PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30224358-7	2018	Using serine- or glutamate-containing HCF-1PRO-repeat sequences, we show that proposed OGT-based or UDP-GlcNAc-based serine-acceptor residue activation mechanisms can be circumvented independently, but not when disrupted together.	Glutamic Acid	17-26	O-linked N-acetylglucosamine (GlcNAc) transferase	Homo sapiens	87-90	
30224358-2	2018	OGT thereby stably glycosylates serines and threonines of numerous proteins and, via a transient glutamate glycosylation, cleaves a single known substrate-the so-called HCF-1PRO repeat of the transcriptional co-regulator host-cell factor 1 (HCF-1).	Glutamic Acid	97-106	O-linked N-acetylglucosamine (GlcNAc) transferase	Homo sapiens	0-3	
26305326-7	2015	The glutamate residue at the cleavage site that is intimately involved in the cleavage reaction specifically inhibits association with OGT and its bound cofactor UDP-GlcNAc.	Glutamic Acid	4-13	O-linked N-acetylglucosamine (GlcNAc) transferase	Homo sapiens	135-138	
28470537-0	2018	Erratum to: O-GlcNAc Glycosylation of nNOS Promotes Neuronal Apoptosis Following Glutamate Excitotoxicity.	Glutamic Acid	81-90	O-linked N-acetylglucosamine (GlcNAc) transferase	Homo sapiens	12-20	
28207246-4	2017	We report here that OGT glycosylates aspartate much faster than it glycosylates glutamate in an otherwise identical model peptide substrate; moreover, once formed, the glycosyl aspartate reacts further to form a succinimide intermediate that hydrolyzes to produce the corresponding isoaspartyl peptide.	Glutamic Acid	80-89	O-linked N-acetylglucosamine (GlcNAc) transferase	Homo sapiens	20-23	
26197318-3	2015	Here we found that nitric oxide synthase adaptor (NOS1AP), which plays an important part in glutamate-induced neuronal apoptosis, carries the modification of O-GlcNAc.	Glutamic Acid	92-101	O-linked N-acetylglucosamine (GlcNAc) transferase	Homo sapiens	158-166	
26197318-5	2015	Higher O-GlcNAc of NOS1AP was detected during glutamate-induced neuronal apoptosis.	Glutamic Acid	46-55	O-linked N-acetylglucosamine (GlcNAc) transferase	Homo sapiens	7-15	
26197318-7	2015	Finally, during glutamate-induced neuronal apoptosis, decreasing the O-GlcNAc modification of NOS1AP results in more severe neuronal apoptosis.	Glutamic Acid	16-25	O-linked N-acetylglucosamine (GlcNAc) transferase	Homo sapiens	69-77	
26197318-8	2015	All these results suggest that O-GlcNAc modification of NOS1AP exerts protective effects during glutamate-induced neuronal apoptosis.	Glutamic Acid	96-105	O-linked N-acetylglucosamine (GlcNAc) transferase	Homo sapiens	31-39