Title : ARID1A influences HDAC1/BRD4 activity, intrinsic proliferative capacity and breast cancer treatment response.

Pub. Date : 2020 Feb

PMID : 31913353






1 Functional Relationships(s)
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1 Deletion of ARID1A resulted in loss of histone deacetylase 1 binding, increased histone 4 lysine acetylation and subsequent BRD4-driven transcription and growth. tyrosyl-lysine AT-rich interaction domain 1A Homo sapiens