PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 31948828-1 2020 Pharmacological inhibition of the enzyme fatty acid amide hydrolase (FAAH), which terminates signaling of the endocannabinoid N-arachidonoylethanolamine (or anandamide, AEA), exerts favourable effects in rodent models of stress-related depression. N,N,N-trimethyl-N'-cholesteryl amidoethyl ammonium 169-172 fatty-acid amide hydrolase-like Rattus norvegicus 69-73 31948828-8 2020 Conversely, pharmacological inhibition of FAAH activity with URB694 restored both AEA and BDNF levels within the hippocampus of socially isolated rats and prevented the development of behavioral and physiological alterations. N,N,N-trimethyl-N'-cholesteryl amidoethyl ammonium 82-85 fatty-acid amide hydrolase-like Rattus norvegicus 42-46