PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29903185-8	2017	07), the TGF-beta +DPI group is( 0.	3-aminodiphenyleneiodium	19-22	transforming growth factor beta 1	Homo sapiens	9-17	
29903185-15	2017	6), the TGF-beta + DPI group is( 1.	3-aminodiphenyleneiodium	19-22	transforming growth factor beta 1	Homo sapiens	8-16	
29903185-21	2017	1), the TGF-beta + DPI group is( 3.	3-aminodiphenyleneiodium	19-22	transforming growth factor beta 1	Homo sapiens	8-16	
29903185-27	2017	Then TGF-beta + DPI group compared with the TGF-beta group, the scratch healing rate is decreased( F = 33.	3-aminodiphenyleneiodium	16-19	transforming growth factor beta 1	Homo sapiens	5-13	
29903185-31	2017	CONCLUSION: After the NOX4 was inhibited by DPI, TGF-beta-induced migration of A549 cells was inhibited.	3-aminodiphenyleneiodium	44-47	transforming growth factor beta 1	Homo sapiens	49-57	
25175743-8	2014	Using a NOX inhibitor (DPI) and cells expressing a shRNA for NOX4, we demonstrated that TGF-beta1 promotes an oxidative environment that favors myofibroblastic differentiation.	3-aminodiphenyleneiodium	23-26	transforming growth factor beta 1	Homo sapiens	88-97	
20204677-6	2010	Our results shows that TGF-beta1 stimulation of uPA and MMP-9 expression involve NOXs-dependent ROS and NFkappaB, activation, demonstrated by using DPI, NOXs inhibitor, ROS scavenger N-acetylcysteine and SN50, an NFkb inhibitor.	3-aminodiphenyleneiodium	148-151	transforming growth factor beta 1	Homo sapiens	23-32	
32736702-5	2020	TGF-beta1 treatment increased reactive oxygen species (ROS) via NOX4 upregulation, which acts downstream of ERK and mTORC1, as the ROS scavenger N-acetylcysteine and a pan-NADPH oxidase (NOX) inhibitor DPI dissipated excess ROS generation.	3-aminodiphenyleneiodium	202-205	transforming growth factor beta 1	Homo sapiens	0-9	
16159901-6	2005	TGF-beta stimulation of ROS was mediated by the NAPDH oxidase homolog Nox4 as DPI, an inhibitor of NADPH oxidase, and dominant-negative Nox4 adenovirus blocked ROS production.	3-aminodiphenyleneiodium	78-81	transforming growth factor beta 1	Homo sapiens	0-8	
11494050-5	2001	We also observed that TGF-beta-induced p38 activation was inhibited by PDTC, pyrrolidinedithiocarbamate, a known antioxidant, and DPI, diphenylene iodonium chloride, one of the known NADPH oxidase inhibitors.	3-aminodiphenyleneiodium	130-133	transforming growth factor beta 1	Homo sapiens	22-30	
11494050-9	2001	DPI, an inhibitor of NADPH oxidase, inhibited TGF-beta-induced ROS production.	3-aminodiphenyleneiodium	0-3	transforming growth factor beta 1	Homo sapiens	46-54	
11494050-12	2001	Pretreatment with DPI dramatically reduced TGF-beta-induced NADPH oxidase activity.	3-aminodiphenyleneiodium	18-21	transforming growth factor beta 1	Homo sapiens	43-51	
32736702-6	2020	TGF-beta1-induced oxidative stress resulted in EMT and fibrotic changes, as NAC and DPI prevented alpha-SMA, Col4alpha3 expression and cell migration.	3-aminodiphenyleneiodium	84-87	transforming growth factor beta 1	Homo sapiens	0-9