Title : HDAC11 is a novel regulator of fatty acid oxidative metabolism in skeletal muscle.

Pub. Date : 2021 Feb

PMID : 32563202






1 Functional Relationships(s)
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1 In particular, we showed that HDAC11 depletion increased mitochondrial fatty acid beta-oxidation through activating the AMPK-ACC pathway and reducing acylcarnitine levels in vivo, thus providing a mechanistic explanation for the improved muscle strength and fatigue resistance. Fatty Acids protein kinase AMP-activated catalytic subunit alpha 1 Homo sapiens