PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23568759-1	2013	The dihydropyrimidinase-like 3 (DPYSL3) or Collapsin Response Mediator Protein 4a (CRMP4a) expression is modified in neurodegeneration and is involved in several ALS-associated pathways including axonal transport, glutamate excitotoxicity, and oxidative stress.	Glutamic Acid	214-223	dihydropyrimidinase like 3	Homo sapiens	4-30	
23568759-1	2013	The dihydropyrimidinase-like 3 (DPYSL3) or Collapsin Response Mediator Protein 4a (CRMP4a) expression is modified in neurodegeneration and is involved in several ALS-associated pathways including axonal transport, glutamate excitotoxicity, and oxidative stress.	Glutamic Acid	214-223	dihydropyrimidinase like 3	Homo sapiens	32-38	
16987501-12	2006	These results indicate that changes in Ca(2+) homeostasis resulting from ROS-dependent activation of L-VGCC are sufficient to induce probable calpain-mediated DPYSL3 truncation and demonstrate for the first time the role of ROS in the mechanism leading to glutamate-induced calpain activation and DPYSL3 protein degradation.	Glutamic Acid	256-265	dihydropyrimidinase like 3	Homo sapiens	159-165	
16135096-2	2005	In primary cortical cultures, glutamate (NMDA) excitotoxicity and oxidative stress (H2O2) caused the cleavage of DPYSL3, resulting in the appearance of a doublet of 62 kDa and 60 kDa.	Glutamic Acid	30-39	dihydropyrimidinase like 3	Homo sapiens	113-119