PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
12962151-4	2003	The ROS formation in the AD treatment was evidenced by flow cytometry, and further supported by suppression of caspase-3 activation by superoxide radical-forming enzyme inhibitors (TTFA, rotenone, and DPI).	3-aminodiphenyleneiodium	201-204	caspase 3	Homo sapiens	111-120	
22038300-11	2013	The Hcy-induced increase in caspase-3 activity was prevented by DPI and apocynin, suggesting involvement of NOX activity.	3-aminodiphenyleneiodium	64-67	caspase 3	Homo sapiens	28-37	
17517376-5	2007	A flow cytometry based DCFH-DA analysis and inhibitory studies with DPI, a pharmacological inhibitor of NADPH oxidase, NAC (N-acetyl cysteine) and GSH revealed that NADPH oxidase-mediated generation of ROS is responsible for caspase-3 activation and subsequent induction of apoptosis in the K-562 cell line.	3-aminodiphenyleneiodium	68-71	caspase 3	Homo sapiens	225-234