PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28442777-5	2017	Treatment of wild type mice with paroxetine, a chemical inhibitor of SERT, also resulted in Cyp19a1 suppression, decreased circulating 17beta-estradiol levels, abnormal fat accumulation, and glucose intolerance.	Paroxetine	33-43	cytochrome P450, family 19, subfamily a, polypeptide 1	Mus musculus	92-99	
31308195-4	2019	Chronic paroxetine treatment in nonpregnant mice resulted in visceral adiposity and glucose intolerance accompanied by reduced circulating 17beta-estradiol levels and ovarian expression of the aromatase (CYP19a1).	Paroxetine	8-18	cytochrome P450, family 19, subfamily a, polypeptide 1	Mus musculus	204-211	
31308195-5	2019	Remarkably, pregnancy significantly reduced adiposity and improved glucose tolerance in paroxetine-treated mice by rebooting ovarian CYP19a1 expression and 17beta-estradiol production.	Paroxetine	88-98	cytochrome P450, family 19, subfamily a, polypeptide 1	Mus musculus	133-140