PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25135648-7	2014	In conclusion, our data support that DMF stimulates Nrf2 activity to attenuate hyperphosphatamia in vitro or Vitamin D3-induced in vivo vascular calcification, which would be a beneficial effect on vascular diseases induced by oxidative stress such as vascular calcification.	Cholecalciferol	109-119	nuclear factor, erythroid derived 2, like 2	Mus musculus	52-56	
34824670-0	2021	Vitamin D3 Protects Mice from Diquat-Induced Oxidative Stress through the NF-kappaB/Nrf2/HO-1 Signaling Pathway.	Cholecalciferol	0-10	nuclear factor, erythroid derived 2, like 2	Mus musculus	84-88	
34824670-7	2021	In addition, the results indicated that vitamin D3 could significantly reduce the phosphorylation level of NF-kappaB (p65) and enhance the expression of Nrf2 and HO-1 in the jejunum compared with the diquat-induced group.	Cholecalciferol	40-50	nuclear factor, erythroid derived 2, like 2	Mus musculus	153-157	
34824670-8	2021	This study suggested that oral administration of vitamin D3 can protect mice against oxidative damage by inhibiting the phosphorylation level of NF-kappaB (p65) and activating Nrf2-related signaling pathways.	Cholecalciferol	49-59	nuclear factor, erythroid derived 2, like 2	Mus musculus	176-180	
27484042-0	2016	The protective role of vitamin D3 in a murine model of asthma via the suppression of TGF-beta/Smad signaling and activation of the Nrf2/HO-1 pathway.	Cholecalciferol	23-33	nuclear factor, erythroid derived 2, like 2	Mus musculus	131-135	
27484042-8	2016	Furthermore, treatment with vitamin D3 enhanced activation of the Nrf2/HO-1 pathway in the airways of asthmatic mice.	Cholecalciferol	28-38	nuclear factor, erythroid derived 2, like 2	Mus musculus	66-70	
27484042-9	2016	In conclusion, these findings suggest that vitamin D3 may protect airways from asthmatic damage via the suppression of TGF-beta/Smad signaling and activation of the Nrf2/HO-1 pathway; however, these protective effects were shown to be accompanied by hypercalcemia.	Cholecalciferol	43-53	nuclear factor, erythroid derived 2, like 2	Mus musculus	165-169