Title : Mineralocorticoid excess and inhibition of 11 beta-hydroxysteroid dehydrogenase in patients with ectopic ACTH syndrome.

Pub. Date : 1992 Dec

PMID : 1337504

1 Functional Relationships(s)
Compound Name
Protein Name
1 In ectopic ACTH syndrome the characteristic mineralocorticoid excess can be accounted for by a combination of increased secretion of cortisol, corticosterone and of 11-deoxycorticosterone and decreased inactivation of cortisol and corticosterone by 11 beta-dehydrogenase. Desoxycorticosterone proopiomelanocortin Homo sapiens