PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27183464-2	2016	As a biomarker of neuroinflammatory processes, we designed (11)C-labeled ketoprofen methyl ester ([(11)C]KTP-Me) to increase the blood-brain barrier permeability of ketoprofen (KTP), a selective cyclooxygenase-1 (COX-1) inhibitor.	Ketoprofen	73-83	prostaglandin-endoperoxide synthase 1	Homo sapiens	195-211	
27183464-2	2016	As a biomarker of neuroinflammatory processes, we designed (11)C-labeled ketoprofen methyl ester ([(11)C]KTP-Me) to increase the blood-brain barrier permeability of ketoprofen (KTP), a selective cyclooxygenase-1 (COX-1) inhibitor.	Ketoprofen	73-83	prostaglandin-endoperoxide synthase 1	Homo sapiens	213-218	
27183464-2	2016	As a biomarker of neuroinflammatory processes, we designed (11)C-labeled ketoprofen methyl ester ([(11)C]KTP-Me) to increase the blood-brain barrier permeability of ketoprofen (KTP), a selective cyclooxygenase-1 (COX-1) inhibitor.	Ketoprofen	105-108	prostaglandin-endoperoxide synthase 1	Homo sapiens	195-211	
27183464-2	2016	As a biomarker of neuroinflammatory processes, we designed (11)C-labeled ketoprofen methyl ester ([(11)C]KTP-Me) to increase the blood-brain barrier permeability of ketoprofen (KTP), a selective cyclooxygenase-1 (COX-1) inhibitor.	Ketoprofen	105-108	prostaglandin-endoperoxide synthase 1	Homo sapiens	213-218	
24853403-2	2014	As a biomarker of neuroinflammatory processes, (11)C-labeled ketoprofen methyl ester ([(11)C]KTP-Me) was designed to allow cerebral penetration of ketoprofen (KTP), an active form of a selective cyclooxygenase-1 inhibitor that acts as an NSAID.	Ketoprofen	61-71	prostaglandin-endoperoxide synthase 1	Homo sapiens	195-211	
16937749-2	2006	OBJECTIVE: To classify patients with a history of aspirin-induced urticaria as cross-reactors or single-drug reactors by oral challenges with another strong COX-1 inhibitor, namely, ketoprofen.	Ketoprofen	182-192	prostaglandin-endoperoxide synthase 1	Homo sapiens	157-162	
8581280-9	1995	Under these experimental conditions, ketoprofen enantioselectively inhibited the cyclo-oxygenase activity of both PGHS-1 and PGHS-2 with equal potency (IC50 ratio: approx.	Ketoprofen	37-47	prostaglandin-endoperoxide synthase 1	Homo sapiens	114-120