Title : The modulation of TRPM7 currents by nafamostat mesilate depends directly upon extracellular concentrations of divalent cations.

Pub. Date : 2010 Dec 1

PMID : 21122141

1 Functional Relationships(s)
Compound Name
Protein Name
1 Here we report that a synthetic serine protease inhibitor, nafamostat mesylate (NM), and several of its analogues, block recombinant TRPM7 currents expressed in HEK293T cells in inverse relationship to the concentration of extracellular divalent cations. nafamostat transient receptor potential cation channel subfamily M member 7 Homo sapiens