PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15495159-3	2004	In this study, we observed that exogenous peptides that were artificially fused with an endoplasmic reticulum (ER) retrieval signal, a C-terminal Lys-Asp-Glu-Leu sequence, could be efficiently presented by intracellular MHC class I molecules in a TAP- and proteasome-independent, but brefeldin A-sensitive manner.	Glutamic Acid	154-157	major histocompatibility complex, class I, C	Homo sapiens	220-223	
9271317-2	1997	The synthetic random copolymer of the amino acids, L-Glu, L-Lys, L-Ala and L-Tyr, termed GLAT, with promiscuous binding to multiple MHC class II alleles, reduces the incidence, onset and severity of disease in the BIO.D2 --> BALB/c model of lethal GVHD.	Glutamic Acid	51-56	major histocompatibility complex, class I, C	Homo sapiens	132-135	
3997878-6	1985	Comparison of the homologous MHC sequences shows two positions at which MHC from dystrophic chicks differs from that of the White Leghorn chicks *(Glu----Gln and Met----Ile).	Glutamic Acid	147-150	major histocompatibility complex, class I, C	Homo sapiens	29-32	
8093457-7	1993	However, these two effects of glutamic acid were not complementary and were mediated by distinct mechanisms, as the change in the peptide altered the extent of binding to class II, but the change in the MHC molecule decreased recognition without inhibiting peptide binding.	Glutamic Acid	30-43	major histocompatibility complex, class I, C	Homo sapiens	203-206	
3997878-6	1985	Comparison of the homologous MHC sequences shows two positions at which MHC from dystrophic chicks differs from that of the White Leghorn chicks *(Glu----Gln and Met----Ile).	Glutamic Acid	147-150	major histocompatibility complex, class I, C	Homo sapiens	72-75