PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32624316-6	2021	RESULTS: AITC decreased CAR cell viability, induced cell death of CAR cells and inhibited the confluences of cultured CAR cells.	allyl isothiocyanate	9-13	CXADR pseudogene 1	Homo sapiens	24-27	
32624316-6	2021	RESULTS: AITC decreased CAR cell viability, induced cell death of CAR cells and inhibited the confluences of cultured CAR cells.	allyl isothiocyanate	9-13	CXADR pseudogene 1	Homo sapiens	66-69	
32624316-6	2021	RESULTS: AITC decreased CAR cell viability, induced cell death of CAR cells and inhibited the confluences of cultured CAR cells.	allyl isothiocyanate	9-13	CXADR pseudogene 1	Homo sapiens	66-69	
32624316-7	2021	When CAR cells were treated with AITC, activation of caspase-3 and caspase-9 by AITC was observed and could be reversed by Z-VAD-fmk (pan-caspase inhibitor).	allyl isothiocyanate	33-37	CXADR pseudogene 1	Homo sapiens	5-8	
32624316-8	2021	Furthermore, the protein expressions of phosphorylated protein kinase B (p-AKT) and phosphorylated mammalian target of rapamycin (p-mTOR) were suppressed in AITC-treated CAR cells, whereas protein expressions of Bax, cytochrome c, Apaf-1, cleaved caspase-3, and cleaved caspase-9 were upregulated in AITC-treated CAR cells.	allyl isothiocyanate	157-161	CXADR pseudogene 1	Homo sapiens	170-173	
32624316-8	2021	Furthermore, the protein expressions of phosphorylated protein kinase B (p-AKT) and phosphorylated mammalian target of rapamycin (p-mTOR) were suppressed in AITC-treated CAR cells, whereas protein expressions of Bax, cytochrome c, Apaf-1, cleaved caspase-3, and cleaved caspase-9 were upregulated in AITC-treated CAR cells.	allyl isothiocyanate	157-161	CXADR pseudogene 1	Homo sapiens	313-316	
32624316-9	2021	CONCLUSION: AITC can inhibit Akt/mTOR proliferation signaling and promote mitochondria-dependent apoptotic pathway through AITC-enhanced activities of caspase-3 and caspase-9 in CAR cells.	allyl isothiocyanate	12-16	CXADR pseudogene 1	Homo sapiens	178-181	
32624316-9	2021	CONCLUSION: AITC can inhibit Akt/mTOR proliferation signaling and promote mitochondria-dependent apoptotic pathway through AITC-enhanced activities of caspase-3 and caspase-9 in CAR cells.	allyl isothiocyanate	123-127	CXADR pseudogene 1	Homo sapiens	178-181