate more DSBs in comparison to normal cells.	dsbs	85-89	BCR activator of RhoGEF and GTPase	Homo sapiens	37-40