PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23629449-0	2013	Design and synthesis of tryptophan containing dipeptide derivatives as formyl peptide receptor 1 antagonist.	Dipeptides	46-55	formyl peptide receptor 1	Homo sapiens	71-96	
32206242-0	2019	Aurantiamide-related dipeptide derivatives are formyl peptide receptor 1 antagonists.	Dipeptides	21-30	formyl peptide receptor 1	Homo sapiens	47-72	
32206242-7	2019	Since FPR1 is a key regulator of the inflammatory environment, the dipeptide derivatives described here may represent important leads for the development of new potent and selective FPR1 antagonists for the treatment of neutrophil-mediated inflammatory diseases.	Dipeptides	67-76	formyl peptide receptor 1	Homo sapiens	6-10	
32206242-7	2019	Since FPR1 is a key regulator of the inflammatory environment, the dipeptide derivatives described here may represent important leads for the development of new potent and selective FPR1 antagonists for the treatment of neutrophil-mediated inflammatory diseases.	Dipeptides	67-76	formyl peptide receptor 1	Homo sapiens	182-186	
28232203-0	2017	Dipeptide HCH6-1 inhibits neutrophil activation and protects against acute lung injury by blocking FPR1.	Dipeptides	0-9	formyl peptide receptor 1	Homo sapiens	99-103	
28232203-3	2017	The purpose of this study was to identify a synthetic dipeptide N-(N-benzoyl-L-tryptophanyl)-D-phenylanlanine methyl ester (HCH6-1) as a FPR1 inhibitor and to investigate its protective effects against acute lung injury (ALI).	Dipeptides	54-63	formyl peptide receptor 1	Homo sapiens	137-141	
27454150-8	2016	Expert opinion: In this review, FPR1 modulators comprise beta-1,3-glucan synthase inhibitors containing an FPR ligand moiety, template-fixed peptidomimetics, cyclosporin H, and dipeptide derivatives.	Dipeptides	177-186	formyl peptide receptor 1	Homo sapiens	32-36	
23629449-1	2013	Our previous studies identified an Fmoc-(S,R)-tryptophan-containing dipeptide derivative, 1, which selectively inhibited neutrophil elastase release induced by formyl-L-methionyl-L-leucyl-L-phenylalanine (FMLP) in human neutrophils.	Dipeptides	68-77	formyl peptide receptor 1	Homo sapiens	205-209	
23629449-4	2013	Further studies indicated that inhibition of superoxide production in human neutrophils by these dipeptides was associated with the selective inhibition of formyl peptide receptor 1 (FPR1).	Dipeptides	97-107	formyl peptide receptor 1	Homo sapiens	156-181	
23629449-4	2013	Further studies indicated that inhibition of superoxide production in human neutrophils by these dipeptides was associated with the selective inhibition of formyl peptide receptor 1 (FPR1).	Dipeptides	97-107	formyl peptide receptor 1	Homo sapiens	183-187	
16714011-9	2006	O-phosphoserylethanolamine, a new dipeptide isolated from mamushi, also suppressed fMLP-induced superoxide generation in human neutrophils in a dose-dependent manner.	Dipeptides	34-43	formyl peptide receptor 1	Homo sapiens	83-87