PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31073146-0	2019	The selective disruption of presynaptic JNK2/STX1a interaction reduces NMDA receptor-dependent glutamate release.	Glutamic Acid	95-104	syntaxin 1A	Homo sapiens	45-50	
31073146-4	2019	Our previous work showed that presynaptic c-Jun N-terminal kinase 2 (JNK2) specifically regulates NMDA-evoked glutamate release and here we demonstrate that an interaction between Syntaxin-1a and JNK2 is fundamental to this mechanism.	Glutamic Acid	110-119	syntaxin 1A	Homo sapiens	180-191	
31073146-7	2019	In conclusion, the JNK2/STX1 interaction is involved in presynaptic NMDA-evoked glutamate release and the novel CPP, JGRi1, acts as a pharmacological tool that promotes neuroprotection.	Glutamic Acid	80-89	syntaxin 1A	Homo sapiens	24-28	
16959903-0	2006	Syntaxin 1A promotes the endocytic sorting of EAAC1 leading to inhibition of glutamate transport.	Glutamic Acid	77-86	syntaxin 1A	Homo sapiens	0-11	
11312277-6	2001	Activation of protein kinase C via phorbol esters or site-directed mutagenesis of three putative PKC consensus sites in the syntaxin 1A binding region of the channel (S802, S896, S898) to glutamic acid (to mimic a permanently phosphorylated state) did not affect the syntaxin 1A-mediated G-protein modulation of the channel.	Glutamic Acid	188-201	syntaxin 1A	Homo sapiens	124-135	
11312277-6	2001	Activation of protein kinase C via phorbol esters or site-directed mutagenesis of three putative PKC consensus sites in the syntaxin 1A binding region of the channel (S802, S896, S898) to glutamic acid (to mimic a permanently phosphorylated state) did not affect the syntaxin 1A-mediated G-protein modulation of the channel.	Glutamic Acid	188-201	syntaxin 1A	Homo sapiens	267-278