PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
20138953-7	2010	Ethanol and CCl(4)-induced the rat liver damage, and significantly increased (p<0.05) the GPT, gamma-GT and MDA levels, and decreased the SOD, CAT and GPx levels.	Cefaclor	12-15	glutamic--pyruvic transaminase	Rattus norvegicus	93-96	
21511893-3	2011	Intraperitoneal administration of CCl(4) (2 ml/kg) to rats for 4 days resulted in significantly elevated (p < 0.05) serum levels of glutamic oxaloacetic transaminase (SGOT), glutamic pyruvate transaminase (SGPT), alkaline phosphatase (ALP) and lactate dehydrogenase (LDH), when compared to normal rats.	Cefaclor	34-37	glutamic--pyruvic transaminase	Rattus norvegicus	177-207	
21540102-11	2011	Administration of CCl(4) in rats elevated the serum GPT, GOT, and ALP level whereas silymarin, Gimjeng and Chakapat extracts prevented these increases significantly.	Cefaclor	18-21	glutamic--pyruvic transaminase	Rattus norvegicus	52-55	
21598418-3	2011	The results showed that CCW (0.1, 0.5 and 1.0 g/kg) significantly reduced the elevated levels of GPT and GOT by CCl(4) administration (p < 0.05).	Cefaclor	112-115	glutamic--pyruvic transaminase	Rattus norvegicus	97-100	
16220574-8	2005	Histology evaluation revealed a normal hepatic parenchyma at 48 h; the injury was fully restored after 72 h. Moreover, a significant reduction in CCl(4)-induced increase of GOT and GPT plasma levels is evident; these data are in agreement with the functional improvement of hepatocytes.	Cefaclor	146-149	glutamic--pyruvic transaminase	Rattus norvegicus	181-184	
20128046-6	2010	CCl(4) induced liver poisoning in all treated animals was evident by elevated serum GOT, GPT, ALP, GGT and bilirubin levels.	Cefaclor	0-3	glutamic--pyruvic transaminase	Rattus norvegicus	89-92	
18481019-4	2008	CCl(4)-treatment was found to increase the levels of GOT, GPT, ALP, LDH and MDA, as well as decrease levels of SOD, CAT and GPx significantly.	Cefaclor	0-3	glutamic--pyruvic transaminase	Rattus norvegicus	58-61	
20161932-8	2008	There was a significant rise in the levels of serum GOT, GPT, and ALP and other biochemical parameters, decrease in the levels of SOD, catalase and peroxidase after administration of CCl(4).	Cefaclor	183-186	glutamic--pyruvic transaminase	Rattus norvegicus	57-60	
16914248-2	2006	Intraperitoneal administration of CCl(4) (2ml/kg) to rats for 4 days resulted in significantly elevated (p<0.05) serum levels of glutamic oxaloacetic transaminase (SGOT), glutamic pyruvate transaminase (SGPT) and alkaline phosphatase (SALP) compared to controls.	Cefaclor	34-37	glutamic--pyruvic transaminase	Rattus norvegicus	174-204	
18404313-9	2008	In the CCl(4)-treated control group, there were marked increases in LDH, GOT, and GPT activities compared with the normal group.	Cefaclor	7-10	glutamic--pyruvic transaminase	Rattus norvegicus	82-85	
11819383-5	1999	At 48h, the survivability of rat hepatocytes was assayed by the MTT colormetric method.RESULTS:After CCl(4) challenge, the release of GPT and the formation of MDA in rat hepatocytes markedly increased and maintained at a high level in 48h, whereas PD with different concentrations could markedly inhibit this elevation with 10(-5)mol/L PD having the strongest effects and inhibiting rate was over 50%.	Cefaclor	101-104	glutamic--pyruvic transaminase	Rattus norvegicus	134-137	
23195938-5	1994	Similarly, a hepatotoxic dose of CCl(4) (1.5 mL/kg; orally) significantly raised (P < 0.01), the serum ALP, GOT and GPT levels to 312 +- 20, 503 +- 98 and 407 +- 109 IU/L (n = 10) respectively, compared to respective control values of 215 +- 16, 79 +- 18 and 49 +- 10.	Cefaclor	33-36	glutamic--pyruvic transaminase	Rattus norvegicus	119-122	
23195938-6	1994	The same dose of plant extract (500 mg/kg) was able to prevent significantly (P < 0.05) the CCl(4)-induced rise in serum enzymes and the estimated values of ALP, GOT and GPT were 222 +- 27, 114 +- 23 and 68 +- 14 respectively.	Cefaclor	95-98	glutamic--pyruvic transaminase	Rattus norvegicus	173-176