Tetracycline

epidermal growth factor receptor ; Homo sapiens







12 Article(s)
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1 35113224 Topical corticosteroid therapy for facial acneiform eruption due to EGFR inhibitors in metastatic colorectal cancer patients: a randomized controlled trial comparing starting with a very strong or a weak topical corticosteroid (FAEISS study, NCCH1512, colorectal part). 2022 May 2
2 35352492 Prevention and management of acneiform rash associated with EGFR inhibitor therapy: A systematic review and meta-analysis. 2022 Mar 29 1
3 34362003 The Impact of Long-Term Antibiotic Therapy of Cutaneous Adverse Reactions to EGFR Inhibitors in Colorectal Cancer Patients. 2021 Jul 21 1
4 33096790 De Novo T790M Mutation in an L858R Epidermal Growth Factor Receptor Mutant-Associated Lung Adenocarcinoma. 2020 Oct 21 1
5 28003335 The RAS-related GTPase RHOB confers resistance to EGFR-tyrosine kinase inhibitors in non-small-cell lung cancer via an AKT-dependent mechanism. 2017 Feb 1
6 20820817 Prophylactic tetracycline does not diminish the severity of epidermal growth factor receptor (EGFR) inhibitor-induced rash: results from the North Central Cancer Treatment Group (Supplementary N03CB). 2011 Oct 2
7 20428186 Amphiregulin carboxy-terminal domain is required for autocrine keratinocyte growth. 2010 Aug 1
8 18543329 Tetracycline to prevent epidermal growth factor receptor inhibitor-induced skin rashes: results of a placebo-controlled trial from the North Central Cancer Treatment Group (N03CB). 2008 Aug 15 1
9 17234777 A novel mechanism of resistance to epidermal growth factor receptor antagonism in vivo. 2007 Jan 15 2
10 15115657 Epstein-Barr virus latent membrane protein 1 modulates epidermal growth factor receptor promoter activity in a nuclear factor kappa B-dependent manner. 2004 Jul 1
11 15177893 Nuclear factor kappa B (NFkappaB) dependent modulation of Epstein-Barr virus latent membrane protein 1 (LMP1) in epidermal growth factor receptor (EGFR) promoter activity. 2004 Aug 1
12 19877917 Studies on engineered autocrine systems: requirements for ligand release from cells producing an artificial growth factor. 1995 Spring 2